ライフサイエンスコーパス: alveolar bone

1 of root cementum, periodontal ligament, and alveolar bone).

2 zed bone structure was the main character of alveolar bone.

3 mice were subjected to mechanical loading in alveolar bone.

4 n and thus may exhibit a favorable effect on alveolar bone.

5 acial mesenchymal cells that form dentin and alveolar bone.

6 ncluding periodontal ligament, cementum, and alveolar bone.

7 l unit with its surrounding bony socket, the alveolar bone.

8 llular cementum, and osteoid accumulation in alveolar bone.

9 leads to the resorption of tooth-supporting alveolar bone.

10 hat anchors the cementum of the teeth to the alveolar bone.

11 -supporting tissue and the resorption of the alveolar bone.

12 o solution for the long-term preservation of alveolar bone.

13 nt acid phosphatase (TRAP)-positive cells in alveolar bone.

14 on (HFA) has an osteogenic effect on healthy alveolar bone.

15 correlation between age and microhardness of alveolar bone (0.7 +/- 0.1 to 0.9 +/- 0.2 GPa) and cemen

16 However, it is not known if HFA can preserve alveolar bone after extraction without negatively affect

17 er additional benefit in the preservation of alveolar bone after the extraction of molar teeth.

18 the periodontal diagnostic acumen regarding alveolar bone alterations influenced by orthodontic toot

19 t dental pulp (dental pulp cells [DPCs]) and alveolar bone (alveolar bone cells [ABCs]) were isolated

20 The quality of alveolar bone and attachment loss (AL) were measured by

21 reactivity, as well as a greater decrease in alveolar bone and attachment loss and MMP-9 immunoreacti

22 ts the role of PHOSPHO1 in mineralization of alveolar bone and cellular cementum, further revealing t

23 ficant protective effect was not observed on alveolar bone and collagen tissue in this model.

24 e that is characterized by resorption of the alveolar bone and mediated by commensal bacteria that tr

25 on of antibiotics significantly improved the alveolar bone and PDL damage of the knockdown phenotype,

26 th that will progressively cause the loss of alveolar bone and periodontal ligaments and eventually t

27 disease that degrades connective tissue and alveolar bone and results in tooth loss.

28 is vital for maintenance and regeneration of alveolar bone and supporting structures around teeth and

29 -pigs suggest that PTG may integrate well in alveolar bone and supports osseous regrowth in degree II

30 ding the teeth that lead to the breakdown of alveolar bone and tooth loss.

31 o periodontal ligament detachment, extensive alveolar bone and tooth root resorption, and incisor mal

32 P in the normal development of the calvaria, alveolar bone, and dentin-pulp complex.

33 es followed by periodontitis, destruction of alveolar bone, and loss of primary and permanent teeth.

34 inging attention to direct effects of HPP on alveolar bone, and offering a new model for testing pote

35 Heterogeneous distribution of Ca and P in alveolar bone, and relatively lower contents at the enth

36 ar cementum, periodontal ligament (PDL), and alveolar bone, are critical for tooth function.

37 There was a greater increase in alveolar bone area and VEGF immunoreactivity, as well as

38 Histomorphometric analysis, which included alveolar bone area, alveolar bone level, and attachment

39 eolar bone crest and the thickness of facial alveolar bone at points 1 to 5 mm from the bone crest fo

40 cal stimulation contributes to the health of alveolar bone, but no therapy using the osteogenic effec

41 olume in the extraction site and surrounding alveolar bone by 44% when compared with static, while fu

42 dental pulp cells [DPCs]) and alveolar bone (alveolar bone cells [ABCs]) were isolated and separately

43 pact of genetic background on comorbidity of alveolar bone change and glucose tolerance after HFD con

44 Interleukin-6 significantly correlated with alveolar bone changes (P <0.05), whereas adipsin showed

45 Alveolar bone changes significantly varied among CC line

46 nd IL-10, serum B-ALP and TRAP-5b levels, or alveolar bone compared with conventional periodontal the

47 ce from cemento-enamel junction (CEJ) to the alveolar bone crest (ABC) at 20 molar sites.

48 etween the cemento-enamel junction (CEJ) and alveolar bone crest and the thickness of facial alveolar

49 nces between the cemento-enamel junction and alveolar bone crest were evaluated.

50 orptive lesions, osteoid accumulation on the alveolar bone crest, and significant differences in seve

51 location of the microgap with respect to the alveolar bone crest, occlusion, and use of a polished co

52 l status, i.e., alveolar bone loss (ABL) and alveolar bone crest, was examined by stereomicroscopy an

53 ed with periodontal disease that can lead to alveolar bone damage and resorption, promoting tooth los

54 nic expression of DSPP rescued the tooth and alveolar bone defects of the Dmp1 KO mice.

55 20C(fl/fl) mice showed remarkable dentin and alveolar bone defects, while their enamel did not show a

56 le methods to stimulate bone regeneration in alveolar bone defects.

57 Alveolar bone deficiency is a major clinical problem in

58 Posterior vertical bitewings were taken for alveolar bone density (ABD) and alveolar bone height (AB

59 Alendronate (ALN) increases alveolar bone density with systemic use and, has been fo

60 ally treated root canal with periodontal and alveolar bone-derived cells.

61 murine oral cavity and to prevent subsequent alveolar bone destruction and osteoclastogenesis.

62 s did not protect from or exacerbate crestal alveolar bone destruction but were responsible for promo

63 Th17 cells are redundant in contributing to alveolar bone destruction in a murine model of periodont

64 he pathogenesis of periodontitis with severe alveolar bone destruction.

65 sease is characterized by destruction of the alveolar bone due to an aberrant host inflammatory respo

66 nd patterns of chronicity leading to loss of alveolar bone due to inflammation in Rac-null mice.

67 bable, and 3-dimensionally printed) used for alveolar bone engineering around teeth and implants and

68 In many instances, surrounding alveolar bone extended into the existing resorptive defe

69 etter results on periodontium with regard to alveolar bone findings.

70 ative imaging modality, DI length, available alveolar bone for DI placement, placement site, timing o

71 therapy that may play a significant role in alveolar bone formation and maintenance.

72 tatin (RSV), are known to be associated with alveolar bone formation and periodontal improvements.

73 eogenic effects of these stimuli to increase alveolar bone formation has been developed.

74 (recently shown to play key roles in normal alveolar bone formation), significant loss in alveolar b

75 of the key contribution of the PDL in normal alveolar bone formation, the pathologic changes of the O

76 and osteoclast activity, and an increase in alveolar bone formation.

77 equency acceleration significantly increased alveolar bone formation.

78 Subjective radiographic classifications of alveolar bone have been proposed and correlated with imp

79 re taken for alveolar bone density (ABD) and alveolar bone height (ABH) measurements.

80 istically significant difference in residual alveolar bone height (P <0.001).

81 first (M1) and second (M2) molars: relative alveolar bone height (RBH), crestal bone width (CBW), bo

82 Both cKO models exhibited reduced alveolar bone height and 4-fold increased numbers of ost

83 matory reaction corresponded to reduction in alveolar bone height and density (r = 0.74; P <0.05; Spe

84 compared with static, while fully preserving alveolar bone height and width long-term.

85 Prior reports that alveolar bone height is significantly less in normal SPF

86 When characterizing ENAM(-/-) mice, alveolar bone height reduction was observed, and it was

87 Alveolar bone height was measured from orthopantomograms

88 hy to generalized disease categorized by the alveolar bone height-to-tooth length (AB/T) ratio were s

89 isms in response to the normal oral flora on alveolar bone height.

90 tors showed significant correlation with the alveolar bone height.

91 ibution of mucosal Langerhans cells (LCs) to alveolar bone homeostasis in mice following oral coloniz

92 o the normal oral flora, mediating catabolic alveolar bone homeostasis in the healthy periodontium.

93 gated the histologic changes of cementum and alveolar bone in a pycnodysostosis patient, caused by no

94 tal ligament (PDL) and altered remodeling of alveolar bone in dKO mice.

95 g protein osteopontin (OPN) was increased in alveolar bone in Phospho1(-/-) mice.

96 f the interface between the root surface and alveolar bone in the replantation/transplantation model,

97 increased the expression of IL-33 and ST2 in alveolar bone in vivo and in osteoblastic cells in vitro

98 pl(+/A116T) mice featured alterations in the alveolar bone, including radiolucencies and resorptive l

99 Furthermore, destruction of crestal alveolar bone induced by P. gingivalis colonization occu

100 The growth of the tooth and alveolar bone is co-ordinated so that a studied distance

101 ament (PDL), which connects the teeth to the alveolar bone, is essential for periodontal tissue homeo

102 f the M1 from the surrounding mesenchyme and alveolar bone leads to an expansion of the tooth germ, d

103 luded evaluation of attachment loss (AL) and alveolar bone level (ABL) on the distal root of the mand

104 d not demonstrate significant differences in alveolar bone level compared to EP (P >0.05).

105 bing depth </=4 mm, and a lower radiographic alveolar bone level than individuals without psoriasis (

106 analysis, which included alveolar bone area, alveolar bone level, and attachment loss, and immunohist

107 Alveolar bone level, serum antibody, and lymphocyte resp

108 eriodontal disease often result in decreased alveolar bone levels and a loss of connective tissue hom

109 10, serum B-ALP and TRAP-5b, and calcium and alveolar bone levels between the groups receiving SRP an

110 However, drug administration did not affect alveolar bone levels during the study period.

111 id phosphatase 5b (TRAP-5b), and calcium and alveolar bone levels in rats with experimentally induced

112 Alveolar bone levels in the periodontitis group were sig

113 Differences in alveolar bone levels were no longer significant, particu

114 Probing depths (PDs), tooth loss, alveolar bone levels, and systemic health were studied a

115 olved, and there was progressive loss of the alveolar bone, likely as a result of increased colonizat

116 miRNAs direct periodontal fibroblasts toward alveolar bone lineage differentiation and new bone forma

117 re, and periodontal bone loss was defined as alveolar bone loss >/=3 mm on >/=1 permanent tooth site

118 ttachment loss >/=5 mm (1.19; 1.00 to 1.41), alveolar bone loss >/=40% (1.25; 1.00 to 1.56), and toot

119 nt loss (>/=5 mm), mobility (>/=0.5 mm), and alveolar bone loss (>/=40% of the distance from the ceme

120 Periodontal status, i.e., alveolar bone loss (ABL) and alveolar bone crest, was ex

121 Higher linear alveolar bone loss (ABL) and lower interradicular bone d

122 effects of a 2% cholesterol-enriched diet on alveolar bone loss (ABL) and serum levels of pro-oxidant

123 depth (PD), myeloperoxidase (MPO) activity, alveolar bone loss (ABL) for periodontal tissues; histop

124 related orphan receptor (ROR) gammat; and 3) alveolar bone loss (ABL) in experimental periodontitis.

125 nt on serum oxidative stress index (OSI) and alveolar bone loss (ABL) in rats with diabetes mellitus

126 tigate effects of strontium ranelate (SR) on alveolar bone loss (ABL) in rats with experimental perio

127 diet-induced obesity/hyperlipidemia (CAF) on alveolar bone loss (ABL) in rats.

128 se tolerance development are associated with alveolar bone loss (ABL) in susceptible individuals.

129 es) demonstrated that EP-TIL1 presented less alveolar bone loss (ABL) than EP (P <0.05), whereas EP-T

130 This study examines: 1) alveolar bone loss (ABL), a hallmark of periodontitis, i

131 proinflammatory cytokine levels, apoptosis, alveolar bone loss (ABL), lipid metabolism, and diabetic

132 (PD), bleeding on probing, and radiographic alveolar bone loss (ABL), measured on intraoral periapic

133 f bleeding on probing (BOP) and radiographic alveolar bone loss (ABL).

134 treatment was accompanied by lower rates of alveolar bone loss (P <0.05) and maintenance of the amou

135 Rats with PD exhibited increased alveolar bone loss (P <0.05), as well as increased level

136 with AMD had fewer teeth (P <0.001) and more alveolar bone loss (P = 0.004) compared with non-AMD par

137 Alveolar bone loss among the groups was estimated by mea

138 , and osteopontin as potential biomarkers of alveolar bone loss and 2) determine whether the glycemic

139 ed with DTrp(8)-gammaMSH presented decreased alveolar bone loss and a lower degree of neutrophil infi

140 fective in the stabilization or reduction of alveolar bone loss and collagen degradation in rats.

141 s fractured molar roots, distorted incisors, alveolar bone loss and compressed temporomandibular join

142 s HN019 promotes a protective effect against alveolar bone loss and CTALs attributable to EP in rats,

143 Positive correlations were found between alveolar bone loss and density of inflammation (rho = 0.

144 rated that simvastatin inhibited LPS-induced alveolar bone loss and periodontal tissue inflammation i

145 jection of anti-DC-STAMP-mAb also suppressed alveolar bone loss and reduced the total number of multi

146 stomorphometric analyses confirmed increased alveolar bone loss and revealed increased numbers of TRA

147 ice infected with P. gingivalis demonstrated alveolar bone loss and serum anti-P. gingivalis antibody

148 rformed to study the association of AMD with alveolar bone loss and the number of teeth by controllin

149 s study was to evaluate the effect of HFA on alveolar bone loss and the rate of bone formation after

150 classification of disease severity based on alveolar bone loss and tooth loss during follow-up.

151 e safe treatment that can be used to prevent alveolar bone loss and/or accelerate bone healing after

152 nship between this biochemical parameter and alveolar bone loss around natural teeth and dental impla

153 dental disease which results in irreversible alveolar bone loss around teeth, and subsequent tooth lo

154 Using a model involving inflammation-driven alveolar bone loss attributable to infection, we showed

155 severity of periodontitis for premolars with alveolar bone loss based on 3D's or 2D's measurement is

156 Alveolar bone loss can be a major clinical concern affec

157 Mixed infection with capsulated Pg augmented alveolar bone loss compared with that of mixed infection

158 Self-report questions related to alveolar bone loss exhibit excellent convergent validity

159 F-deficient (Tnf(-/-)) mice are resistant to alveolar bone loss following oral infection with P. ging

160 eria, and neutralizing TNF in vivo abrogated alveolar bone loss following P. gingivalis infection.

161 protection takes place in infection-induced alveolar bone loss has not been investigated.

162 is being required for the pathogen to induce alveolar bone loss in a model of periodontitis and revea

163 timulate the host immune response and induce alveolar bone loss in a murine experimental periodontiti

164 specific elevated fatty acid (FA) levels on alveolar bone loss in a Porphyromonas gingivalis-induced

165 with increased periodontal inflammation and alveolar bone loss in an LPS-induced periodontitis anima

166 d TIL solution (1 mg/kg body weight) reduced alveolar bone loss in experimental periodontitis and the

167 We hypothesized that SOCS-3 regulates alveolar bone loss in experimental periodontitis.

168 e SOCS-3 as a critical negative regulator of alveolar bone loss in periodontitis.

169 9 (MMP-9), interleukin-1beta (IL-1beta), and alveolar bone loss in rats with diabetes.

170 that PROB supplementation 1) reduces AL and alveolar bone loss in rats with LIP and 2) can protect t

171 ting the up-regulated osteoclastogenesis and alveolar bone loss in SPF mice compared with GF mice.

172 e immune response contributes to physiologic alveolar bone loss in the healthy periodontium.

173 nly the ligature model displayed significant alveolar bone loss in the initial period (7 days), which

174 PT reduced alveolar bone loss in unstressed animals.

175 t TLR2 is required for P. gingivalis-induced alveolar bone loss in vivo, and our in vitro work implic

176 tored the ability of P. gingivalis to induce alveolar bone loss in vivo.

177 rtin agonism as a viable strategy to control alveolar bone loss induced by oral infection.

178 ounterparts suggest that naturally occurring alveolar bone loss is a normal component of healthy peri

179 Alveolar bone loss is a result of an aggressive form of

180 In this population-based health survey, alveolar bone loss is independently associated with AMD

181 icate that Porphyromonas gingivalis mediates alveolar bone loss or osteoclast modulation through enga

182 Alveolar bone loss resulting from LPS-induced periodonti

183 ed with heat-killed Pg displayed significant alveolar bone loss starting from day 15, which continued

184 TLR9(-/-) mice exhibited significantly less alveolar bone loss than their wild-type (WT) counterpart

185 ence of inflammation, it was the presence of alveolar bone loss that lead to significantly higher val

186 d clinical measures of inflammation and less alveolar bone loss under severe inflammatory conditions

187 Alveolar bone loss was alleviated in JQ1-treated mice be

188 Alveolar bone loss was also evaluated radiographically i

189 del adjusted for age, smoking, and diabetes, alveolar bone loss was associated with AMD in males with

190 Conversely, P. gingivalis infection-induced alveolar bone loss was attenuated in mice lacking ST2.

191 Alveolar bone loss was determined by macroscopic and his

192 After 4 wk of treatment, alveolar bone loss was determined by micro-computed tomo

193 Ligature-induced alveolar bone loss was diminished in chemR23tg mice.

194 Alveolar bone loss was evaluated morphometrically under

195 The alveolar bone loss was evaluated using microcomputed tom

196 Greater radiographic alveolar bone loss was observed among participants repor

197 Compared to the ligature + placebo group, alveolar bone loss was reduced in the fluoxetine group (

198 ntages of fat (P = nonsignificant); however, alveolar bone loss was significantly greater in animals

199 Alveolar bone loss was significantly greater in groups 2

200 Alveolar bone loss was significantly higher in the PED g

201 onstrated that group EP/EA presented reduced alveolar bone loss when compared to group EP (P <0.05).

202 alis-infected mice significantly exacerbated alveolar bone loss when compared with infection or IL-33

203 Treatment with simvastatin improved alveolar bone loss within all of the parameters studied,

204 ge, sex, smoking, diabetes, body mass index, alveolar bone loss, and number of teeth), having WPSs as

205 Periodontitis, alveolar bone loss, and tooth loss are associated with l

206 ion with the two species induces synergistic alveolar bone loss, characterized by bone loss which is

207 a reduction of serum inflammatory cytokines, alveolar bone loss, cholesterol, and atherosclerotic les

208 ificant increases in inflammatory cytokines, alveolar bone loss, cholesterol, and atherosclerotic les

209 ot 500 nmol caused significant inhibition of alveolar bone loss, increase of bone alkaline phosphatas

210 Postextraction alveolar bone loss, mostly affecting the buccal plate, o

211 nd that a HFD markedly increased LPS-induced alveolar bone loss, osteoclastogenesis, and inflammatory

212 argeting oral bacteria protect the host from alveolar bone loss, recent studies show that particular

213 )) were protected from P. gingivalis-induced alveolar bone loss, with a reduction in anti-P. gingival

214 ceptible to A. actinomycetemcomitans-induced alveolar bone loss, with different patterns of immune re

215 nt acid phosphatase-positive (TRAP+) OCs and alveolar bone loss.

216 ces LPS-induced periodontal inflammation and alveolar bone loss.

217 efense responses to oral bacteria can induce alveolar bone loss.

218 ostimulatory activity, which is critical for alveolar bone loss.

219 result in irreversible inflammation-mediated alveolar bone loss.

220 t against pathobionts, but also by promoting alveolar bone loss.

221 recently to stimulate osteoblasts and reduce alveolar bone loss.

222 eter for monitoring periodontal/peri-implant alveolar bone loss.

223 hich is characterized by inflammation-driven alveolar bone loss.

224 s that are able to modulate inflammation and alveolar bone loss.

225 bone metabolism and can therefore influence alveolar bone loss.

226 ith P. gingivalis (W50) or placebo to induce alveolar bone loss.

227 STAMP-mAb downregulated the ligature-induced alveolar bone loss.

228 teinase 9 (Mmp9) in the gingiva; support and alveolar bone loss; connective tissue attachment; and th

229 bone loss which is greater than the additive alveolar bone losses induced by each species alone.

230 We harvested 103 alveolar bone marrow samples from 45 patients using 1 of

231 he isolation and clinical-scale expansion of alveolar bone marrow-derived MSCs (aBMSCs).

232 lveolar bone formation), significant loss in alveolar bone mass ( P < 0.01), and a sharp reduction in

233 and ultrastructural changes of cementum and alveolar bone might be affected by CTSK mutation via red

234 Alveolar bone mineral density and alveolar bone volume w

235 Concomitantly, alveolar bone mineral density was significantly lower in

236 Despite alveolar bone mineralization defects, periodontal attach

237 Phospho1(-/-) mice featured disturbances in alveolar bone mineralization, shown by accumulation of u

238 al techniques to characterize the dentin and alveolar bone of Dmp1 KO/DSPP Tg mice compared with Dmp1

239 ion of DMP1 in vivo in cellular cementum and alveolar bone of mice treated with a single dose (50 mic

240 of osteoclasts, especially noted around the alveolar bone of molars (buccal side) and incisors.

241 rimary human osteoblasts were retrieved from alveolar bone of patients undergoing oral surgery.

242 The alveolar bone of the maxilla was evaluated by microcompu

243 nt, focusing on the impact of the developing alveolar bone on the development of the mouse first mola

244 nd PHOSPHO1 protein were expressed by active alveolar bone osteoblasts and cementoblasts during cellu

245 function resulted in an increased number of alveolar bone osteoclasts and increased RANKL expression

246 ppeared to reflect changes in the underlying alveolar bone over time.

247 ppeared to reflect changes in the underlying alveolar bone over time.

248 Mean thickness of alveolar bone ranged from 6.66 to 4.51 mm (standard devi

249 fficiently induces new bone formation in the alveolar bone regeneration model.

250 ens and their contribution to destruction of alveolar bone remain unknown.

251 maintaining proper periodontal function and alveolar bone remodeling and point to dental dysfunction

252 tomographic investigation is to analyze the alveolar bone remodeling around immediate implants place

253 Alveolar bone repair was evaluated histomorphometrically

254 In rat alveolar bone repair, meloxicam did not affect VEGF expr

255 oinflammatory cytokines and its influence on alveolar bone resorption (ABR) in rats.

256 in treatment significantly inhibits regional alveolar bone resorption and contributes to periodontal

257 On day 14, there were no differences in alveolar bone resorption and gingival RANKL expression b

258 Alveolar bone resorption and myeloperoxidase activity we

259 rovides therapeutic effects on inhibition of alveolar bone resorption and periodontal tissue destruct

260 epletion reduced P. gingivalis infection and alveolar bone resorption by modulating the host immune r

261 ects of systemic melatonin administration on alveolar bone resorption in experimental periodontitis i

262 ALA and Vit-C substances in the treatment of alveolar bone resorption in periodontal diseases.

263 nfected mice had higher levels of horizontal alveolar bone resorption than sham-infected mice and an

264 phyromonas gingivalis resulted in infection, alveolar bone resorption, and a significant increase in

265 ulatory role for LCs in inflammation-induced alveolar bone resorption, by inhibiting IFN-gamma secret

266 Bsp (-/-) mice displayed extensive root and alveolar bone resorption, mediated by increased RANKL an

267 al epithelium, periodontal pocket formation, alveolar bone resorption, osteoclast activation, bacteri

268 levated levels of MMP-13 are associated with alveolar bone resorption, periodontal ligament breakdown

269 mine the roles of TLR2 and TLR4 signaling in alveolar bone resorption, using a Porphyromonas gingival

270 duced periodontitis in mice, as evidenced by alveolar bone resorption.

271 tory M1 macrophages into gingival tissue and alveolar bone resorption.

272 s significantly correlated with the level of alveolar bone resorption.

273 o in gingival connective tissue and reducing alveolar bone resorption.

274 mmatory cytokines, and a marked reduction in alveolar bone resorption.

275 esponse plays a key role in bacteria-induced alveolar bone resorption.

276 ted tomography (micro-CT) was used to assess alveolar bone structure and tissue compositions.

277 Ridge preservation can minimize the loss of alveolar bone subsequent to tooth extraction in preparat

278 ents have fewer teeth, greater CAL, and less alveolar bone support compared with controls after adjus

279 mbers of TRAP+ osteoclastic cells lining the alveolar bone surface in SPF compared with GF mice.

280 nt acid phosphatase-positive cells along the alveolar bone surface was significantly decreased after

281 osteocalcin expression were described on the alveolar bone surfaces in etidronate-treated rats, with

282 g the positive or deleterious changes on the alveolar bone surrounding natural teeth undergoing ortho

283 thought to contribute to the destruction of alveolar bone surrounding teeth by influencing osteoclas

284 ition affecting tooth-supporting tissues and alveolar bone that surround the tooth, leading to format

285 he rodent incisor apex, the dental pulp, the alveolar bone, the periodontal ligament, the cementum, a

286 Changes in the alveolar bone thickness and height around natural teeth

287 The "3-mm rule" has dictated the amount of alveolar bone to be removed during CL surgery for decade

288 nt between the responses of human and rodent alveolar bone to osteotomy site preparation.

289 1(phox) KO mice revealed significant loss of alveolar bone volume and enhanced inflammatory cell infi

290 mellitus (t2DM) development and significant alveolar bone volume change (P <0.05), whereas others sh

291 there was no significant correlation between alveolar bone volume changes and increased BW or glucose

292 es were quantified by multiplex immunoassay, alveolar bone volume was quantified by microcomputed tom

293 Alveolar bone mineral density and alveolar bone volume were quantified by three-dimensiona

294 points after osteotomy, the fate of the dead alveolar bone was followed.

295 d ameloblast defects, while their dentin and alveolar bone were not significantly affected.

296 edic treatment on periodontal tissues (i.e., alveolar bone) were included.

297 sive destruction of gingival soft tissue and alveolar bone, which is initiated by inflammation in res

298 s to gingival tissues and osteoclasts to the alveolar bone, which mediate tissue and bone destruction

299 t to the alveolar crest (vertical distance), alveolar bone width (bone width) between adjacent implan

300 allenge, Rac-null mice had increased loss of alveolar bone with patterns of resorption characteristic