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   1  be reduced by therapy with phentolamine, an alpha-adrenergic antagonist.                            
     2 ty was also delayed by treating mice with an alpha-adrenergic antagonist.                            
     3 inistration of a beta(2) (but not beta(1) or alpha) adrenergic antagonist.                           
     4 nociception partially blocked by intrathecal alpha-adrenergic antagonists, but the mechanism underlyi
  
     6  hormone epinephrine and show that beta- and alpha-adrenergic antagonists can block the bacterial res
     7 pamine was administered with and without the alpha-adrenergic antagonist dapiprazole, and its effects
  
     9 To update the physician regarding the use of alpha-adrenergic antagonists in the management of variou
    10 ipulations in vivo, we show that infusion of alpha-adrenergic antagonists into the NIf (nucleus inter
  
  
  
    14  noradrenaline release) and phentolamine (an alpha-adrenergic antagonist), profound vasodilatation wa
    15 agonist to the microvessel preparation or an alpha-adrenergic antagonist to the myocytes and was augm
    16 e derived vasoconstriction, phentolamine, an alpha-adrenergic antagonist was administered prior to MA
  
  
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