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1 anced tidal volume recruitment, and improved alveolar ventilation.
2 to a complete cessation or even inversion of alveolar ventilation.
3 matching between pulmonary gas exchange and alveolar ventilation.
4 inute ventilation during exercise maintained alveolar ventilation and arterial blood gas homeostasis
5 O(2) levels) occurs as a consequence of poor alveolar ventilation and impairs alveolar fluid reabsorp
6 re effectively as a pump, thereby increasing alveolar ventilation and reducing baseline resting PaCO2
7 eduction in tidal volume without a change in alveolar ventilation, and (3) may be a useful adjunct to
8 ition, the vertical distribution of specific alveolar ventilation became more uniform in the prone po
9 Some suggest this results from an increased alveolar ventilation, but others imply that ventilation-
10 s as an effective strategy to restore intact alveolar ventilation by a mechanism independent of alveo
11 of injury mode, ALI resulted in asynchronous alveolar ventilation characteristic of alveolar pendellu
12 with amplitude adjusted to maintain constant alveolar ventilation, gas flow through the chest tube wa
16 tory rate and included tidal volume, maximal alveolar ventilation, inspiratory and expiratory mean ar
17 derstand when improvement in oxygenation and alveolar ventilation is related to a lower degree or ris
18 AP) therapy on the relative distributions of alveolar ventilation ([OV0312]a) and perfusion ([OV0422]
19 1.8% on supplemental O2 [p < 0.00001]) while alveolar ventilation remained unchanged (PETCO2 > 50 mm
20 contributions from diffusion limitation and alveolar ventilation-to-perfusion inequality, the A-aDO2
21 sent a PET-based method to estimate regional alveolar ventilation-to-perfusion ratios (V(A)/Q) predic
22 ties (PMLE) elicits significant increases in alveolar ventilation (V A) in awake children with congen
24 s were within the normal range and effective alveolar ventilation was not significantly different fro
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