ライフサイエンスコーパス: alveolar wall

1 pectrum of injury and abnormal repair of the alveolar wall.

2 membrane to directly disseminate through the alveolar wall.

3 e lung to the most peripheral vessels in the alveolar wall.

4 hyperoxia-induced apoptosis of cells in the alveolar wall.

5 t localized in intrapulmonary airways and in alveolar walls.

6 nd with a decrease in apoptotic cells in the alveolar walls.

7 ury and caspase-3 activation in cells of the alveolar walls.

8 the epithelium of bronchi, bronchioles, and alveolar walls.

9 l airways, airway epithelium, submucosa, and alveolar walls.

10 ed pulmonary inflammation and destruction of alveolar walls.

11 modeling process in the connective matrix in alveolar walls.

12 alveolar surface area due to destruction of alveolar walls.

13 ha5 is prominent in the basement membrane of alveolar walls, airways, and pleura in developing and ad

14 nd overall inflammation in small airways and alveolar walls, along with alveolar wall thickening in 6

15 blast migration and proliferation within the alveolar wall and airspace after lung injury can lead to

16 n the lungs of CD18-/- mice by 68 and 35% in alveolar walls and by 28 and 26% in venous walls, respec

17 y, absence of TLOs attenuated destruction of alveolar walls and inflammation in bronchoalveolar lavag

18 zed initially by lymphocytic infiltration of alveolar walls and spaces, followed by an exuberant mono

19 incorporation of fibroblastic material into alveolar walls, and (c) cigarette smoke-related inflamma

20 ith a type II cell phenotype, grew along the alveolar walls, and projected into the alveolar septa.

21 alveolar septa; congested capillaries within alveolar walls; and congested capillaries within the wal

22 peroxia demonstrated only modest increase in alveolar wall apoptosis compared to room air.

23 prilysin had no effect on baseline airway or alveolar wall architecture, vessel density, cardiac func

24 tronger structural breakdown at the level of alveolar walls, as well as accelerated decay of stiffnes

25 expressed by epithelial cells that lined the alveolar walls, as well as by inflammatory cells and slo

26 rs pulmonary host defense, beyond effects on alveolar wall barrier function.

27 al bronchioles accompanied by destruction of alveolar walls, but without obvious fibrosis.

28 pase-3, Bax, and p53) were more prevalent in alveolar wall cells from the patients who died with ALI

29 aspase 3 demonstrated increased apoptosis in alveolar wall cells in wild-type mice in hyperoxia compa

30 eads to fixed narrowing of small airways and alveolar wall destruction (emphysema).

31 t adeno-associated virus markedly attenuated alveolar wall destruction and oxidative stress caused by

32 sema is a pulmonary disease characterized by alveolar wall destruction, resulting in enlargement of g

33 d inflammation in bronchoalveolar lavage and alveolar wall destruction.

34 The degree of alveolar space cellularity, alveolar wall fibrosis, and cellularity did not affect s

35 we show that a subset of AMs attached to the alveolar wall form connexin 43 (Cx43)-containing gap jun

36 BAL fluid probably reflect a high burden of alveolar wall granulomas and/or the removal of granuloma

37 ngs at day 4 revealed less disruption of the alveolar wall in SP-C-GM mice compared to wild-type mice

38 fter zoster revealed SVV antigen in the lung alveolar wall, in ganglionic neurons and nonneuronal cel

39 ter tail vein injection of tumor cells, with alveolar wall infiltration seen with AQP1-expressing tum

40 Emphysema causes a permanent destruction of alveolar walls leading to airspace enlargement, loss of

41 compartments, studies on the composition of alveolar wall liquid, and neutrophil and platelet traffi

42 Mechanical stress to alveolar walls may cause progressive damage after an ear

43 interstitial cells were demonstrated in the alveolar wall of normal Lewis rat lung.

44 valuate the binding kinetics of f-PPE in the alveolar walls of normal mouse lungs.

45 To explore the mechanism of alveolar wall remodeling after elastolytic injury, we ex

46 Multiple gross disruptions of the alveolar walls, suggestive of stress fractures.

47 e COPD: in spite of the destructive process, alveolar wall thickening and focal fibrosis may be detec

48 ulmonary neutrophil infiltration and reduced alveolar wall thickening by 45% (p < 0.05).

49 small airways and alveolar walls, along with alveolar wall thickening in 67 subjects undergoing lung

50 Histopathological correlates of acid injury (alveolar wall thickening, edema, and leukocyte infiltrat

51 prenatal hormone exposure was a decrease in alveolar wall thickness and an increase in aerated paren

52 ar myofibroblast proliferation and increased alveolar wall thickness compared with saline-treated con

53 Alveolar wall thickness was increased in all smokers, bu

54 ormone exposure, but there was a decrease in alveolar wall thickness with advancing gestation and at

55 cient mice exhibited significantly decreased alveolar wall thickness, inflammatory cell infiltration,

56 which Mycobacterium tuberculosis crosses the alveolar wall to establish infection in the lung is not

57 fibroblastic foci, an abnormal adjacency of alveolar walls was seen, suggesting alveolar collapse.

58 ent stages of bacterial crossing through the alveolar wall, we established a two-layer transwell syst

59 ficantly larger in stretched than in relaxed alveolar walls with a linear relation between k(off) and

60 sease in which fibroblasts accumulate in the alveolar wall within a type I collagen-rich matrix.