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1 in for adenylyl cyclase, in complex with the alpha2 adrenergic receptor.
2 h Galphaz coupled strongly to the endogenous alpha2 adrenergic receptor.
3 K1/2 is inhibited by epinephrine through the alpha2-adrenergic receptor.
4 restin is required for ERK activation by the alpha2-adrenergic receptor.
5 inhibition of presynaptic TRPV1 channels by alpha2 adrenergic receptors.
6 y yohimbine, consistent with contribution of alpha2 adrenergic receptors.
7 irming that it was mediated by activation of alpha2 adrenergic receptors.
8 n is specific for post-junctional alpha1- or alpha2-adrenergic receptors.
9 ined whether this was specific to alpha1- or alpha2-adrenergic receptors.
10 ecline (PHFD) requires central activation of alpha2-adrenergic receptors.
12 te whether transdermal administration of the alpha2 adrenergic-receptor agonist, clonidine, can be us
14 ermal administration of the centrally acting alpha2-adrenergic receptor agonist, clonidine), and hypo
16 mpatholytic effects we observed suggest that alpha2 adrenergic-receptor agonists may offer a pharmaco
18 syndrome indicates that chronic exposure to alpha2-adrenergic receptor agonists produces strong depe
21 ers in cognition and emotion, by alpha1- and alpha2-adrenergic receptors (alpha1-ARs, alpha2-ARs) in
23 ole of arrestins in the trafficking of human alpha2-adrenergic receptors (alpha2-ARs) and the effect
24 the evolutionary precursor to the vertebrate alpha2-adrenergic receptors (alpha2-ARs) based upon sequ
25 the regulation of cell surface transport of alpha2-adrenergic receptors (alpha2-ARs) by GGA3 (Golgi-
26 strates subserving the inhibitory actions of alpha2-adrenergic receptors (alpha2-ARs) in the locus co
29 ve, and sympatholytic responses in vivo, and alpha2 adrenergic receptor (alpha2AR) agonists are used
34 ined the selectivity of interactions between alpha2-adrenergic receptors (alpha2R) and various combin
35 ng properties on I1 imidazoline receptors vs alpha2-adrenergic receptors and their blood pressure eff
36 sors are tested for the beta2-, alpha1-, and alpha2- adrenergic receptors, and adenosine type 1 recep
37 rolled by negative feedback from presynaptic alpha2-adrenergic receptors, and the targets of the rele
39 factor 1 (CRF1) antagonist (antalarmin), and alpha2-adrenergic receptor antagonist (yohimbine; used a
40 d PLD activity was completely blocked by the alpha2-adrenergic receptor antagonist rauwolscine and by
41 can be reversed by a potent centrally acting alpha2 adrenergic receptor (AR) agonist (dexmedetomidine
44 ediated via both post-junctional alpha1- and alpha2-adrenergic receptors are blunted in contracting h
46 was a result of activation of muscarinic and alpha2-adrenergic receptors because a specific alpha2-ad
47 ms using drug treatments targeting beta- and alpha2-adrenergic receptors, but little is known about t
48 that PHFD does not require the activation of alpha2-adrenergic receptors, but that alpha2-adrenergic
49 ion of alpha2-adrenergic receptors, but that alpha2-adrenergic receptors can modulate resting and pos
50 ctivity, which was increased by 5-HT(1A) and alpha2-adrenergic receptor co-activation in an activator
51 teract with a photoaffinity derivative of an alpha2-adrenergic receptor-derived peptide from the thir
52 voltage-dependent Ca2+ channels mediated by alpha2-adrenergic receptors desensitizes slowly with pro
53 vious studies in several animal species, the alpha2 adrenergic receptors in the human ciliary body, r
54 Stimulated GTPgammaS binding indicated that alpha2-adrenergic receptors in HET hippocampus were more
55 hile rolipram and PDE4D deficiency shortened alpha2 adrenergic receptor-mediated anesthesia, a surrog
56 eleased glutamate in the mNTS and results in alpha2 adrenergic receptor-mediated inhibition of DMV ne
58 ctions by approximately 50% and enhanced the alpha2-adrenergic receptor-mediated inhibition of forsko
59 btype influence signaling efficiency for the alpha2-adrenergic receptor-mediated signaling pathway.
60 e from studies in animals demonstrating that alpha2-adrenergic receptor-mediated vasoconstrictor resp
61 he level of arousal by selectively targeting alpha2 adrenergic receptors on LC neurons, resulting in
62 ic actions of norepinephrine are exerted via alpha2 adrenergic receptors residing on targets of norad
66 cular properties of each Gialpha, Gbeta, and alpha2-adrenergic receptor subtype influence signaling e
67 ecluded understanding the role of individual alpha2 adrenergic receptor subtypes in the antiepileptog
70 at low dose SNRIs, by acting on 5-HT(1A) and alpha2-adrenergic receptors, synergistically reduced AMP
71 Here, we show that noradrenaline activates alpha2-adrenergic receptors to control short-term and lo
72 lymorphisms (GPIIb, FcgammaRIIa, P-selectin, alpha2 adrenergic receptor, transforming growth factor [
73 a high binding affinity of M1 muscarinic and alpha2 adrenergic receptors was associated with a greate
76 ution and concentration of beta1, beta2, and alpha2 adrenergic receptors were examined in the frontal
77 mice to sedation elicited by stimulation of alpha2 adrenergic receptors, whereas arrestin 3 knockout
78 ne binding sites in plasma membrane and lack alpha2-adrenergic receptors, which recognize many I1-imi
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