ライフサイエンスコーパス: alpha2 receptor

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1 [D1/D2/D3/D4] and two adrenergic [alpha1 and alpha2] receptors).

2 d kinase) activity via adrenergic alpha1 and alpha2 receptors.

3 he generation of platelets that lack surface alpha2 receptors.

4 of this modulation was due to activation of alpha2 receptors.

5 ell as dopamine D2 and adrenergic alpha1 and alpha2 receptors.

6 n of the alpha1-receptor depolarized whereas alpha2-receptor activation hyperpolarized these neurons

7 nhibitor rapamycin as well as the adrenergic alpha2 receptor agonist clonidine.

8 noradrenergic-receptor antagonists, or of an alpha2-receptor agonist, into the bed nucleus of the str

9 Interestingly, the alpha2 receptor agonists had no effect on the high K(+)

10 se-dependent manner by brimonidine and other alpha2 receptor agonists, such as medetomidine.

11 Blockade of alpha1 but not of alpha2 receptors also reduced the numbers of S-phase nuc

12 d and assayed for their affinity at both the alpha2-receptor and the 5-HT uptake site.

13 istered either: (1) saline (sham) or (2) the alpha2-receptor antagonist, RX821002 HCl (2-[2-(2-Methox

14 detomidine was completely blocked by classic alpha2 receptor antagonists, such as yohimbine, rauwolsc

15 Inhibiting alpha1, but not alpha2, receptors blocked the evoked responses.

16 ol, one group of animals was pretreated with alpha2-receptor blocker, yohimbine, before injection of

17 ng compounds retaining high affinity for the alpha2-receptor but lacking affinity for the 5-HT uptake

18 apamycin along with activation of adrenergic alpha2 receptors by clonidine represents a double-hit to

19 1A but lower N-methyl-D-aspartate (NMDA) and alpha2 receptor densities.

20 ed distinctive ventral subdivisions; 2) high alpha2 receptor expression, which rendered distinctive a

21 entify ligand-dependent removal of activated alpha2 receptors from the cell surface as a mechanism by

22 indicating that nimodipine acts through the alpha2 receptor-G(alphai)-coupled pathway.

23 In contrast, blockade of noradrenergic-alpha2 receptors had no effect.

24 The levels of cerebellar alpha2 receptors in aggressive AD patients were slightly

25 sed (by approximately 70%) concentrations of alpha2 receptors in the cerebellar cortex compared with

26 nd observed to possess high affinity for the alpha2-receptor (K(i) = 6.71 nM) and the 5-HT uptake sit

27 We report that the alpha2 receptor-mediated inhibition of presynaptic Ca(2+

28 indings provide the first direct evidence of alpha2 receptor-mediated modulation of L-type Ca(2+) cha

29 The alpha2-receptor-mediated current in LSI neurons displaye

30 rat locus ceruleus prolonged the duration of alpha2-receptor-mediated IPSCs even when reuptake was in

31 s precludes definitive classification of the alpha2-receptor-mediated responses into different subtyp

32 t astrocytic laminin, by binding to integrin alpha2 receptor, prevents pericyte differentiation from

33 o distinguish between the alpha1, alpha2 and alpha2* receptor responses.

34 lcholine receptors; noradrenergic alpha1 and alpha2 receptors; serotonergic 5-HT1A receptors; dopamin

35 ha1-receptor or activation of the inhibitory alpha2-receptor significantly attenuated the analgesia i

36 chniques to compare the ability of the three alpha2-receptor subtypes to undergo agonist-mediated int

37 s in the inhibitory gamma-amino butyric acid alpha2 receptor subunit (GABRA2) gene.

38 ate analysis showed that GABARAP and GABA(A) alpha2 receptor subunit gene expression levels and GAP s

39 t this brimonidine effect is mediated by the alpha2 receptor through a reduction of intracellular cAM

40 s insulin secretion by modulating adrenergic alpha2 receptors through the mTOR pathway.

41 We found that alpha1- and alpha2-receptors were colocalized in the majority of a c

42 nt pooling of noradrenaline activated distal alpha2-receptors, which prolonged the duration of alpha2

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