1                         Using the assay, the alpha2a adrenoceptor agonist UK14,304 was shown to simul

2                  Stimulation of postsynaptic alpha2A adrenoceptors (alpha2A-ARs) is critical for WM.

3 pecially vulnerable to inflammation, and how alpha2A-adrenoceptor (alpha2A-AR) actions throughout the

4 le source mediating improved flexibility are alpha2A adrenoceptors (alpha2AR) in prefrontal cortex (P

5 l how a series of molecular events involving alpha2A-adrenoceptors and a class of ion channels gated

6            Since intrathecal injection of an alpha(2A)-adrenoceptor antagonist (BRL 44408) partially

7 ice is influenced by the genetic deletion of alpha2A adrenoceptors (ARs).

8                                              alpha2A-Adrenoceptors did not differ significantly from

9 in occipital cortex and hippocampus (and for alpha2A-adrenoceptors in caudate and amygdala) compared

10 ults indicate that activation of presynaptic alpha2A-adrenoceptors on inputs to SPNs decreases glutam

11 ion of calcium-cAMP signaling by stimulating alpha2A-adrenoceptors on spines strengthens synaptic eff

12 ine were prevented by coadministration of an alpha2A-adrenoceptor-preferring antagonist.

13                               Therefore, the alpha2A adrenoceptor subtype is responsible for the hypn