1 were frequently in the interior of thickened alveolar septa.

2 , was primarily found at the free margins of alveolar septa.

3 c-expressing lung DCs, preferentially in the alveolar septa.

4 g the alveolar walls, and projected into the alveolar septa.

5 chyma shows hypercellularity, with thickened alveolar septa and an increase in the number of endothel

6 issue) shows hypercellularity with thickened alveolar septa and an increase in the number of vascular

7 he only vessels present were the ones in the alveolar septa, and their presence highlighted, through

8 malformed, dilated, muscular arteries within alveolar septa; congested capillaries within alveolar wa

9 everely reduced capillary density, thickened alveolar septa containing centrally located dilated capi

10 displacement, destruction, or replacement of alveolar septa, distal airways, and small vessels within

11 ormation of the elastin ECM, thereby driving alveolar septa formation to increase the gas-exchange su

12 e nucleic acid (microg) was increased in the alveolar septa of emphysema lungs (14.2 +/- 2.0/microg,

13 wed marked inflammation and edema within the alveolar septa of SNV-infected hamsters, results which a

14 ed to correlate changes in components of the alveolar septa surrounding enlarged airspaces in human e

15 haracterized by the formation of millions of alveolar septa that constitute the vast gas-exchange sur

16 lveolar epithelial type I and II cells, form alveolar septa, the functional units in the lung.