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1 veolar pressure/area curves, plus changes in alveolar duct and airway size, and the elastic forces in
2 al cells lining the terminal bronchioles and alveolar ducts as well as macrophages and alveolar type
3 mice showed prominent lesions at bronchiolar-alveolar duct (BAD) junctions where asbestos fibers depo
4 t the number of proliferating cells at first alveolar duct bifurcations and in adjacent terminal bron
5                 Staining was most evident at alveolar duct bifurcations and within bronchiolar epithe
6 1 (SFRP1) is a critical molecule involved in alveolar duct formation in the lung and here we demonstr
7  fibers deposit initially at the bronchiolar-alveolar duct regions and alveolar macrophages accumulat
8 were clearly up-regulated at the bronchiolar-alveolar duct regions by 24 hours after the single asbes
9 ithelial cells lining distal bronchioles and alveolar ducts, sites of initial lung deposition of asbe

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