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1  from six patients with idiopathic pulmonary alveolar proteinosis.
2 with chronic inflammation, and two had focal alveolar proteinosis.
3 rupt surfactant clearance, causing pulmonary alveolar proteinosis.
4 bodies isolated from patients with pulmonary alveolar proteinosis.
5 contributor to the pathogenesis of pulmonary alveolar proteinosis.
6 omatous inflammation leading to fibrosis and alveolar proteinosis.
7 tic of atherosclerotic lesions and pulmonary alveolar proteinosis.
8 n 79% and ventilatory defects 63%, pulmonary alveolar proteinosis 18%, pulmonary arterial hypertensio
9        We studied 12 subjects with pulmonary alveolar proteinosis, 61 healthy control subjects, and 1
10 t a lung disorder similar to human pulmonary alveolar proteinosis, a rare disease with congenital, in
11 fluids from healthy donors and patients with alveolar proteinosis also binds to M.tb.
12 ions are impaired in patients with pulmonary alveolar proteinosis and that GM-CSF autoantibodies caus
13 e syndrome, cigarette smoking, and pulmonary alveolar proteinosis and the implications of obesity and
14 plasia, cytogenetic abnormalities, pulmonary alveolar proteinosis, and myeloid leukemias.
15  atherosclerosis, type 2 diabetes, pulmonary alveolar proteinosis, and obesity, have a chronic inflam
16 uman GM-CSF and IL-3 and developed pulmonary alveolar proteinosis because of elimination of mouse GM-
17 are linked to the pathogenesis of congenital alveolar proteinosis (CAP).
18  polyendocrine syndrome type I and pulmonary alveolar proteinosis, detecting ACAA levels consistent w
19 ptible to respiratory infections and develop alveolar proteinosis due to defects in innate immune fun
20 ant human Prdx6 and SP-A isolated from human alveolar proteinosis fluid were studied.
21     Neutrophils from subjects with pulmonary alveolar proteinosis had normal ultrastructure and diffe
22                         Hereditary pulmonary alveolar proteinosis (hPAP) caused by granulocyte-macrop
23 l disorder identical to hereditary pulmonary alveolar proteinosis (hPAP) in children with CSF2RA or C
24 -/- mice resulting in complete correction of alveolar proteinosis in bitransgenic GM-/-, SP-C-GM+ mic
25  functional and prevented the development of alveolar proteinosis in mice transplanted with GM-Csf-re
26 CSF) gene by homologous recombination caused alveolar proteinosis in mice.
27 monary chronic interstitial inflammation and alveolar proteinosis, inflammation of the glandular stom
28                         Idiopathic pulmonary alveolar proteinosis is caused by autoantibodies against
29         Mice lacking beta c show a pulmonary alveolar proteinosis-like disease and reduced numbers of
30    Both human SP-A (isolated from normal and alveolar proteinosis lungs) and SP-D (recombinant protei
31 ecombinant protein and protein isolated from alveolar proteinosis lungs) bound the conidia.
32 bronchiolitis obliterans (n = 21), pulmonary alveolar proteinosis (n = 12), pulmonary fibrosis (n = 1
33 ty from infection in patients with pulmonary alveolar proteinosis occurs in association with high lev
34 e at risk for later development of pulmonary alveolar proteinosis or other opportunistic infections,
35 hils are impaired in patients with pulmonary alveolar proteinosis, owing to the presence of GM-CSF au
36 erved that it is elevated in human pulmonary alveolar proteinosis (PAP) and in the GM-CSF knockout mo
37 -) mice, respectively) resulted in pulmonary alveolar proteinosis (PAP) but no hematologic abnormalit
38 rize the frequency and features of pulmonary alveolar proteinosis (PAP) in patients with ADA deficien
39                                    Pulmonary alveolar proteinosis (PAP) is a rare disease characteriz
40                                    Pulmonary alveolar proteinosis (PAP) is a rare disorder in which s
41                                    Pulmonary alveolar proteinosis (PAP) is a rare lung disease charac
42                                    Pulmonary alveolar proteinosis (PAP) is a rare lung disorder in wh
43                                    Pulmonary alveolar proteinosis (PAP) is a rare lung syndrome cause
44                                    Pulmonary alveolar proteinosis (PAP) is a rare syndrome characteri
45                            Primary pulmonary alveolar proteinosis (PAP) is a rare syndrome characteri
46                                    Pulmonary alveolar proteinosis (PAP) is an idiopathic lung disease
47                   In patients with pulmonary alveolar proteinosis (PAP) syndrome, disruption of granu
48 utoantibodies are thought to cause pulmonary alveolar proteinosis (PAP), a rare syndrome characterize
49 d tomography is characteristic for pulmonary alveolar proteinosis (PAP), it is not specific and has n
50 asis and how its disruption causes pulmonary alveolar proteinosis (PAP), we evaluated lipid compositi
51 cal meningitis who later developed pulmonary alveolar proteinosis (PAP).
52 imicrobial function in patients with primary alveolar proteinosis (PAP).
53 lineage cells and severe secondary pulmonary alveolar proteinosis (PAP).
54 ophages that partially rescued the pulmonary alveolar proteinosis syndrome.
55 heir mRNAs, distinguishing the disorder from alveolar proteinosis syndromes.
56 phil dysfunction characteristic of pulmonary alveolar proteinosis was reproduced in a dose-dependent
57 ce of Stat5 signaling in AMs, mice developed alveolar proteinosis with altered lipid homeostasis.
58            Five patients developed pulmonary alveolar proteinosis without mutations in the granulocyt

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