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1  by reducing lung inflammation and enhancing alveolar fluid clearance.
2 nary edema by up-regulating sodium-dependent alveolar fluid clearance.
3 eolar epithelial fluid transport measured as alveolar fluid clearance.
4 ndothelial barrier permeability and restored alveolar fluid clearance.
5 mechanisms may contribute to the decrease in alveolar fluid clearance.
6 bited both basal and beta agonist-stimulated alveolar fluid clearance.
7 is, neutrophil activation and clearance, and alveolar fluid clearance.
8 elial permeability to protein, and decreased alveolar fluid clearance.
9 ed elevated lung inflammation and attenuated alveolar fluid clearance.
10 a level comparable to maximal cAMP-dependent alveolar fluid clearance.
11 samples were associated with slower rates of alveolar fluid clearance.
12 genous catecholamines did not correlate with alveolar fluid clearance.
13 in and an approximately 50% reduction in net alveolar fluid clearance.
14 id instillation led to a 50% decrease in net alveolar fluid clearance.
15 ANTU-induced edema formation by potentiating alveolar fluid clearance.
16  mild pulmonary oedema (24/29 [83%]), intact alveolar fluid clearance (17/23 [74%]), and normal or mi
17 ith serial samples, there was a high rate of alveolar fluid clearance (19 +/- 9%/h, mean +/- SD).
18 lar epithelial dysfunction, as determined by alveolar fluid clearance (AFC) and intra-alveolar levels
19                  Furthermore, measurement of alveolar fluid clearance (AFC) demonstrated that A2BAR s
20 h acute lung injury (ALI) who retain maximal alveolar fluid clearance (AFC) have better clinical outc
21       Moreover, induced Na,K-ATPase improved alveolar fluid clearance (AFC) in IAV-infected mice.
22 of the NO donor, DETANONOate, would decrease alveolar fluid clearance (AFC) in the rabbit in vivo.
23 nfected with M. pulmonis for measurements of alveolar fluid clearance (AFC) in vivo and isolation of
24  reactive byproducts inhibit Na(+)-dependent alveolar fluid clearance (AFC) in vivo and the activity
25                                              Alveolar fluid clearance (AFC) is necessary for the reso
26    Whether these receptors are essential for alveolar fluid clearance (AFC) or if other mechanisms ar
27                                              Alveolar fluid clearance (AFC), an index of active Na(+)
28                                              Alveolar fluid clearance (AFC), an index of alveolar act
29 A(2a)R- or A(3)R-specific agonists increased alveolar fluid clearance (AFC), whereas physiologic conc
30 g the inhibition of the c-AMP stimulation of alveolar fluid clearance (ALC) in rats.
31 aspiration-induced lung injury by increasing alveolar fluid clearance and decreasing endothelial perm
32 hese data suggest that claudin-4 may promote alveolar fluid clearance and demonstrate that the amount
33 subunit plasmid showed a twofold increase in alveolar fluid clearance and Na(+),K(+)-ATPase activity
34 ce to lung injury, db/db mice had diminished alveolar fluid clearance and reduced Na,K-ATPase functio
35                  In isolated perfused lungs, alveolar fluid clearance and secretion were determined b
36 nce, fibrinogenesis, inflammatory cytokines, alveolar fluid clearance, and endothelial injury and act
37 panied by a 2.4-fold increase in the rate of alveolar fluid clearance at 4 hrs in the salmeterol-trea
38       Recently, we showed that impairment of alveolar fluid clearance because of inhibition of epithe
39 with control, LTD4 (1 x 10(-11) M) increased alveolar fluid clearance by 41% (p < 0.001) in isolated,
40 in BALB/c mice increased amiloride-sensitive alveolar fluid clearance by approximately 30%, consisten
41 ung and restored the normal up-regulation of alveolar fluid clearance by catecholamines after prolong
42 e lung prevented the normal up-regulation of alveolar fluid clearance by catecholamines following hem
43                                              Alveolar fluid clearance contributes to graft function a
44                                       Intact alveolar fluid clearance correlated with less histologic
45                    Because a decrease in net alveolar fluid clearance could be due to lung endothelia
46                                              Alveolar fluid clearance driven by active epithelial Na(
47                 The two patients with no net alveolar fluid clearance had persistent hypoxemia and mo
48                        Patients with maximal alveolar fluid clearance had significantly lower mortali
49 edema and acute lung injury, we measured net alveolar fluid clearance in 79 patients with acute lung
50 ring the beta- adrenergic agonist-stimulated alveolar fluid clearance in acute lung injury, an effect
51                               The unimpaired alveolar fluid clearance in AQP5-null mice indicates tha
52 nfection has been shown to reduce Na+-driven alveolar fluid clearance in BALB/c mice in vivo.
53 g injury and the loss of claudin-4 decreases alveolar fluid clearance in mice.
54  in contrast to hydrostatic pulmonary edema, alveolar fluid clearance in patients with acute lung inj
55 K-ATPase in alveolar epithelial cells and on alveolar fluid clearance in rat lungs.
56                            The high rates of alveolar fluid clearance indicate that the fluid transpo
57 red in the majority of patients, and maximal alveolar fluid clearance is associated with better clini
58                                   Airway and alveolar fluid clearance is mainly governed by vectorial
59                            However, impaired alveolar fluid clearance is present in most of the patie
60            Of the patients, 56% had impaired alveolar fluid clearance (&lt; 3%/h), 32% had submaximal cl
61                               Measurement of alveolar fluid clearance may be useful to assess the sev
62 ins (including ZO-1) was not associated with alveolar fluid clearance or claudin-4 levels.
63 nction protein claudin-4 are associated with alveolar fluid clearance or clinical measures of lung fu
64 3) and in patients with an absence of intact alveolar fluid clearance (p =.03).
65                       Maximal cAMP-dependent alveolar fluid clearance rate was 32.9 +/- 10.9 %/hr (p
66                                              Alveolar fluid clearance rates were measured in ex vivo
67 human lungs, mesenchymal stem cells restored alveolar fluid clearance, reduced inflammation, and exer
68 ing demonstrated a positive correlation with alveolar fluid clearance (Spearman rank correlation [r(s
69     Concomitantly, we observed a reversal of alveolar fluid clearance, suggesting that reversed trans
70 ts with acute lung injury (ALI) have reduced alveolar fluid clearance that has been associated with h
71 , and isoproterenol 10(-6) M each stimulated alveolar fluid clearance to a level comparable to maxima
72 -grade human mesenchymal stem cells restored alveolar fluid clearance to a normal level, decreased in
73  across the alveolar epithelium and restored alveolar fluid clearance to normal.
74 th hydrostatic pulmonary edema, in whom mean alveolar fluid clearance was 13%/h; only 25% had impaire
75                                         Mean alveolar fluid clearance was 6%/h.
76                                        Basal alveolar fluid clearance was 7.6 +/- 2.2 %/hr.
77                                          Net alveolar fluid clearance was calculated from sequential
78                                              Alveolar fluid clearance was calculated from serial samp
79                   Compared with basal rates, alveolar fluid clearance was increased by both racemic a
80                                              Alveolar fluid clearance was measured by change in conce
81                                              Alveolar fluid clearance was measured in anesthetized, v
82                                              Alveolar fluid clearance was measured in living ventilat
83                                              Alveolar fluid clearance was measured in vivo.
84 r without electroporation, and 3 days later, alveolar fluid clearance was measured.
85                                              Alveolar fluid clearance was preserved in SP-C-GM mice i
86               Acute lung injury with maximal alveolar fluid clearance were more likely to be female (
87 eduction of Na,K-ATPase expression decreases alveolar fluid clearance, which in turn leads to pulmona

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