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1 including the m5 muscarinic receptor and the alpha1B adrenergic receptor.
2 te allosteric modulation of the alpha1A- and alpha1B-adrenergic receptors.
3 lls transfected with mutant versus wild-type alpha1b-adrenergic receptors.
4                   In contrast, activation of alpha1B-adrenergic receptors (alpha1B-ARs) induced trans
5 ection of NF1/L, NF1/Red1, or NF1/X with the alpha1B adrenergic receptor (alpha1BAR) gene middle (P2)
6  Progress toward elucidating the function of alpha1B-adrenergic receptors (alpha1BARs) in the central
7                                    Since the alpha1B adrenergic receptor and dopamine receptor type 1
8 ]prazosin dissociation from the alpha1A- and alpha1B-adrenergic receptors and noncompetitively inhibi
9 ial-targeted overexpression of the wild-type alpha1B adrenergic receptor (AR) (Tg alpha43), we studie
10              We previously demonstrated that alpha1B-adrenergic receptor (AR) gene transcription, mRN
11 pecific overexpression of the wild-type (WT) alpha1B-adrenergic receptor (AR) using the murine alpha-
12 both the alpha1a-adrenergic receptor and the alpha1b-adrenergic receptor (AR), that account almost en
13                    The analogous residues in alpha1b-adrenergic receptors, aspartic acid 125 and lysi
14  type A receptor, dopamine D1A receptor, and alpha1b adrenergic receptor) bound betaarrestin2 with hi
15                    Upon agonist stimulation, alpha1B-adrenergic receptors couple to Gq proteins, calc
16    We investigated the role of this motif in alpha1B-adrenergic receptor function and regulation.
17 and CP1 bind to the major P2 promoter of the alpha1B adrenergic receptor gene to generate footprint I
18 specific regulation of the expression of the alpha1B adrenergic receptor gene.
19                                        Y348A alpha1B-adrenergic receptors in which this sequence was
20 tivation, (ii) provide further evidence that alpha1B-adrenergic receptor internalization can be separ
21                                The expressed alpha1b-adrenergic receptor mutant demonstrated a 6-fold
22 ere obtained for a lysine to a glutamic acid alpha1b-adrenergic receptor mutation.
23                        Taken together, these alpha1b-adrenergic receptor mutations suggest a molecula
24 were also observed for two aspartic acid 125 alpha1b-adrenergic receptor mutations, consistent with t
25 icular traffic were investigated by studying alpha1B-adrenergic receptor-Rab protein interactions, us
26 of radioligand binding sites for two mutated alpha1B-adrenergic receptors reported previously was inv
27                                         This alpha1b-adrenergic receptor salt bridge constraint is th
28  beta2 but not in cells transfected with the alpha1B-adrenergic receptor, suggesting that the PLC bet
29                                         When alpha1B-adrenergic receptors that had been mutated at pr
30 an important role for Tyr348 in coupling the alpha1B-adrenergic receptor to G protein and subsequent
31 Data suggest that protein kinase C modulates alpha1B-adrenergic receptor transfer to late endosomes a

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