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1 on as well as cointernalization of micro and alpha2A receptors.
2 cross-desensitization nor internalization of alpha2A receptors.
3 th simultaneous internalization of micro and alpha2A receptors.
4  cross-regulation between neuronal micro and alpha2A receptors.
5 serotonin) 5-HT(1b) receptors and adrenergic alpha(2a) receptors.
6 uated guanfacine immediate release (GIR), an alpha(2A) receptor agonist, as a novel treatment aimed a
7  5-HT(2B), 5-HT(1A), 5-HT(6), and adrenergic alpha(2A) receptors, and lacked activity at monoamine tr
8  excellent selectivity over D2-D4 receptors, alpha2a receptor, and the 5-HT transporter.
9                    These results reveal that alpha2A receptors are part of the causal chain of flexib
10                                           mu-alpha2A Receptor complexes can be isolated from heterolo
11 to-Kakizaki rats that overexpress adrenergic alpha2A receptors, confirming the role of leucine in ins
12 st that physical associations between mu and alpha2A receptors could play a role in the functional in
13                                          The alpha2a-receptor does not internalize after agonist trea
14 otonin 5-HT(1A) and 5-HT(2A), and adrenergic alpha(2A) receptors in comparison with 5-(4-methylpipera
15 oglial process dynamics in resting cells and alpha2A receptors in activated cells.
16 al interactions between endogenous micro and alpha2A receptors in mouse dorsal root ganglion neurons.
17 beneficial hypotensive effect of stimulating alpha2a receptors in the central nervous system.
18 and kappa-opioid receptors and by adrenergic alpha2A receptors in the spinal cord.
19  these cells, the activation of either mu or alpha2A receptor leads to a significant increase in the
20      We find that activation of either mu or alpha2A receptors leads to an increase in the extent of
21 tual cross-desensitization between micro and alpha2A receptor-mediated current inhibition.
22 udies reveal the crucial contribution of the alpha2A receptor subtype in suppression of epileptogenes
23 nts that promote selective activation of the alpha2A receptor subtype may provide novel therapeutic s
24 ess beta2 receptors but switch expression to alpha2A receptors under proinflammatory conditions model
25 gulated the surface expression of adrenergic alpha2A receptor via activation of the mTOR pathway.
26 ation rate of the agonist [(3)H]UK14304 from alpha(2A)-receptors was decreased by the amilorides in a
27 The potency of norepinephrine for adrenergic alpha2A receptor was only about 20-fold higher compared