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1 other agents which are more specific for the alpha1 adrenergic receptor.
2 mIPSCs frequency results from activation of alpha1 adrenergic receptors.
3 these modulatory effects are attributable to alpha1 adrenergic receptors.
4 nformation describing this drug class at the alpha1-adrenergic receptors.
5 , thereby ruling out [3H]CUMI-101 binding to alpha1-adrenergic receptors.
6 yl cyclase, and protein kinase A, as well as alpha1-adrenergic receptors.
7 E release and that this might be specific to alpha1-adrenergic receptors.
8 inity for dopamine D2, serotonin 5-HT1A, and alpha1-adrenergic receptors.
9 h suggested an association between Panx1 and alpha1D-adrenergic receptor.
14 Consistent with this idea, the selective alpha1-adrenergic receptor agonist methoxamine was still
15 cked the induction of these responses by the alpha1-adrenergic receptor agonist phenylephrine, wherea
16 ith either insulin or phenylephrine (PE), an alpha1-adrenergic receptor agonist, induced Ser-9 phosph
17 itoneally) or saline, the response to the a) alpha1-adrenergic receptor agonist, phenylephrine; b) en
18 was perturbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure
19 was perturbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure
21 sylated Rho and inhibited the characteristic alpha1-adrenergic receptor agonist-induced myofibrillar
24 are induced in cultured myocardial cells by alpha1- adrenergic receptor agonists, such as phenylephr
25 inately induced by growth promoters, such as alpha1-adrenergic receptor agonists (e.g. phenylephrine
26 through vasopressin receptor (V1A-R) but not alpha1 adrenergic receptor (alpha1-AR), suggesting that
27 rat ventricular myocytes, stimulation of the alpha1-adrenergic receptor (alpha1-AdrR) activates a pro
28 oth muscle, or to phenylephrine, a selective alpha1-adrenergic receptor (alpha1-AR) agonist, was simi
30 alpha1ABKO) mice that lacked cardiac myocyte alpha1-adrenergic receptor (alpha1-AR) binding, aortic c
31 ing mediated by the angiotensin (Ang) II and alpha1-adrenergic receptor (alpha1-AR) pathways is impor
37 loned cDNAs encoding three subtypes of human alpha1-adrenergic receptors (alpha1ARs), alpha1a, alpha1
39 botropic neurotransmitter receptors (mGluRs, alpha1 adrenergic receptors, and muscarinic acetylcholin
40 ease of dopamine, activation of postsynaptic alpha1 adrenergic receptors, and suppression of InsP3-in
41 us losartan (10 mg/kg b.wt) nor prazosin, an alpha1 adrenergic receptor antagonist, at doses of 5 mic
43 e Cl- current to 5.9 nM, and addition of the alpha1-adrenergic receptor antagonist prazosin did not c
45 61W retinal cells in high glucose and of the alpha1-adrenergic receptor (AR) on vascular lesions of t
48 hat the type 1 muscarinic receptor (m1R) and alpha1-adrenergic receptor (AR)-mediated SRF activation
49 nd novel cytoplasmic binding partners of the alpha1D-adrenergic receptor (AR), a yeast two-hybrid scr
50 ion of vascular smooth muscle, activation of alpha1D-adrenergic receptors (AR) induces smooth muscle
54 of blood pressure through interactions with alpha1-adrenergic receptors (ARs) in vascular smooth mus
55 direct evidence suggests that stimulation of alpha1-adrenergic receptors (ARs) increases smooth muscl
59 ral-midbrain dopaminergic neurons through an alpha1 adrenergic receptor-dependent mechanism within th
60 tes interfered with the basal expression and alpha1-adrenergic receptor-dependent activation of a TEF
63 he mechanism underlying the critical role of alpha1 adrenergic receptors in the regulation of dopamin
64 ed if insulin or IGF-I induced expression of alpha1 adrenergic receptors in vascular smooth muscle ce
65 hat significantly altered mRNA levels of the alpha1- adrenergic receptor in the basal forebrain as we
66 our recently demonstrated mechanism whereby alpha1-adrenergic receptors in the same cell type regula
70 70 gene expression induced by stimulation of alpha1 adrenergic receptors is markedly attenuated, whic
71 concurrent episodic activation of 5-HT2 and alpha1-adrenergic receptors is necessary to produce long
74 ases neural stimulation of BAT and increases alpha1-adrenergic receptor number; however, the alpha1 r
75 that in MDCK-D1 cells, epinephrine acting on alpha1-adrenergic receptors, promotes a rapid increase i
76 ikewise, ANP and 8-bromo-cGMP attenuated the alpha1-adrenergic receptor- stimulated increase in prepr
77 imulated increase in prepro-ANP mRNA and the alpha1-adrenergic receptor-stimulated decrease in sarcop
78 re, in myocytes, 8-bromo-cGMP attenuated the alpha1-adrenergic receptor-stimulated increases in c-fos
80 d signal transduction mechanisms involved in alpha1, adrenergic receptor stimulation of hsp7O gene ex
83 or (ANF) reporter gene expression induced by alpha1-adrenergic receptor stimulation with phenylephrin
84 ing assays were used to examine the basis of alpha1-adrenergic receptor subtype selectivity for a ser
86 with that previously described for the other alpha1 adrenergic receptor subtypes, may shed light on t
87 nsfected with each of the three cloned human alpha1 adrenergic receptor subtypes, norepinephrine stro
88 y episodic but not continuous stimulation of alpha1-adrenergic receptors that requires protein kinase
89 slated and active in costimulating, with the alpha1-adrenergic receptor, the firing of DR 5-HT neuron
91 was to determine the effect of endotoxin on alpha1-adrenergic receptor vasoconstriction and both end
92 tors, is also shown to be a modulator of the alpha1-adrenergic receptors, which suggests a general la
93 ncreases intracellular Ca(2+) similar to the alpha1 adrenergic receptor, while OAMB-K3 additionally s
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