ライフサイエンスコーパス: alveolarization

1 both distal air and vascular tubulogenesis (alveolarization).

2 kely explanation for our finding is catch-up alveolarization.

3 gainst a model of lung expansion with no new alveolarization.

4 mice inhibits production of MMP14, impairing alveolarization.

5 ventricular hypertrophy, and decreased lung alveolarization.

6 ults in decreased inflammation and increased alveolarization.

7 lation-fixed for histopathologic analyses of alveolarization.

8 ronic lung disease characterized by arrested alveolarization.

9 cluding interstitial fibrosis and diminished alveolarization.

10 no inflammatory response and undergo normal alveolarization.

11 in antibodies reduce lung injury and promote alveolarization.

12 interstitial fibrosis but reduced defects in alveolarization.

13 st of lung development and interference with alveolarization.

14 ion, IL1beta and inflammation, and decreased alveolarization.

15 lecular defects were associated with blocked alveolarization and airway collapse in the lung.

16 hyperoxia-induced lung injury, with improved alveolarization and alveolar integrity compared with wil

17 Increased expression of TTF-1 altered alveolarization and caused chronic pulmonary inflammatio

18 SDF-1/CXCR4 axis significantly improved lung alveolarization and decreased pulmonary hypertension, ri

19 FOXA2 is required for alveolarization and regulates airway epithelial cell dif

20 Defects in alveolarization and vasculogenesis were observed in subs

21 unctions in mid-pulmonary patterning (during alveolarization), and is distinct from the Wnt canonical

22 In particular, defective alveolarization appears to be a common and potentially a

23 ological abnormalities: e.g., disrupted lung alveolarization, atrophy of intestinal villus and colon-

24 2 in mouse leads to defective postnatal lung alveolarization, contributing to postnatal lethality.

25 die in respiratory distress, with diminished alveolarization, decreased Shh, Fgf9, Fgf10, and Bmp4 mR

26 es demonstrate that premature birth disrupts alveolarization, decreasing the gas exchange surface are

27 istent with this, Cic deficiency causes lung alveolarization defect.

28 lial cell adhesion and migration, as well as alveolarization defects and persistent macrophage-mediat

29 es using liposomal clodronate, we found that alveolarization defects were secondary to persistent alv

30 mpletion of airway branching, we showed that alveolarization defects, characterized by disrupted seco

31 ce have hydrocephalus, omphalocele, and lung alveolarization defects.

32 If new alveolarization does not occur, alveolar size should inc

33 regulate mesenchymal development as well as alveolarization during the saccular phase of lung morpho

34 proliferation and differentiation as well as alveolarization during the saccular stage of lung develo

35 number of alveoli, but rather from increased alveolarization early in life.

36 Second, bombesin diminished alveolarization in C57BL/6 (but not Swiss-Webster) mice.

37 ctomy (PNX) model that promotes regenerative alveolarization in the remaining intact lung.

38 The current hypothesis that human pulmonary alveolarization is complete by 3 years is contradicted b

39 nsgene in the postnatal period did not alter alveolarization, lung size, or histology.

40 However, there is new evidence that human alveolarization might continue throughout childhood and

41 novel research aimed at promoting postnatal alveolarization offers a unique opportunity to develop e

42 veloping lung during the period of postnatal alveolarization, resulting in markedly enlarged parenchy

43 GRP, but only part of the bombesin effect on alveolarization, suggesting that novel receptors may med

44 demonstrate that a significant component of alveolarization, the final stage of lung development, oc

45 y 3 years is contradicted by new evidence of alveolarization throughout adolescence in mammals.

46 vironmental exposures could adversely affect alveolarization throughout childhood.

47 in had multiple effects on Day 14 lung, when alveolarization was about half complete.

48 genesis early in lung development, postnatal alveolarization was not influenced by FGFR-HFc.

49 g morphology and physiology during postnatal alveolarization were assessed in transgenic mice express

50 ung mechanics, engraftment, lung growth, and alveolarization were evaluated 8 weeks after transplanta

51 compliance, non-parenchymal lung volume and alveolarization, were increased in both AAD and Control

52 when expressed from E16.5 but did not alter alveolarization when expressed after birth.