ライフサイエンスコーパス: alveolar epithelium

1 t IL-1beta may promote repair of the injured alveolar epithelium.

2 of local factors that cause apoptosis in the alveolar epithelium.

3 ed with transcripts located in bronchial and alveolar epithelium.

4 (2+)](i) increases and cPLA(2) activation in alveolar epithelium.

5 al Na+ flux and Na, K-ATPase function in rat alveolar epithelium.

6 ithelium yet robust staining was seen in the alveolar epithelium.

7 um transport and Na,K-ATPase function in the alveolar epithelium.

8 depended on active ion transport across the alveolar epithelium.

9 r epithelium yet no staining was seen in the alveolar epithelium.

10 ction in the fluid transport capacity of the alveolar epithelium.

11 of P. carinii organisms aggregated along the alveolar epithelium.

12 on of hypoxia-inducible factor 1alpha in the alveolar epithelium.

13 e, pulmonary interstitium, and bronchial and alveolar epithelium.

14 e electrophysiologic properties of the human alveolar epithelium.

15 es the bioelectric barrier properties of the alveolar epithelium.

16 the existence of other water channels in the alveolar epithelium.

17 s, there was marked staining of hyperplastic alveolar epithelium.

18 ms of injury to the lung endothelium and the alveolar epithelium.

19 the production of surfactant proteins in the alveolar epithelium.

20 murine model of RelA mutated throughout the alveolar epithelium.

21 within tracheal, bronchial, bronchiolar, and alveolar epithelium.

22 e mediators is selectively elaborated by the alveolar epithelium.

23 uired for wound repair and remodeling of the alveolar epithelium.

24 ch activated Notch1 was overexpressed in the alveolar epithelium.

25 fferences between the cells that make up the alveolar epithelium.

26 as enriched in the lung and localized to the alveolar epithelium.

27 onstitute approximately 60% of the pulmonary alveolar epithelium.

28 in (average 0.2 mum) liquid layer lining the alveolar epithelium.

29 and secreted into the alveolar space by the alveolar epithelium.

30 vascular endothelium, plus CCR6 to traverse alveolar epithelium.

31 ells (AT1s) during the repair of the damaged alveolar epithelium.

32 fluid reabsorption, via Na,K-ATPase, in the alveolar epithelium.

33 hat causes endocytosis of Na,K-ATPase by the alveolar epithelium.

34 robably by downregulating Na,K-ATPase in the alveolar epithelium.

35 cell proliferation and decreased survival of alveolar epithelium.

36 y accounted for by greater protection of the alveolar epithelium.

37 drenergic receptor-cAMP signaling pathway in alveolar epithelium.

38 the formation and differentiation of mammary alveolar epithelium.

39 cular phenotype of type I pneumocytes of the alveolar epithelium.

40 tivation and inflammation along the delicate alveolar epithelium.

41 d the normal fluid transport capacity of the alveolar epithelium after prolonged hemorrhagic shock, w

42 ation between the levels of 4-HNE adducts in alveolar epithelium, airway endothelium, and neutrophils

43 We conclude that injury to the alveolar epithelium allows the release of proinflammator

44 er-specific histone modifications in primary alveolar epithelium and A549 lung adenocarcinoma cells.

45 betaAR) regulate active Na+ transport in the alveolar epithelium and accelerate clearance of excess a

46 ) are collectins expressed in the airway and alveolar epithelium and could have a role in the regulat

47 aracterized by apoptosis and necrosis of the alveolar epithelium and endothelium.

48 and up-regulation of Fas/FasL expression in alveolar epithelium and in infiltrating cells during the

49 growth factor expression by the bronchiolar-alveolar epithelium and lung macrophages.

50 We showed that AKAP13 is expressed in the alveolar epithelium and lymphoid follicles from patients

51 -directional transport of protein across the alveolar epithelium and restored alveolar fluid clearanc

52 lentivirus vectors for gene transfer to the alveolar epithelium and suggests that differences exist

53 w tidal volume ventilation protects both the alveolar epithelium and the endothelium in this model of

54 rting water across the apical surface of the alveolar epithelium and the epithelia of submucosal glan

55 The Na,K-ATPase has been localized to the alveolar epithelium and the importance of its role in co

56 in scleroderma lung tissue localized to the alveolar epithelium and the pulmonary interstitium which

57 events would affect the permeability of the alveolar epithelium and ultimately lead to epithelial ba

58 proliferation and differentiation of mammary alveolar epithelium and up-regulation of p21(WAF1) and p

59 upregulation after injury, its induction by alveolar epithelium, and its release into both lumenal a

60 idirectional transport of protein across the alveolar epithelium, and restored alveolar liquid cleara

61 d-induced lung injury on the function of the alveolar epithelium, and secondly to determine whether p

62 Na+ transport across the alveolar epithelium, and thus alveolar fluid resorption,

63 ffects of chronic alcohol abuse on the human alveolar epithelium are essentially unknown.

64 by a number of mechanisms (e.g., damage the alveolar epithelium, biotrauma).

65 at Mycobacterium tuberculosis penetrates the alveolar epithelium by downregulating its barrier proper

66 xpressing L188Q SFTPC exclusively in type II alveolar epithelium by using the Tet-On system.

67 The transfer of protective genes to the alveolar epithelium can attenuate lung injury if accompl

68 d, and their ability to transfect model lung alveolar epithelium cells in vitro was demonstrated.

69 Of the 14 claudins expressed by the alveolar epithelium, claudin-3, claudin-4, and claudin-1

70 The alveolar epithelium consists of type II cells, which sec

71 ed that overexpression of Na,K-ATPase in the alveolar epithelium could counterbalance these changes a

72 mmary glands exhibited a progressive loss of alveolar epithelium, culminating in lactation failure.

73 med to determine the role of beta-catenin in alveolar epithelium during bleomycin-induced lung fibros

74 ls resulted in precocious differentiation of alveolar epithelium during pregnancy and the activation

75 resence resulted in the dedifferentiation of alveolar epithelium followed by a transdifferentiation i

76 tion of vectorial fluid transport across the alveolar epithelium following haemorrhagic shock is medi

77 res a normal fluid transport capacity of the alveolar epithelium following hemorrhagic shock by inhib

78 xchange, but a functional cell that protects alveolar epithelium from injury.

79 ugh the interaction of Pneumocystis with the alveolar epithelium has been well documented, very littl

80 uggest that compromising p53 function in the alveolar epithelium impairs recovery of the lung from bl

81 ng was increased in alveolar macrophages and alveolar epithelium in 95% O(2).

82 e other important functions in repair of the alveolar epithelium in acute lung injury (ALI).

83 force for reabsorption of water through the alveolar epithelium in addition to other ion channels su

84 However, the role of the alveolar epithelium in any of these pathogenic processes

85 ietic progenitor cells to regenerate injured alveolar epithelium in newborn mice.

86 res a normal fluid transport capacity of the alveolar epithelium in the early phase following haemorr

87 findings demonstrate a pivotal role for the alveolar epithelium in the maintenance of alveolar homeo

88 study was to investigate the function of the alveolar epithelium in the setting of reperfusion lung i

89 ving effects on injured lung endothelium and alveolar epithelium, including enhancing the resolution

90 ,K-ATPase subunit gene overexpression in the alveolar epithelium increases Na,K-ATPase function and l

91 ents in lung encounters serial barriers: the alveolar epithelium, interstitium, and capillary endothe

92 Injury to the alveolar epithelium is a critical event in ALI, and accu

93 The alveolar epithelium is a key regulator of the innate imm

94 thogenesis, in which recurrent injury to the alveolar epithelium is believed to drive aberrant wound

95 The alveolar epithelium is characteristically abnormal in fi

96 The alveolar epithelium is composed of alveolar type 1 (AT1)

97 The alveolar epithelium is composed of the flat type I cells

98 Repair of the alveolar epithelium is critical for clinical recovery; h

99 Active re-absorption of Na+ across the alveolar epithelium is essential to maintain lung fluid

100 The alveolar epithelium is lined by surfactant, a lipoprotei

101 Improper regeneration of the alveolar epithelium is often associated with severe pulm

102 These findings suggest that the alveolar epithelium is the primary target of Fas-mediate

103 ral infection from conducting airways to the alveolar epithelium is therefore a pivotal event in infl

104 ch damage pulmonary vascular endothelium and alveolar epithelium, leads to alveolar oedema and pulmon

105 ate that the fluid transport capacity of the alveolar epithelium may be well preserved in the allogra

106 ator-induced nitric oxide (NO) production by alveolar epithelium may exceed that of other lung cell t

107 on, functional differentiation, and death of alveolar epithelium occur repeatedly with each pregnancy

108 energic receptor (beta2AR) function into the alveolar epithelium of beta1AR-/-/beta2AR-/- and beta1AR

109 Directed expression of TGFbeta3 in the alveolar epithelium of lactating mice using a beta-lacto

110 tein were constitutively expressed in normal alveolar epithelium of mice, and IL-10R were constitutiv

111 NA (adCFTR) to increase CFTR function in the alveolar epithelium of normal rats and mice.

112 beta(2)AR gene transfer to the alveolar epithelium of normal rats improved membrane-bou

113 ivary and lacrimal glands and is abundant in alveolar epithelium of the lung.

114 s, including heart, skeletal muscle, and the alveolar epithelium of the lung.

115 beta-catenin gene in differentiating mammary alveolar epithelium of the mouse results in the generati

116 well as gamma-GCS mRNA levels in airway and alveolar epithelium (p < 0.01).

117 beta-Catenin in the alveolar epithelium protects against bleomycin-induced f

118 Progenitors of the alveolar epithelium remain largely mysterious, so the pr

119 eased proliferation and reduced apoptosis of alveolar epithelium, resulting in increased formation of

120 Overexpression of a human beta2AR in the alveolar epithelium significantly increased AFC in norma

121 hat IL-10, constitutively produced by normal alveolar epithelium, stimulates signal transduction thro

122 itment maneuvers on the lung endothelium and alveolar epithelium, the net effect in clinical acute lu

123 r ability to protect the endothelium and the alveolar epithelium through multiple paracrine mechanism

124 d endogenous beta-catenin in differentiating alveolar epithelium through the deletion of exon 3 (amin

125 and Cl(-) transport occurs across the entire alveolar epithelium (TI and TII cells) rather than only

126 ologic concentrations of adenosine allow the alveolar epithelium to counterbalance active Na(+) absor

127 The ability of the alveolar epithelium to prevent and resolve pulmonary ede

128 trategies aimed at increasing the ability of alveolar epithelium to resorb the edema should lead to b

129 nsible for the shock-mediated failure of the alveolar epithelium to respond to catecholamines in rats

130 nsible for the shock-mediated failure of the alveolar epithelium to respond to catecholamines in rats

131 esized that FRH augments the response of the alveolar epithelium to TNF-alpha receptor family signali

132 e lung production of NO and a failure of the alveolar epithelium to up-regulate vectorial fluid trans

133 he role of beta-catenin signaling in mammary alveolar epithelium, we have stabilized endogenous beta-

134 expression becomes restricted to the distal alveolar epithelium whereas Foxp1 expression is observed

135 damage occurs primarily in distal airway and alveolar epithelium, whereas sFasL is present throughout

136 es are regulated by ion transport across the alveolar epithelium, which is composed of alveolar type

137 extracellularly in the hypophase lining the alveolar epithelium, which is highly enriched in lung su

138 ted suppression of NF-kappaB activity in the alveolar epithelium with a resultant increase in suscept

139 bility of the injured lung to repopulate the alveolar epithelium with functional cells.