ライフサイエンスコーパス: alveolar macrophage

1 the amount of cellular H2O2 in M2 polarized alveolar macrophages.

2 lung epithelial cells, dendritic cells, and alveolar macrophages.

3 C subset, but not other lung CD11b(+) DCs or alveolar macrophages.

4 r signaling in the pulmonary endothelium and alveolar macrophages.

5 ch provides a useful in vitro model to study alveolar macrophages.

6 matory cytokine responses of responder human alveolar macrophages.

7 d preventing overresponse to TLR4 ligands in alveolar macrophages.

8 iminished release of infectious virions from alveolar macrophages.

9 es in the lung, notably, dendritic cells and alveolar macrophages.

10 disease, infects and replicates within human alveolar macrophages.

11 transmission to humans, the bacteria hijack alveolar macrophages.

12 ion of markers for alternative activation on alveolar macrophages.

13 least in part by inhibiting phagocytosis by alveolar macrophages.

14 d to acid resulted in suppression of iNOS in alveolar macrophages.

15 s by murine peritoneal macrophages and human alveolar macrophages.

16 ygen species-mediated killing of bacteria by alveolar macrophages.

17 tant accumulation from impaired clearance in alveolar macrophages.

18 came increasingly similar to tissue-resident alveolar macrophages.

19 oduction of reactive oxygen species (ROS) by alveolar macrophages.

20 artment or changes in the number of resident alveolar macrophages.

21 e the bacteria first encounter lung-resident alveolar macrophages.

22 rculosis in humans and predominantly infects alveolar macrophages.

23 aused proinflammatory cytokine production in alveolar macrophages.

24 uorescent particles showed reduced uptake by alveolar macrophages.

25 tiologic agent of TB, usually resides in the alveolar macrophages.

26 cally downregulates cell cycling pathways in alveolar macrophages.

27 sive genes in murine bone marrow-derived and alveolar macrophages.

28 is required for the perinatal development of alveolar macrophages.

29 ies promote opsonophagocytosis of bacilli by alveolar macrophages.

30 ition from prealveolar macrophages to mature alveolar macrophages.

31 Alveolar macrophages, a major host cell niche for M. tub

32 Selective depletion of alveolar macrophages abolished the PGD2-enhanced inflamm

33 Alveolar macrophages acquired a classical activation (M1

34 In primary human alveolar macrophages, administration of a beta2AR agonis

35 adsA strain induced sPLA2-IIA expression by alveolar macrophages after phagocytic process via NOD2-N

36 pecific genetic deletion of monocyte-derived alveolar macrophages after their recruitment to the lung

37 s both DP1 and DP2 receptors were located on alveolar macrophages along with hematopoietic PGD syntha

38 usceptibility may be caused by impairment of alveolar macrophage (AM) function and/or mycobacteria-sp

39 sted the role of Stat5 in dendritic cell and alveolar macrophage (AM) homeostasis in the lung using C

40 The effect of COPD on alveolar macrophage (AM) microbicidal responses was inve

41 Alveolar macrophages (AM) are at the center of the patho

42 Abnormal alveolar macrophages (AM) are found in chronic obstructi

43 ns of tissue resident macrophages, including alveolar macrophages (AM), in cancer were not well studi

44 interleukin-1alpha (IL-1alpha) from necrotic alveolar macrophages (AM), which activated endothelial c

45 part through frontline epithelial cells and alveolar macrophages (AM).

46 f apoptotic cells (ACs) ("efferocytosis") by alveolar macrophages (AMos) reduces their ability to com

47 Toll-like receptors in alveolar macrophages (AMPhi) recognize the molecular con

48 onsible for activation of p38 in sarcoidosis alveolar macrophages (AMs) and PBMCs.

49 Human alveolar epithelial cells (AECs) and alveolar macrophages (AMs) are the first lines of lung d

50 The role and origin of alveolar macrophages (AMs) in asthma are incompletely de

51 Alveolar macrophages (AMs) obtained by bronchoalveolar l

52 In the lung, resident alveolar macrophages (AMs) provide a sentinel function a

53 Alveolar macrophages (AMs) reside on the luminal surface

54 ized that S. aureus impairs efferocytosis by alveolar macrophages (AMs) through the activity of the s

55 ken to determine the susceptibility of human alveolar macrophages (AMs) to influenza A virus (IAV) in

56 In this study, the phagocytic activity of alveolar macrophages (AMs) was found to decrease by 40%

57 Murine alveolar macrophages (AMs) were cultured ex vivo with/wi

58 e found to express high levels of PD-L1, and alveolar macrophages (AMs) were found to express high le

59 ry microvascular endothelial cells (PMVECs), alveolar macrophages (AMs), and polymorphonuclear leukoc

60 s harvested by bronchoalveolar lavage (BAL), alveolar macrophages (AMs), are routinely used in studie

61 to invade due to virus-induced depletion of alveolar macrophages (AMs), but this is not the only con

62 Professional phagocytes, including alveolar macrophages (AMs), have specialized pathways th

63 tes such as aluminum salts and silica killed alveolar macrophages (AMs), which then released interleu

64 lveolar epithelial cells (AECs) and resident alveolar macrophages (AMs).

65 red IFN responses to rhinovirus by asthmatic alveolar macrophages (AMs); the molecular mechanisms und

66 histocompatibility complex class II(+)) and alveolar macrophages (AMs; CD11c(+) sialic acid-binding

67 SP-A affects IFN-gamma-induced activation of alveolar macrophages (aMvarphis) remains unknown.

68 ell subsets in the lungs, including resident alveolar macrophages and 4 types of cells that recruited

69 eta2-adrenergic receptor (beta2AR) on murine alveolar macrophages and augment the release of IL-6.

70 Both in vivo and in cultured alveolar macrophages and BMDM, expression of several M1

71 ureus and B. anthracis compared with E. coli Alveolar macrophages and CD14(+) cells were overall more

72 P12 promotes the survival of tissue-resident alveolar macrophages and contributes to local production

73 ency in the AMPK catalytic alpha1 subunit in alveolar macrophages and conventional dendritic cells pr

74 , R848 induced production of IL-27 by murine alveolar macrophages and dendritic cells and enhanced ex

75 n of innate immunity mediators, initiated by alveolar macrophages and dependent on transcription driv

76 proinflammatory cytokine production in both alveolar macrophages and epithelial cells.

77 (fl/fl)) exhibited significant reductions in alveolar macrophages and failed to effectively clear pul

78 hus, aging induces defective phagocytosis by alveolar macrophages and increases lung damage.

79 Legionella pneumophila infects human alveolar macrophages and is responsible for Legionnaire'

80 sis (PAP), we evaluated lipid composition in alveolar macrophages and lung surfactant, macrophage-med

81 entify TRIM29 as a key negative regulator of alveolar macrophages and might have important clinical i

82 d proinflammatory cytokine response of human alveolar macrophages and more severe inflammatory lung d

83 FN-gamma blocked EHV-1 replication in murine alveolar macrophages and mouse lungs and protected mice

84 Alveolar macrophages and neutrophils also express A1-Ado

85 Collectively, our data suggest that alveolar macrophages and neutrophils are both an intrace

86 Our data reveal that alveolar macrophages and neutrophils both are the primar

87 Moreover, both alveolar macrophages and neutrophils from infected mice

88 Resistance was mediated by alveolar macrophages and neutrophils, and was associated

89 y dendritic cells is to inhibit apoptosis of alveolar macrophages and neutrophils, which sequester th

90 racellular reactive oxygen species levels in alveolar macrophages and neutrophils.

91 infected, most prominently CD45(neg) cells, alveolar macrophages and neutrophils.

92 In humans, C. burnetii infects alveolar macrophages and promotes phagosomal fusion with

93 FN-gamma inhibited EHV-1 infection of murine alveolar macrophages and protected mice against lethal E

94 sion studies demonstrated robust recovery of alveolar macrophages and recruitment of CD4+ lymphocytes

95 by mediating both chemokine production from alveolar macrophages and T cell trafficking.

96 show for the first time TREK-1 expression on alveolar macrophages and unimpaired tumor necrosis facto

97 found principally in type I and II cells and alveolar macrophages and was also detected in vascular e

98 population shared many characteristics with alveolar macrophages and was retained in the alveolar sp

99 ate critical regulators of the generation of alveolar macrophages and, thus, effective pulmonary inna

100 y inflammation, alternative M2 activation of alveolar macrophage, and increased serum IgE.

101 n human macrophage-like cells, primary human alveolar macrophages, and Chinese hamster ovary cells.

102 was examined in murine peritoneal and human alveolar macrophages, and its mechanisms were investigat

103 ) mice had significantly less active Rac1 in alveolar macrophages, and macrophages from Akt(+/-) mice

104 is required for phenotypic determination of alveolar macrophages, and support translation of PMT as

105 that Akt1-mediated mitophagy contributes to alveolar macrophage apoptosis resistance and is required

106 that lack MyD88 only in dendritic cells and alveolar macrophages are competent for early cytokine pr

107 Alveolar macrophages are critical effectors for CD4(+) T

108 In summary, alveolar macrophages are critical regulators of Th2 immu

109 Alveolar macrophages are found in the alveoli of the lun

110 Alveolar macrophages are lung-resident sentinel cells th

111 mune cells present in the respiratory tract, alveolar macrophages are poised to defend against hantav

112 Alveolar macrophages are sentinels of the pulmonary muco

113 Alveolar macrophages are the first line of defense again

114 Macrophages, including alveolar macrophages, are primary phagocytic cells of th

115 scence-activated cell sorting, we identified alveolar macrophages as the first cell type infected in

116 ite burden, and increased numbers of CD68(+) alveolar macrophages as well as apoptotic cells in the l

117 Alveolar macrophages at both day 4 and 14 and IMs at day

118 acologic inhibition of the beta2AR on murine alveolar macrophages attenuated PM-induced IL-6 release

119 LPS-induced inflammatory response in bovine alveolar macrophages (bAMs).

120 phils, lung- and lamina propria-resident and alveolar macrophages, bone marrow-derived DCs, and lung-

121 Viral DNA isolated from blood monocytes and alveolar macrophages (but not T cells) of drug-suppresse

122 posomes resulted in significant reduction in alveolar macrophages, but depletion did not prevent path

123 o isoforms 5a and 5b) is highly expressed in alveolar macrophages, but its function there is unclear

124 In human alveolar macrophages, C. burnetii uses a Dot/Icm type IV

125 Here we report that alveolar macrophages can secrete SOCS1 and -3 in exosome

126 Silica taken up by alveolar macrophages causes phagolysosomal membrane dama

127 llular bacterial pathogen that replicates in alveolar macrophages, causing a severe form of pneumonia

128 hila is a bacterial pathogen that thrives in alveolar macrophages, causing a severe pneumonia.

129 Small alveolar macrophage CD206 expression was lower in COPD p

130 led DK128 was correlated with an increase in alveolar macrophage cells in the lungs and airways, earl

131 single-cell metabolomic profiling using rat alveolar macrophage cells incubated with different conce

132 id not affect the viral replication in swine alveolar macrophage cells.

133 With aging, we found reduced numbers of alveolar macrophages, cells essential for lung homeostas

134 challenge correlated with reduced numbers of alveolar macrophages, consistent with a critical role fo

135 e imaging showed rapid uptake of spores into alveolar macrophages, conversion to yeast, and intracell

136 cation in MARC-145 cells and primary porcine alveolar macrophages could also be reversed by overexpre

137 Depletion of alveolar macrophages curtailed infection in mice infecte

138 ondiabetic recipients confirmed an intrinsic alveolar macrophage defect that hindered T-cell priming.

139 ue interstitial macrophages were elevated in alveolar macrophage-deficient mice identifying a new hom

140 Alveolar macrophage-dependent and -independent (calculat

141 um size, bacterial replication, capsule, and alveolar macrophage-dependent and -independent clearance

142 me increased to 56 min and the S. pneumoniae alveolar macrophage-dependent clearance half-life improv

143 veolar macrophage-independent and 31 min for alveolar macrophage-dependent clearance of unencapsulate

144 half-life of 24 min compared to 135 min for alveolar macrophage-dependent clearance.

145 nfection was exacerbated under conditions of alveolar macrophage depletion and in mice with a macroph

146 ated with global monocyte or tissue-resident alveolar macrophage depletion.

147 Adoptive transfer of alveolar macrophages derived from Abx-treated mice was s

148 Alveolar macrophage-derived MVs were fully internalized

149 We thus identify LPL as being key to alveolar macrophage development and essential to an effe

150 ntify a molecular pathway governing neonatal alveolar macrophage development and show that genetic di

151 We next identified the phase of alveolar macrophage development requiring LPL.

152 elopment and show that genetic disruption of alveolar macrophage development results in immunodeficie

153 Rgamma is known to promote M2-macrophage and alveolar macrophage development.

154 rated lung fibrosis, whereas tissue-resident alveolar macrophages did not contribute to fibrosis.

155 During the fibrotic phase, monocyte-derived alveolar macrophages differ significantly from tissue-re

156 flow-sorted cells, we found that monocyte to alveolar macrophage differentiation unfolds continuously

157 findings suggest that selectively targeting alveolar macrophage differentiation within the lung may

158 Alveolar macrophages displayed gene expression for, cont

159 ic genes expressed by mouse monocyte-derived alveolar macrophages during fibrosis were up-regulated i

160 Similarly, depletion of alveolar macrophages during house dust mite sensitizatio

161 feration and recruitment of perivascular and alveolar macrophages during hypoxia exposure.

162 Furthermore, while Atg5 is dispensable in alveolar macrophages during M. tuberculosis infection, l

163 icrobial pathogens, other sentinels, such as alveolar macrophages, epithelial cells, dendritic cells,

164 A majority of their alveolar macrophages exhibit lysosomal accumulations of

165 Diabetic alveolar macrophages exhibited reduced phagocytosis of M

166 eaction in mammalian macrophages (NR8383 rat alveolar macrophages) exposed to a centrifuge regime of

167 Similar to adult alveolar macrophages, fetal lung macrophages responded t

168 Although the importance of alveolar macrophages for host immunity during early Stre

169 ntrinsically required in dendritic cells and alveolar macrophages for initial cytokine production.

170 Alveolar macrophages from acid-injured Akt2(-/-) mice de

171 Alveolar macrophages from diabetic mice had reduced expr

172 Transfer of infected alveolar macrophages from diabetic mice into nondiabetic

173 We tested the capacity of alveolar macrophages from diabetic mice to phagocytose M

174 Alveolar macrophages from female mice exhibited greater

175 that IL-4-stimulated bone marrow-derived and alveolar macrophages from female mice exhibited greater

176 s during fibrosis were up-regulated in human alveolar macrophages from fibrotic compared with normal

177 xposed mice showed fibrosis development, and alveolar macrophages from fibrotic mice showed increased

178 This study is the first to demonstrate that alveolar macrophages from HCMV carriers express immediat

179 Alveolar macrophages from KGF-treated mice also exhibite

180 Moreover, alveolar macrophages from MARCO(-/-) mice polarize to an

181 (PiZZ) and healthy (PiMM) individuals and in alveolar macrophages from normal (60 mg/kg) and high-dos

182 ung histopathology characteristic of PAP; 2) alveolar macrophages from Rasgrp1-deficient mice are enl

183 ying these transcriptional programs to human alveolar macrophages from smokers and patients with chro

184 Alveolar macrophages from toll-like receptor 3 (-/-) mic

185 ure increased mitochondrial Ca(2+) influx in alveolar macrophages from wild-type, but not MCU(+/-), m

186 A production by CD103(+) dendritic cells and alveolar macrophages functions with TGF-beta to promote

187 Molecular mechanisms controlling alveolar macrophage generation have not been fully defin

188 Moreover, IPF alveolar macrophages had evidence of increased mitophagy

189 Alveolar macrophages had similar numbers of small and la

190 Small alveolar macrophages had the highest phagocytic ability.

191 ammatory gene expression levels, while large alveolar macrophages had the lowest.

192 We recently characterized human alveolar macrophage (hAM) infection in vitro and found t

193 Alveolar macrophages have emerged as major mediators of

194 ed neutralization of GM-CSF thereby inhibits alveolar macrophage homeostasis and function, leading to

195 ed metabolic and functional studies on human alveolar macrophages, human monocyte-derived macrophages

196 lung inflammation and significantly reduced alveolar macrophage IL-10 production.

197 WTI induced a pronounced reduction of alveolar macrophages in both strains that recovered with

198 s not sufficiently upregulated in developing alveolar macrophages in LPL(-/-) pups, suggesting that p

199 e a key role for pro-IL-1alpha from necrotic alveolar macrophages in LPS-mediated ALI, as a critical

200 abolism and accumulation of CD103(+) DCs and alveolar macrophages in lung.

201 role for DAP12 expression in tissue-resident alveolar macrophages in mediating acute noninfectious ti

202 We demonstrate that CLEC5A is expressed on alveolar macrophages in mice exposed long-term to cigare

203 ic airway disease to investigate the role of alveolar macrophages in regulating pulmonary inflammatio

204 L. pneumophila is able to infect alveolar macrophages in the lung and replicates intracel

205 es differ significantly from tissue-resident alveolar macrophages in their expression of profibrotic

206 nisms, with reduced half-lives of 14 min for alveolar macrophage-independent and 31 min for alveolar

207 ize, the capsule, and rapid replication) and alveolar macrophage-independent clearance mechanisms dur

208 dose inoculum of encapsulated S. pneumoniae, alveolar macrophage-independent clearance mechanisms wer

209 Murine alveolar macrophages infected with DeltakatG display abn

210 e exogenous addition of 14-oxoDHA to primary alveolar macrophages inhibited LPS-induced proinflammato

211 Transplantation of infected Cftr-deficient alveolar macrophages into the lungs of noninfected CF mi

212 , although mitochondrial oxidative stress in alveolar macrophages is critical for fibrosis developmen

213 Mitochondrial oxidative stress in alveolar macrophages is directly linked to pulmonary fib

214 Alternative activation of alveolar macrophages is linked to fibrosis following exp

215 eviously shown that the release of IL-6 from alveolar macrophages is required for a prothrombotic sta

216 us, autophagy in myeloid cells, particularly alveolar macrophages, is critical for inhibiting spontan

217 Compared with normal subjects, alveolar macrophages isolated from patients with asbesto

218 sine downregulated S. aureus phagocytosis by alveolar macrophages, leading to inhibition of sPLA2-IIA

219 MARC-145 cells and primary porcine pulmonary alveolar macrophages led to significant reduction of STA

220 ion, mucus production, serum lgE levels, and alveolar macrophage M2 activation in STUB1(-/-) mice.

221 activity and solubility-were studied in rat alveolar macrophages (MAC) and epithelial cells (AEC).

222 nally replaced in one-year-old mice, whereas alveolar macrophages may be progressively replaced in ag

223 that therapies that enhance the function of alveolar macrophages may improve outcomes in older peopl

224 These data demonstrate that alveolar macrophages may play a limited protective role

225 opment of fetal lung macrophages into mature alveolar macrophages may therefore include features of b

226 lso reveal the potential mechanisms by which alveolar macrophages mediate protection in vivo, namely

227 due to its intrinsic resistance to resident alveolar macrophage-mediated intracellular killing.

228 A1AT protein activated PP2A in human alveolar macrophages, monocytes, neutrophils, airway epi

229 Here, we examine the roles of alveolar macrophages, natural killer cells, and neutroph

230 hat, like in our prior T and B cell studies, alveolar macrophages neither prevent hantavirus infectio

231 poxemia, pulmonary edema, and levels of BALF alveolar macrophages, neutrophils, IFN-gamma, and IL-10

232 ion is characterized by increased numbers of alveolar macrophages, neutrophils, T lymphocytes (predom

233 g that immunodeficiency results from reduced alveolar macrophage numbers.

234 cells of patients with COPD and in isolated alveolar macrophages of patients with COPD or IPF.

235 wide variety of natural amoebal hosts and in alveolar macrophages of the human accidental host.

236 lia), epidermis (Langerhans cells) and lung (alveolar macrophages) originate from a Tie2(+) (also kno

237 CD163, C2+ and WSL, were compared to porcine alveolar macrophage (PAM) in terms of surface marker phe

238 In this study, we established the porcine alveolar macrophages (PAM) cells model co-infected with

239 Infection of primary pulmonary alveolar macrophages (PAMs) also induces interferons.

240 dy compared the interactions between porcine alveolar macrophages (PAMs) and wild-type A. pleuropneum

241 Immunophenotyping of porcine alveolar macrophages (PAMs) showed that pigs with the KO

242 Alveolar macrophages participate in the pathogenesis of

243 PMT-derived alveolar macrophages persisted for at least one year as

244 A population of monocyte-derived alveolar macrophages persisted in the lung for one year

245 Moreover, aging decreases alveolar macrophage phagocytosis of apoptotic neutrophil

246 The diabetic alveolar macrophage phenotype depended in part on expres

247 This alveolar macrophage phenotype was absent in peritoneal a

248 We demonstrate that alveolar macrophages play a dominant role in conferring

249 To determine whether alveolar macrophages play a role in HPS pathogenesis, al

250 unications between lung epithelial cells and alveolar macrophages play an essential role in host defe

251 Despite the fact that alveolar macrophages play an important role in smoking-r

252 Our findings identify mechanisms regulating alveolar macrophage population size in health and diseas

253 cesses essential for correct localization of alveolar macrophage precursors: (1) transmigration into

254 This study implies that alveolar macrophages produce IL-26, which stimulates rec

255 Human alveolar macrophages produced CCL2 in a PGL-dependent fa

256 an immunologically prime the lung to augment alveolar macrophage production of IL-10 and enhance reso

257 ontributes to defective sentinel function of alveolar macrophages, promoting tuberculosis susceptibil

258 ught to address the contribution of resident alveolar macrophages (rAMs) to susceptibility to RSV inf

259 s altered in endotoxemic hepSTAT3(-/-) mice, alveolar macrophage reactive oxygen species generation w

260 However, efficient alveolar macrophage regeneration following irradiation r

261 We assessed the importance of alveolar macrophages, regulatory T cells, and their pote

262 dap12 alveolar macrophages released more tumor necrosis factor

263 ng the defective protein degradation in hPAP alveolar macrophages remains poorly understood.

264 he transition from prealveolar macrophage to alveolar macrophage requires the upregulation of the tra

265 orne bacilli are inhaled and phagocytosed by alveolar macrophages, resulting in the formation of a gr

266 Studies on smoker alveolar macrophages showed a defect in miRNA maturation

267 Large alveolar macrophages showed lower marker expression in C

268 Aside from alveolar macrophages, subsets of Langerin(+), BDCA1(-)CD

269 roduce the molecular and cellular defects of alveolar macrophages that drive the pathogenesis of PAP

270 rk between infected and noninfected AECs and alveolar macrophages that leads to decreased alveolar ep

271 differentiation, survival, and activation of alveolar macrophages, the cells responsible for surfacta

272 s a selective regulator of the activation of alveolar macrophages, the expression of type I interfero

273 lungs during a late fetal stage, maturing to alveolar macrophages through a prealveolar macrophage in

274 stimulates pathogen killing and clearance by alveolar macrophages through extracellular signal-regula

275 therapy further augmented these responses in alveolar macrophages through generation of mitochondrial

276 Th-17 lymphocytes and, conversely, a blunted alveolar macrophage TLR4 response.

277 Alveolar macrophage TNFalpha production was unaltered.

278 Aging impairs the ability of alveolar macrophages to limit lung damage during influen

279 e, phosphatidylserine, and ceramide) and rat alveolar macrophages to show how lipid bilayer propertie

280 localized in an intracellular compartment of alveolar macrophages together with SP-D.

281 l (SAE) cell differential and transcriptome, alveolar macrophage transcriptome, and plasma apoptotic

282 d and basal cells, markedly abnormal SAE and alveolar macrophage transcriptomes, and elevated levels

283 These findings suggest that alveolar macrophages undergo ER stress, which is associa

284 Alveolar macrophages undergo productive infection and re

285 lease, and evaluated for in vivo absorption, alveolar macrophage uptake, and safety.

286 nflammation was enhanced in mice depleted of alveolar macrophages using clodronate liposomes.

287 (A549 and primary human airway epithelia and alveolar macrophages) using chemical inhibition and shRN

288 Finally, we found that the role of alveolar macrophages was also dependent on the genetic b

289 In addition, ex vivo responsiveness of alveolar macrophages was examined.

290 ) dendritic cells (DCs), but not PD-L1(high) alveolar macrophages, was dependent on IFNAR signaling.

291 In addition to a substantial population of alveolar macrophages, we identified subpopulations of mo

292 macrophages play a role in HPS pathogenesis, alveolar macrophages were depleted in an adult rodent mo

293 e is caused by a functional insufficiency of alveolar macrophages, which require GM-CSF signaling for

294 Aerosolized H5N1 exposure decimated alveolar macrophages, which were widely infected and cau

295 Depletion of alveolar macrophages with inhaled clodronate liposomes r

296 phagocytic function respectively, and large alveolar macrophages with low pro-inflammatory and phago

297 subpopulations; Small interstitial and small alveolar macrophages with more pro-inflammatory and phag

298 alveolar epithelia (type I and II cells) and alveolar macrophages with similar trends in reactive mes

299 Most spores were located inside of alveolar macrophages, with smaller numbers in neutrophil

300 lodronate-sensitive, phagocytic SiglecF(low) alveolar macrophages within the airways following ALI.