ライフサイエンスコーパス: alveolar epithelial cell

1 ical stretch (A549, Calu-3, or human primary alveolar epithelial cells).

2 eptor (CNTFR), was expressed only in type II alveolar epithelial cells.

3 rally expressed Na(+)/K(+)-ATPase in type II alveolar epithelial cells.

4 y studied by transfecting the human MMP19 in alveolar epithelial cells.

5 localization predominantly in the nucleus of alveolar epithelial cells.

6 2, in both A549 cells and in primary type II alveolar epithelial cells.

7 an increase in ER stress of resident type II alveolar epithelial cells.

8 for hypoxia-induced AMPK-PKC zeta binding in alveolar epithelial cells.

9 twork and its local mechanical properties in alveolar epithelial cells.

10 the cell that help maintain the integrity of alveolar epithelial cells.

11 to Candida albicans filaments and A549 human alveolar epithelial cells.

12 vated protein kinase (AMPK) at Thr172 in rat alveolar epithelial cells.

13 ces p53-dependent apoptosis in primary human alveolar epithelial cells.

14 ltered differentiation of type I and type II alveolar epithelial cells.

15 cts as a growth and anti-apoptotic factor on alveolar epithelial cells.

16 li to PC, and type I (WI-26 VA4) and type II alveolar epithelial cells.

17 acterial binding to pulmonary surfactant and alveolar epithelial cells.

18 fimbriae mediate bacterial binding to human alveolar epithelial cells.

19 decreased B7-H3 expression on bronchial and alveolar epithelial cells.

20 sed on unstimulated tracheal, bronchial, and alveolar epithelial cells.

21 cells and ICOS-L expression on bronchial and alveolar epithelial cells.

22 H and cystatin B were colocalized in type 2 alveolar epithelial cells.

23 s in K8, leading to the disassembly of IF in alveolar epithelial cells.

24 the presence of the Ad hexon antigen within alveolar epithelial cells.

25 med in vitro by analysis of isolated type II alveolar epithelial cells.

26 regulate proinflammatory cytokine release in alveolar epithelial cells.

27 or T3 stimulation of Na,K-ATPase activity in alveolar epithelial cells.

28 and its cell surface expression in adult rat alveolar epithelial cells.

29 use of increased proliferation of ductal and alveolar epithelial cells.

30 ith effects on both alveolar macrophages and alveolar epithelial cells.

31 roxic mice, predominantly in macrophages and alveolar epithelial cells.

32 ) and 31% (p < 0.001), respectively, in A549 alveolar epithelial cells.

33 rated that Pneumocystis is not cytotoxic for alveolar epithelial cells.

34 auses pulmonary toxicity in part by injuring alveolar epithelial cells.

35 virus vectors for gene transfer to quiescent alveolar epithelial cells.

36 in expression in alveolar macrophages and in alveolar epithelial cells.

37 TPase activity on the basolateral surface of alveolar epithelial cells.

38 is probably due to Na,K-ATPase regulation in alveolar epithelial cells.

39 within U937 macrophage-like cells and WI-26 alveolar epithelial cells.

40 a(+) channels and basolateral Na,K-ATPase in alveolar epithelial cells.

41 ansport across the apical membrane of type I alveolar epithelial cells.

42 ntracellular pools to the plasma membrane of alveolar epithelial cells.

43 r CXCR3 and its ligand CXCL9 modulate EMT in alveolar epithelial cells.

44 ercapnia)-induced Na,K-ATPase endocytosis in alveolar epithelial cells.

45 n of large cytoplasmic lipid droplets in the alveolar epithelial cells.

46 promoting the endocytosis of Na,K-ATPase in alveolar epithelial cells.

47 of cells identifiable as functional type II alveolar epithelial cells.

48 rally localized Na(+)-K(+)-ATPase in type II alveolar epithelial cells.

49 e did not activate PKA in human bronchial or alveolar epithelial cells.

50 goblet, Clara, ciliated, type I and type II alveolar epithelial cells.

51 lted in increased adherence of yeast to A549 alveolar epithelial cells.

52 ortant role in the ubiquitination of ENaC in alveolar epithelial cells.

53 ly regulated by the p38 MAP kinase in murine alveolar epithelial cells.

54 zation and inhibiting TGF-beta1 signaling in alveolar epithelial cells.

55 in 2.8-fold more PGE2 than control mice, and alveolar epithelial cells (2.7-fold), AMs (125-fold), an

56 RANTES promoter activation in human type II alveolar epithelial cells (A549 cells).

57 in vitro model consisting of human PMNs and alveolar epithelial cells (A549) grown on inverted Trans

58 Exposure of the human-type II cell alveolar epithelial cells (A549) to DPM derived from FBC

59 n of RANTES gene expression in human type II alveolar epithelial cells (A549), following RSV infectio

60 cidosis and hypoxia in fibroblasts (N12) and alveolar epithelial cells (A549).

61 n histone acetylation:deacetylation in human alveolar epithelial cells (A549).

62 We conclude that alveolar epithelial cells actively participate in the in

63 (Adora2b(loxP/loxP) VE-cadherin Cre(+)), or alveolar epithelial cells (Adora2b(loxP/loxP) SPC Cre(+)

64 a direct link between injury to the type II alveolar epithelial cell (AEC) and the accumulation of i

65 nitase-2 (Aco-2) each reduce oxidant-induced alveolar epithelial cell (AEC) apoptosis, but it is uncl

66 Alveolar epithelial cell (AEC) apoptosis, proliferation,

67 c depletion approaches, we demonstrated that alveolar epithelial cell (AEC) GM-CSF mediates recovery

68 Alveolar epithelial cell (AEC) injury is a key step that

69 Alveolar epithelial cell (AEC) mitochondrial dysfunction

70 Alveolar epithelial cell (AEC) trans-differentiation is

71 urine cell line derived specifically from an alveolar epithelial cell (AEC) was utilized.

72 lls (AEC II) trans-differentiate into type I alveolar epithelial cells (AEC I) during lung recovery a

73 oliferate and transdifferentiate into type I alveolar epithelial cells (AEC I) when the normal AEC I

74 Type II alveolar epithelial cells (AEC II) proliferate and trans

75 s-differentiation is a process where type II alveolar epithelial cells (AEC II) trans-differentiate i

76 ch, causes disassembly of keratin IF in lung alveolar epithelial cells (AEC) and that this disassembl

77 nt to high altitude and pulmonary edema, the alveolar epithelial cells (AEC) are exposed to hypoxic c

78 tivated by integrin alphavbeta6 expressed on alveolar epithelial cells (AEC) continuously inhibits th

79 Although alveolar epithelial cells (AEC) form an important barrie

80 Primary rat alveolar epithelial cells (AEC) grown on plastic or as p

81 -beta1 (TGF-beta1) interactions in pulmonary alveolar epithelial cells (AEC) in the context of EMT an

82 genous pulmonary cytokine produced by normal alveolar epithelial cells (AEC) that is a key defender o

83 -adrenergic regulation of the Na,K-ATPase in alveolar epithelial cells (AEC).

84 Type 2 alveolar epithelial cells (AEC2s) are stem cells in the

85 anoid colony formation and found that type 2 alveolar epithelial cells (AEC2s), the stem cell-contain

86 ing CO(2)-induced Na,K-ATPase endocytosis in alveolar epithelial cells (AECs) and alveolar epithelial

87 Human alveolar epithelial cells (AECs) and alveolar macrophage

88 ese responses reflect the cross-talk between alveolar epithelial cells (AECs) and resident alveolar m

89 We confirmed the expression of IRAK-M in alveolar epithelial cells (AECs) and showed that hyperox

90 lters Na,K-ATPase expression and function in alveolar epithelial cells (AECs) and the ability of the

91 a type III intermediate filament protein, in alveolar epithelial cells (AECs) are unknown.

92 We and others have shown that activated alveolar epithelial cells (AECs) directly contribute to

93 eviously unrecognized subpopulation of mouse alveolar epithelial cells (AECs) expressing the laminin

94 Human alveolar epithelial cells (AECs) form the key line of lu

95 Apoptosis of alveolar epithelial cells (AECs) has been implicated as

96 We hypothesized that alveolar epithelial cells (AECs) may serve as a source o

97 Alveolar epithelial cells (AECs) participate in the path

98 enchymal transition, involving transition of alveolar epithelial cells (AECs) to pulmonary fibroblast

99 Indeed, alveolar epithelial cells (AECs) undergo EMT in vivo dur

100 Recent studies have shown that alveolar epithelial cells (AECs), in addition to promoti

101 omechanical properties of the KIF network in alveolar epithelial cells (AECs), independent of other c

102 In alveolar epithelial cells (AECs), the membrane-anchored

103 vidence of herpesvirus antigen expression in alveolar epithelial cells (AECs), which correlated with

104 ts an important role of aberrantly activated alveolar epithelial cells (AECs), which produce a large

105 tion involves the attachment of organisms to alveolar epithelial cells (AECs).

106 bition blocked both pY654 and EMT in primary alveolar epithelial cells (AECs).

107 c response is driven by abnormally activated alveolar epithelial cells (AECs).

108 endoplasmic reticulum (ER) stress in type II alveolar epithelial cells (AECs).

109 se-dependent injury and death in healthy rat alveolar epithelial cells (AECs).

110 dance in the plasma membrane and activity in alveolar epithelial cells (AECs).

111 ighly regulated by the adhesion molecules on alveolar epithelial cells (AECs).

112 with GM-CSF, an agent we have shown protects alveolar epithelial cells against apoptosis, decreased F

113 the expression of Egr-1, CTGF, and Cyr61 to alveolar epithelial cells, airway epithelial cells, and

114 rs throughout the lung, including airway and alveolar epithelial cells, airway smooth muscle cells, a

115 These alveolar epithelial cells also expressed the correspondi

116 s within alveolar macrophages, and possibly, alveolar epithelial cells and also within protozoa in th

117 uctance regulator regulates fluid balance in alveolar epithelial cells and appears to modulate the in

118 tivity is restricted in the adult to type II alveolar epithelial cells and bronchial epithelial cells

119 The binding of P. carinii to alveolar epithelial cells and extracellular matrix const

120 idence indicates that aberrant activation of alveolar epithelial cells and fibroblasts in an aging lu

121 replication within mammalian macrophages and alveolar epithelial cells and for intrapulmonary replica

122 ice also demonstrated increased apoptosis of alveolar epithelial cells and greater numbers of fibrobl

123 ffect of Eyjafjallajokull ash on primary rat alveolar epithelial cells and human airway epithelial ce

124 the alveolar surface as a product of type II alveolar epithelial cells and includes PC as the major c

125 stry analysis of IL-6 and IL-8 revealed that alveolar epithelial cells and macrophages and a few inte

126 we detected CLF-1 expression in both type II alveolar epithelial cells and macrophages.

127 ion in airway epithelium, as well as type II alveolar epithelial cells and macrophages.

128 optosis and proliferation in mesothelial and alveolar epithelial cells and may be linked to the devel

129 PF, CLF-1 is a selective stimulus of type II alveolar epithelial cells and may potentially drive an a

130 ry interface of the living human lung, human alveolar epithelial cells and microvascular endothelial

131 D4 (LTD4) on the function of Na,K-ATPase in alveolar epithelial cells and on alveolar fluid clearanc

132 ties of TH are associated with protection of alveolar epithelial cells and restoration of mitochondri

133 YapV acted as an adhesin for alveolar epithelial cells and specific extracellular mat

134 microparticles, respectively, for uptake by alveolar epithelial cells and subsequent inhibition of S

135 an autocrine trophic factor for human adult alveolar epithelial cells and that under situations of p

136 ng SOD1 prevented the PM2.5-induced death of alveolar epithelial cells and the associated increase in

137 ll viability were performed using A549 human alveolar epithelial cells and THP-1 monocyte-derived mac

138 and invasion of human-derived HeLa and A549 alveolar epithelial cells and to its inability to surviv

139 was expressed in ciliated airway and type II alveolar epithelial cells and was targeted for cell-spec

140 creased [Ca(2+)](i) and activated cPLA(2) in alveolar epithelial cells, and increased both endothelia

141 ation produces acute lung injury by inducing alveolar epithelial cell apoptosis and by generating loc

142 strated increased pulmonary inflammation and alveolar epithelial cell apoptosis compared to controls.

143 ligand (FasL) system has been implicated in alveolar epithelial cell apoptosis during pulmonary fibr

144 Although alveolar epithelial cell apoptosis has been documented a

145 flammatory responses or direct inhibition of alveolar epithelial cell apoptosis would improve surviva

146 ll viability in O(2), decreased O(2)-induced alveolar epithelial cell apoptosis, and accelerated alve

147 reducing extracellular matrix production and alveolar epithelial cell apoptosis.

148 ough mice lacking c-Jun specifically in lung alveolar epithelial cells appear normal at the age of 6

149 ells decreases the expression of markers for alveolar epithelial cells (Aqp5 and Sftpc), Clara cells

150 These findings suggest that human alveolar epithelial cells are a target of Bacillus anthr

151 ls lining the respiratory units of the lung, alveolar epithelial cells, are a target of lethal toxin

152 These data reveal alveolar epithelial cells as progenitors for fibroblasts

153 xic DNA double-strand breaks (DSBs) in human alveolar epithelial cells, as indicated by ataxia telang

154 d club cells' capacity to differentiate into alveolar epithelial cells at the single-cell level.

155 tenuated mitochondria-regulated apoptosis in alveolar epithelial cells both in vivo and in vitro.

156 eceptor expressed on both type I and type II alveolar epithelial cells but not vascular endothelium,

157 -8 activation, which was localized to type 1 alveolar epithelial cells by flow cytometric analysis.

158 These studies indicate that bronchial and alveolar epithelial cell C5aR is up-regulated and greatl

159 lens cells, and recent studies indicate that alveolar epithelial cells can be derived from hematopoie

160 Extensive infection of alveolar epithelial cells caused apoptosis and leakage o

161 he genetic deletion of c-Jun specifically in alveolar epithelial cells causes progressive emphysema w

162 expression of Fas on the surface of type II alveolar epithelial cells; conversely, infection with P.

163 Ex vivo, primary alveolar epithelial cells cultured on provisional matrix

164 In patients with severe ARDS, alveolar epithelial cell death is a major mechanism that

165 We have previously shown that in alveolar epithelial cells, deformation induces lipid tra

166 Similar results in A549 lung alveolar epithelial cells demonstrated the biological re

167 in alpha5 in signaling pathways that promote alveolar epithelial cell differentiation and VEGF expres

168 d by disrupted secondary septation, abnormal alveolar epithelial cell differentiation, excessive coll

169 00/beta-catenin interaction, concurrent with alveolar epithelial cell differentiation.

170 otype is consistent with a reduced number of alveolar epithelial cells due to a decrease in cell prol

171 ommunication between macrophages and type II alveolar epithelial cells during influenza infection whe

172 Using LA4 murine lung alveolar epithelial cells, effects of doxycycline and er

173 The role of alveolar epithelial cells, endothelial cells, and alveol

174 d protein levels were elevated in airway and alveolar epithelial cells, endothelial cells, and neutro

175 al resting lung interstitial macrophages and alveolar epithelial cells express high levels of RGMb mR

176 Alveolar epithelial cells expressed lethal toxin recepto

177 Type II alveolar epithelial cell expression of IL-4Rs in mice se

178 Apoptosis of alveolar epithelial cells, followed by abnormal tissue r

179 ularensis also invades and replicates within alveolar epithelial cells following inhalation in a mous

180 there was no change in COX-1/COX-2 levels in alveolar epithelial cells following treatment with CT an

181 To more definitively test the capacity of alveolar epithelial cells for EMT, mice expressing beta-

182 lines (MLE-15, LA-4) and in primary type II alveolar epithelial cells, FRH enhanced apoptosis in res

183 By contrast, alveolar epithelial cells from bid(-/-) mice were resist

184 cells as well as in freshly isolated type II alveolar epithelial cells from different species.

185 sure to PM(2.5) induced apoptosis in primary alveolar epithelial cells from wild-type but not Noxa(-/

186 In alveolar epithelial cells, G-protein coupled-receptors a

187 e, expresses Pdgfralpha, and is critical for alveolar epithelial cell growth and self-renewal.

188 ctrum of RSV-induced chemokines expressed by alveolar epithelial cells has not been fully investigate

189 d surfactant proteins, it is unknown whether alveolar epithelial cells have distinct roles in innate

190 interstitial cells, and additionally type I alveolar epithelial cells immunostained for green fluore

191 tes caspase-3 in macrophages, monocytes, and alveolar epithelial cells in a Dot/Icm-dependent manner

192 phage-derived MVs were fully internalized by alveolar epithelial cells in a time-, dose-, and tempera

193 increased Na,K-ATPase activity of adult rat alveolar epithelial cells in a transcription-independent

194 In this study, we exposed alveolar epithelial cells in culture and mice to fine pa

195 ound efficient transduction of bronchial and alveolar epithelial cells in hDSG2-transgenic mice.

196 at miR-34a expression is increased in type 2 alveolar epithelial cells in neonates with respiratory d

197 was selectively deleted from bronchiolar and alveolar epithelial cells in Stat3(DeltaDelta) mice.

198 e initially believed merely to assist type 2 alveolar epithelial cells in surfactant production durin

199 emely hydrophobic, 4-kDa peptide produced by alveolar epithelial cells in the lung.

200 mediated an increased bacterial adherence to alveolar epithelial cells in the presence of Vn.

201 TES production in macrophages and cocultured alveolar epithelial cells in vitro.

202 correlates with cytolysis of macrophages and alveolar epithelial cells in vitro.

203 hypoxia, and hypoxia directly induced DCK in alveolar epithelial cells in vitro.

204 Alveolar epithelial cell infection was normal, but alveo

205 rms of pathogenesis, IPF is characterized by alveolar epithelial cell injury and activation with inte

206 y sublethal hyperoxic insult, accompanied by alveolar epithelial cell injury and increased pulmonary

207 ratory distress syndrome is characterized by alveolar epithelial cell injury, edema formation, and in

208 mice from hyperoxic lung injury by limiting alveolar epithelial cell injury.

209 nism for this deterioration may be increased alveolar epithelial cell injury.

210 he ATCC 19606(T) type strain with A549 human alveolar epithelial cells is independent of the producti

211 Whether these effects are mediated by alveolar epithelial cells is unclear.

212 that differentiation of type II cells, also alveolar epithelial cells, is normal.

213 nduced similar levels cell death in vitro in alveolar epithelial cells isolated from WT and bid(-/-)

214 hermore, there was minimal growth in a human alveolar epithelial cell line (A549).

215 lial RelA activation, we stimulated a murine alveolar epithelial cell line (MLE-15) with bronchoalveo

216 tion of IL-31 was characterized in the human alveolar epithelial cell line A549 in which the expressi

217 to the keratin IF network in A549 cells (an alveolar epithelial cell line) exposed to 1.5% oxygen.

218 Based on in vitro studies with an alveolar epithelial cell line, A549 cells, the effect of

219 f2 by hyperoxia using a non-malignant murine alveolar epithelial cell line, C10.

220 that Ad7 induces IL-8 release from the A549 alveolar epithelial cell line.

221 g with alveolar macrophages or with the A549 alveolar epithelial cell line.

222 Bronchial and alveolar epithelial cell lines, and primary human bronch

223 that invaded but failed to replicate within alveolar epithelial cell lines.

224 tularensis LVS invaded and replicated within alveolar epithelial cell lines.

225 gen activation upregulated PGE2 synthesis in alveolar epithelial cells, lung fibroblasts, and lung fi

226 sitive correlation between CDC42 and type II alveolar epithelial cells marker SP-A, indicating the po

227 sion of beta-catenin-driven target genes and alveolar epithelial cell markers in the elastase, as wel

228 iciency promotes lung fibrosis by augmenting alveolar epithelial cell mitochondrial DNA damage and ap

229 anical stretch (MS) potentiates LPS-mediated alveolar epithelial cell (MLE-12) expression of the chem

230 NLRP3 improves the integrity of alveolar epithelial cell monolayers by enhancing cellula

231 L-1beta-mediated protein permeability across alveolar epithelial cell monolayers.

232 otein, gp60, was studied in cultured type II alveolar epithelial cells obtained from rat lungs.

233 DCK was elevated in hyperplastic alveolar epithelial cells of patients with IPF and in mi

234 sent in the total lipid extract from type II alveolar epithelial cells of the cell line A549 separate

235 ypes of human cells, such as macrophages and alveolar epithelial cells of the lung.

236 When studied on rat primary alveolar epithelial cells or on immortalized human pulmo

237 g aspects of aberrant wound repair involving alveolar epithelial cells or septal endothelial cells ar

238 tations, IPF patients had short telomeres in alveolar epithelial cells (P < 0.0001).

239 oxygen species generation, and apoptosis of alveolar epithelial cells, potential mechanisms of LYCAT

240 These data provide the first evidence that alveolar epithelial cells produce dopamine and that incr

241 Here we tested whether alveolar epithelial cells produce dopamine and whether i

242 beta1 integrin-deficient alveolar epithelial cells produced excessive monocyte ch

243 l as murine bone marrow macrophages, but not alveolar epithelial cells, produced type I IFNs upon inf

244 markedly reduced due to decreased ductal and alveolar epithelial cell proliferation and decreased sur

245 AL surfactant protein D, a marker of type II alveolar epithelial cell proliferation in a human model

246 ion of pulmonary fibrosis by contributing to alveolar epithelial cell proliferation.

247 ted inflammatory response in IPS-1-deficient alveolar epithelial cells, pulmonary macrophages, and CD

248 ty of IPF, we hypothesized that hyperplastic alveolar epithelial cells regulate the fibrotic response

249 Furthermore, alveolar epithelial cells require H2R to produce CCL24,

250 regulates ADAM10 metalloprotease activity in alveolar epithelial cells, resulting in cleavage of the

251 of the Na,K-ATPase to the plasma membrane of alveolar epithelial cells results in increased active Na

252 This finding was subsequently confirmed in alveolar epithelial cell scratch models.

253 rosis was examined using a DCK inhibitor and alveolar epithelial cell-specific knockout mice.

254 grew normally in murine lung macrophages and alveolar epithelial cells, suggesting that legiobactin p

255 ation of the RANTES promoter in RSV-infected alveolar epithelial cells supports the enhanceosome mode

256 elial sodium channel (ENaC) complex from the alveolar epithelial cell surface, leading to persistence

257 s, particularly fibronectin, associated with alveolar epithelial cell surfaces, triggers organism pro

258 wild-type strain PAO1 were more cytotoxic to alveolar epithelial cells than those from quorum-sensing

259 ying a novel integrin alpha6beta4-expressing alveolar epithelial cell that serves as a multipotent pr

260 oncogene and display morphologic changes in alveolar epithelial cells that recapitulate those of pre

261 In alveolar epithelial cells, the overexpression of SOD1 pr

262 activates the intrinsic apoptotic pathway in alveolar epithelial cells through a pathway that require

263 , dopamine increased Na,K-ATPase activity in alveolar epithelial cells through the exocytosis of Na,K

264 The lung is protected from pathogens by alveolar epithelial cells, tissue-resident alveolar macr

265 We exposed rat primary alveolar epithelial cells to a variety of biaxial stretc

266 Malignant progression of normal alveolar epithelial cells to adenocarcinoma in Kras(LA1)

267 increased with malignant progression (normal alveolar epithelial cells to adenocarcinoma) in K-ras(LA

268 In vitro exposure of primary murine alveolar epithelial cells to Legionella in conjunction w

269 jury requires the repair and regeneration of alveolar epithelial cells to restore the integrity of ga

270 odifications regulate Aqp5 expression during alveolar epithelial cell transdifferentiation, suggestin

271 ryonic fibroblasts from ASK1 knock-out mice, alveolar epithelial cells transfected with dominant nega

272 In studies using isolated murine type II alveolar epithelial cells, treatment with GM-CSF greatly

273 hologically and functionally distinct cells: alveolar epithelial cell types I and II (AEC I and II).

274 relationship between distinct differentiated alveolar epithelial cell types in vivo and in single-cel

275 oxygen species generation, and apoptosis of alveolar epithelial cells under bleomycin challenge.

276 nic mice overexpressing TNF-alpha in type II alveolar epithelial cells under the control of the surfa

277 Alveolar epithelial cells undergoing transforming growth

278 fail to lyse and egress from macrophages and alveolar epithelial cells upon termination of intracellu

279 30 days and co-localized in bronchiolar and alveolar epithelial cells using an antibody to cytokerat

280 on in the lung, we depleted JAM-A in primary alveolar epithelial cells using shRNA.

281 mation in lung microvascular endothelial and alveolar epithelial cells via small Rho GTPase.

282 loped an in vitro system in which changes in alveolar epithelial cell viability can be measured after

283 In vitro, EphrinB2 preserved alveolar epithelial cell viability in O(2), decreased O(

284 Further, no reduction in alveolar epithelial cell viability was observed, but let

285 In primary rat alveolar epithelial cells, we found that exposure to hyp

286 n A2B adenosine receptor-specific agonist to alveolar epithelial cells, we subsequently performed stu

287 The type II alveolar epithelial cells were capable of surfactant pro

288 In vitro, alveolar epithelial cells were exposed to oxidative stre

289 Alveolar epithelial cells were isolated from rats 24 hou

290 When cultured rat alveolar epithelial cells were subjected to cyclic stret

291 blood human or rat mononuclear cells and rat alveolar epithelial cells when stimulated with phorbol e

292 internalization of TGF-beta1 and TbetaRI in alveolar epithelial cells, which inhibited TGF-beta1 sig

293 TGF-beta1 activation mediated by IL-1beta in alveolar epithelial cells, which requires actin stress f

294 of the disease is areas of injury to type II alveolar epithelial cells with attendant accumulation of

295 Treatment of alveolar epithelial cells with bronchoalveolar lavage fl

296 -2 is induced by RSV infection of human lung alveolar epithelial cells with the concomitant productio

297 Treatment of alveolar epithelial cells with the NAD(P)H oxidase inhib

298 o determine the effects of FZD4 signaling on alveolar epithelial cell wound healing and repair, as we

299 ature WNT-5A attenuated canonical WNT-driven alveolar epithelial cell wound healing and transdifferen

300 r epithelial cell apoptosis, and accelerated alveolar epithelial cell wound healing, maintained lung