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1 ntly higher than that for the complete human alpha7 nicotinic acetylcholine receptor.
2 nd as a negative allosteric modulator at the alpha7 nicotinic acetylcholine receptor.
3 mer's drug candidate, selectively stimulates alpha7 nicotinic acetylcholine receptors.
4 ve compounds targeting the cannabinoid 1 and alpha7 nicotinic acetylcholine receptors.
5 nt of cigarette smoke, and it stimulates the alpha7 nicotinic acetylcholine receptors.
6 renic acid, a natural antagonist of NMDA and alpha7-nicotinic acetylcholine receptors.
7 tion of the agonist JN403 with the human (h) alpha7 nicotinic acetylcholine receptor (AChR) was compa
8 the human (h) alpha4beta2, alpha3beta4, and alpha7 nicotinic acetylcholine receptors (AChRs) in diff
10 Here we studied whether GTS-21, a selective alpha7-nicotinic acetylcholine receptor agonist, inhibit
11 dings are of interest for the development of alpha7-nicotinic acetylcholine receptor agonists as a ne
12 line from the ligand-binding domain of human alpha7 nicotinic acetylcholine receptor along four diffe
13 ificant differences in their levels of brain alpha7 nicotinic acetylcholine receptor (alpha7 nAChR) e
14 duction of proinflammatory cytokines via the alpha7 nicotinic acetylcholine receptor (alpha7 nAChR) e
17 id-beta (Abeta) accumulation in BMEC through Alpha7 nicotinic acetylcholine receptor (alpha7 nAChR).
21 roliferation in normal and cancer cells, the alpha7 nicotinic acetylcholine receptor (alpha7-nAChR),
22 ished by alpha-bungarotoxin, an inhibitor of alpha7 nicotinic acetylcholine receptors (alpha7 AChRs).
24 lease of acetylcholine, which interacts with alpha7 nicotinic acetylcholine receptors (alpha7 nAChR)
26 dicated that the columns contained homomeric alpha7 nicotinic acetylcholine receptors (alpha7 nAChR)
28 ophrenia, where there are genetic insults in alpha7 nicotinic acetylcholine receptors (alpha7-nAChRs)
29 , promotes lung cancer proliferation via the alpha7-nicotinic acetylcholine receptor (alpha7-nAChR) s
30 ral arteries that stimulation by nicotine of alpha7-nicotinic acetylcholine receptors (alpha7-nAChRs)
31 high binding affinities and selectivity for alpha7-nicotinic acetylcholine receptors (alpha7-nAChRs)
35 tested the hypothesis that activation of the alpha7 nicotinic acetylcholine receptor (alpha7nAChR) pa
36 myloid beta(1-42) (Abeta(1-42)) binds to the alpha7 nicotinic acetylcholine receptor (alpha7nAChR) wi
37 nd a neuronal pentameric cation channel, the alpha7 nicotinic acetylcholine receptor (alpha7nAChR), a
38 hat VSMCs in brain blood vessels express the alpha7 nicotinic acetylcholine receptor (alpha7nAChR), w
39 ring lethal systemic inflammation through an alpha7 nicotinic acetylcholine receptor (alpha7nAChR)-de
40 phrine and re-establishes neuromodulation in alpha7 nicotinic acetylcholine receptor (alpha7nAChR)-de
41 anti-inflammatory pathway' that requires the alpha7 nicotinic acetylcholine receptor (alpha7nAChR).
42 at auditory cortex through activation of the alpha7 nicotinic acetylcholine receptor (alpha7nAChR).
43 on produced anti-shock effect via activating alpha7 nicotinic acetylcholine receptor (alpha7nAChR).
44 , injury and ischemia through stimulation of alpha7 nicotinic acetylcholine receptors (alpha7nAChR) o
45 linergic anti-inflammatory pathway (CAP) and alpha7 nicotinic acetylcholine receptors (alpha7nAChR).
46 tains a unique, distinct, and human-specific alpha7-nicotinic acetylcholine receptor (alpha7nAChR) ge
47 ity of septal cholinergic fibers through the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), w
49 aling of amyloid-beta(42) (Abeta(42)) by the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), w
50 ruption that resulted from local blockade of alpha7-nicotinic acetylcholine receptors (alpha7nAChR).
51 itates the integration of neural signals and alpha7 nicotinic acetylcholine receptors (alpha7nAChRs)
52 ress synaptic activities by interacting with alpha7 nicotinic acetylcholine receptors (alpha7nAChRs).
53 eatment may, by increasing activation of the alpha7-nicotinic acetylcholine receptor, alter the devel
54 that beta-amyloid peptide can interact with alpha7 nicotinic acetylcholine receptors, although the n
55 ra of the extracellular domain of the native alpha7 nicotinic acetylcholine receptor and acetylcholin
56 Kynurenic acid (KYNA), an antagonist of the alpha7 nicotinic acetylcholine receptor and the N-methyl
60 or (alpha7nAChR) gene [CHRNA7 (gene-encoding alpha7-nicotinic acetylcholine receptor)] called CHRFAM7
61 Positive allosteric modulators (PAMs) of alpha7 nicotinic acetylcholine receptors can enhance ion
62 ography (CMAC) columns containing functional alpha7 nicotinic acetylcholine receptors, CMAC(alpha7 nA
63 matosensory pathway regulates the density of alpha7 nicotinic acetylcholine receptors during the firs
65 equiring acetylcholine signaling through the alpha7 nicotinic acetylcholine receptor expressed on cyt
66 n activation of antiinflammatory effects via alpha7 nicotinic acetylcholine receptor-expressing splen
67 uced inward currents in cultured SCG and the alpha7-nicotinic acetylcholine receptor-expressing Xenop
68 These results suggest that the increase in alpha7 nicotinic acetylcholine receptor expression in th
69 ations have previously been shown to convert alpha7 nicotinic acetylcholine receptors from cationic t
70 utative candidate genes in this area are the alpha7 nicotinic acetylcholine receptor gene (CHRNA7) an
75 [(125)I]alpha-bungarotoxin (btx) binding to alpha7 nicotinic acetylcholine receptors in normal and 1
77 ts confirm the importance of alpha4beta2 and alpha7 nicotinic acetylcholine receptors in the dorsal h
78 ct evidence for linkage between JAK2 and the alpha7 nicotinic acetylcholine receptor-induced neuropro
80 se and pattern of development of hippocampal alpha7 nicotinic acetylcholine receptors is discernibly
81 pecific subtype of this receptor family, the alpha7 nicotinic acetylcholine receptor, is thought to b
82 agus nerve inhibits cytokine release through alpha7-nicotinic acetylcholine receptor-mediated choline
83 these surface expression determinants in the alpha7 nicotinic acetylcholine receptor might influence
84 trong positive allosteric modulator (PAM) of alpha7 nicotinic acetylcholine receptor (nAChR) activati
85 nctional impact of hydrogen bonding on human alpha7 nicotinic acetylcholine receptor (nAChR) activati
87 acute systemic administration of the partial alpha7 nicotinic acetylcholine receptor (nAChR) agonist
88 selectivity in models of the cationic human alpha7 nicotinic acetylcholine receptor (nAChR) and the
89 The effect of nicotine was mimicked by the alpha7 nicotinic acetylcholine receptor (nAChR) antagoni
92 ences and yet are shown to inhibit the human alpha7 nicotinic acetylcholine receptor (nAChR) by targe
95 sts that promote a desensitized state of the alpha7 nicotinic acetylcholine receptor (nAChR) have bee
99 re-activity relationships for a new class of alpha7 nicotinic acetylcholine receptor (nAChR) modulato
103 ased proteasome-dependent degradation of the alpha7 nicotinic acetylcholine receptor (nAChR) subunit,
104 mizygous for CHRNA7, the gene coding for the alpha7 nicotinic acetylcholine receptor (nAChR), and man
106 mer does not affect the activity against the alpha7 nicotinic acetylcholine receptor (nAChR), whereas
108 ar acceleration of DP was observed using the alpha7 nicotinic acetylcholine receptor (nAChR)-selectiv
112 t endogenous kynurenic acid (KYNA) modulates alpha7* nicotinic acetylcholine receptor (nAChR) and NMD
113 ssess whether extending plasma levels of the alpha7-nicotinic acetylcholine receptor (nAChR) agonist
117 A subset of cold thermosensors expressed alpha7 nicotinic acetylcholine receptors (nAChRs) but no
118 ompounds were tested for their inhibition of alpha7 nicotinic acetylcholine receptors (nAChRs) expres
119 representative agonists with alpha4beta2 and alpha7 nicotinic acetylcholine receptors (nAChRs) has be
122 e individuals have significant reductions in alpha7 nicotinic acetylcholine receptors (nAChRs) in sev
127 t the induction of this form of LTP needs CP-alpha7 nicotinic acetylcholine receptors (nAChRs) that,
128 iated, in large part, via desensitization of alpha7 nicotinic acetylcholine receptors (nAChRs), as an
129 II positive allosteric modulator (PAM-II) of alpha7 nicotinic acetylcholine receptors (nAChRs), as lo
130 evaluated for binding to the alpha4beta2 and alpha7 nicotinic acetylcholine receptors (nAChRs), for p
131 the protein acts as a powerful inhibitor of alpha7 nicotinic acetylcholine receptors (nAChRs), which
139 p 9) is a conserved glutamate [E172 in chick alpha7 nicotinic acetylcholine receptors (nAChRs)] that
142 proving survival in animal sepsis models via alpha7 nicotinic acetylcholine receptors on immunocompet
143 postnatal mouse demonstrating high levels of alpha7 nicotinic acetylcholine receptors on layer IV som
144 her immune cells, e.g. through activation of alpha7 nicotinic acetylcholine receptors on macrophages.
145 eceptor)] called CHRFAM7A (gene-encoding dup-alpha7-nicotinic acetylcholine receptor) on a locus of c
146 dines ("spiroimidates") and their utility as alpha7 nicotinic acetylcholine receptor partial agonists
147 gy by Maouche et al, which demonstrates that alpha7 nicotinic acetylcholine receptors play a key role
149 evidence that pharmacological activation of alpha7 nicotinic acetylcholine receptors provide signifi
156 14, which contains a strong candidate in the alpha7-nicotinic acetylcholine receptor subunit gene (CH
157 not been a successful method to express the alpha7 nicotinic acetylcholine receptor such that these
159 1), is conserved in all 5-HT3A receptors and alpha7-nicotinic acetylcholine receptors that have been
160 a specific subtype of neuron, expressing non-alpha7 nicotinic acetylcholine receptors, that directly
161 r, treatment with a selective agonist of the alpha7 nicotinic acetylcholine receptor, the primary rec
162 ow that amyloid-beta peptide (Abeta) engages alpha7 nicotinic acetylcholine receptors to induce relea
163 al dynamics of a homology model of the human alpha7 nicotinic acetylcholine receptor using molecular
164 ssion of the anti-inflammatory alpha7-nAChR (alpha7-nicotinic acetylcholine receptor) was similar in
165 st whether beta-amyloid peptide can activate alpha7 nicotinic acetylcholine receptors, we expressed t
166 ditionally, PrP(C)-STI1 engagement activated alpha7 nicotinic acetylcholine receptors, which particip
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