ライフサイエンスコーパス: alveolitis

1 challenge, with no ERD signs in the form of alveolitis.

2 cytokines is associated with the presence of alveolitis.

3 inflammation in IgG immune complex-mediated alveolitis.

4 lity in models of chemotactic factor-induced alveolitis.

5 , and TNF-alpha and would have a concomitant alveolitis.

6 ty and neutrophil infiltration in a model of alveolitis.

7 ly suppressed endotoxin-induced neutrophilic alveolitis.

8 histologically simulated extrinsic allergic alveolitis.

9 not a consequence of excessive neutrophilic alveolitis.

10 thelium and mild to moderate peribronchiolar alveolitis.

11 y correlated with the severity of the T cell alveolitis.

12 lymorphonuclear neutrophil (PMN)-predominant alveolitis.

13 e in some, but not all, patients with active alveolitis.

14 resence of lung granulomas and a CD4+ T cell alveolitis.

15 reathlessness, the primary symptom of active alveolitis.

16 ents of lung be performed for diagnosing SSc alveolitis.

17 having active alveolitis and 1 as having no alveolitis.

18 ula may underestimate the presence of active alveolitis.

19 o produce lesions resembling human fibrosing alveolitis.

20 ividuals and strongly correlated with T-cell alveolitis.

21 which is the time point of peak neutrophilic alveolitis.

22 lammatory response using a mouse model of IC alveolitis.

23 HIV disease progression than was lymphocytic alveolitis.

24 ave restrictive lung disease and evidence of alveolitis.

25 terstitial mononuclear cell infiltration and alveolitis.

26 st levels were detected in SSc patients with alveolitis.

27 erminal airspace cell population to diagnose alveolitis, a condition that predicts changes in lung fu

28 rstitial pneumonia characterized by a marked alveolitis, accompanied by loss of appetite, weight loss

29 ell numbers, and resolution of CD8(+) T-cell alveolitis after ART in 9 of 12 individuals.

30 dients predict the intensity of neutrophilic alveolitis after treatment with endotoxin.

31 In SSc patients with alveolitis, all 8 domains of the SF-36 were significantl

32 T lymphocytic alveolitis also was found in the affected but not the un

33 Lymphocytic alveolitis, although present at all stages of HIV infect

34 patients were reclassified as having active alveolitis and 1 as having no alveolitis.

35 ving 890 subjects with cryptogenic fibrosing alveolitis and 5, 884 control subjects drawn from the Un

36 In the lung, CO suppressed LPS-induced lung alveolitis and associated edema formation, while in the

37 ease (CBD) is characterized by a CD4+ T cell alveolitis and granulomatous inflammation in the lung.

38 hereas these mice had increased neutrophilic alveolitis and greater lung injury compared with WT cont

39 mma-producing CD8 T cells mediated pulmonary alveolitis and inflammation, which were dependent upon C

40 d enhanced pulmonary pathology consisting of alveolitis and interstitial pneumonitis after a live-vir

41 ere were marked differences in severities in alveolitis and interstitial pneumonitis when each of the

42 als primed with FG vaccine showed quite mild alveolitis and interstitial pneumonitis, which were elim

43 with Go6976 showed significantly suppressed alveolitis and neutrophil influx in bronchial alveolar l

44 eased mortality, consistent with more severe alveolitis and pneumonitis.

45 lung disorder characterized by CD4(+) T cell alveolitis and progressive lung fibrosis.

46 Extrinsic allergic alveolitis and pulmonary sarcoidosis are granulomatous d

47 ogically similar to human extrinsic allergic alveolitis and sarcoidosis, respectively.

48 symptoms in patients with evidence of active alveolitis and scleroderma-related interstitial lung dis

49 is higher than those in SSc patients without alveolitis and than those in normal controls), and RANTE

50 formed on patients with SSc (with or without alveolitis) and on normal control subjects.

51 alveolar damage, hyaline membrane formation, alveolitis, and death were noted in 12-month-old mice in

52 to the recruitment of alveolar macrophages, alveolitis, and subsequent granuloma development.

53 eased in patients with cryptogenic fibrosing alveolitis, and that this effect is independent of the e

54 onary edema and develops into a neutrophilic alveolitis, and, later, pulmonary fibrosis.

55 reduced histological evidence of PcP-related alveolitis as compared with infected wild-type mice.

56 y be a risk factor for cryptogenic fibrosing alveolitis as well as for lung cancer, and so may confou

57 C5a-induced alveolitis, as measured by cell counts and total protein

58 These findings suggest that alveolitis associated with HIV-1 infection is caused by

59 o the lungs with acute, focal bronchitis and alveolitis associated with massive pulmonary oedema, hae

60 fibrosing alveolitis (CFA) and in fibrosing alveolitis associated with systemic sclerosis (FASSc).

61 At low RSV F doses, alveolitis-associated histopathology was unexpectedly ob

62 th the development of a macrophage-dominated alveolitis at sites of infection, with increased synthes

63 This evidence suggests that fibrosing alveolitis begins much earlier in the course of clinical

64 itical pathway to end-stage fibrosis is not "alveolitis" but rather the ongoing epithelial damage and

65 t CRP has a significant protective effect in alveolitis by reducing neutrophil influx and protein lea

66 stitial vascularity in cryptogenic fibrosing alveolitis (CFA) and in fibrosing alveolitis associated

67 Lone cryptogenic fibrosing alveolitis (CFA) is a progressive interstitial lung dise

68 viously suggested that cryptogenic fibrosing alveolitis (CFA) may be caused by occupational exposures

69 use model of intratracheal endotoxin-induced alveolitis, coexposure to FRT (core temperature approxim

70 dence in patients with cryptogenic fibrosing alveolitis compared with the general population in a pop

71 Interventions to limit neutrophilic alveolitis could either be targeted to block local lung

72 imited (n = 57) cutaneous disease and active alveolitis (determined by bronchoalveolar lavage and/or

73 Thus, fibrosing alveolitis develops after intratracheal bleomycin irresp

74 s, neither crescentic glomerulonephritis nor alveolitis ensued, likely because of the predominance of

75 Fibrosing alveolitis (FA) is characterized by persistent inflammat

76 long-term followup, patients with persistent alveolitis had a decline in lung function (mean +/- SD c

77 Although lymphocytic alveolitis has been associated with viremia and an incre

78 Cryptogenic fibrosing alveolitis has been reported to be associated with an in

79 ected mouse lung tissues demonstrated severe alveolitis, hemorrhaging, and spread of the virus throug

80 P = 0.0005, with levels in SSc patients with alveolitis higher than those in normal controls), IL-8 (

81 = 0.03, with levels in SSc patients without alveolitis higher than those in normal controls).

82 (P = 0.009, with levels in SSc patients with alveolitis higher than those in SSc patients without alv

83 Open lung biopsy revealed extrinsic allergic alveolitis (hypersensitivity pneumonitis).

84 D8(+) T cells, often resolving CD8(+) T-cell alveolitis in active smokers.

85 Influenza is known to cause persistent alveolitis in animal models; however, little is known ab

86 Lymphocytic alveolitis in HIV-1-infected individuals is associated w

87 These data suggest that lymphocytic alveolitis in HIV-infected subjects occurs in response t

88 The usefulness of BAL to define alveolitis in SSc is questionable.

89 s opacification on HRCT accurately predicted alveolitis in the middle lung fields, HRCT did not detec

90 hoalveolar lavage (BAL), in the diagnosis of alveolitis in these patients.

91 necessary for the full development of acute alveolitis in this model of lung injury.

92 protein C and proCPB, attenuated C5a-induced alveolitis in WT but not in proCPB-/- mice, indicating t

93 C5a instillation was ineffective in reducing alveolitis in WT mice, suggesting that the beneficial ef

94 To study the effect of CRP on alveolitis induced by different chemoattractants, transg

95 Extrinsic allergic alveolitis is caused by inhaled antigens, whereas the na

96 othesized that the intensity of neutrophilic alveolitis is related to establishing a gradient of neut

97 ells in the lung, and this failure of a CD4+ alveolitis limits an effective immune response.

98 t histologic features, including lymphocytic alveolitis, lymphocytic interstitial pneumonitis, bronch

99 though early identification and treatment of alveolitis may prevent deterioration of lung function, t

100 Lung inflammation (alveolitis) may cause lung fibrosis in scleroderma.

101 the results derived from our immune complex alveolitis model.

102 sma protein affects leukocyte recruitment in alveolitis models of lung inflammation.

103 ype DBP(+/+) counterparts in three different alveolitis models, two acute and one chronic.

104 round-glass opacification and the finding of alveolitis on BAL from segments in the same lung regions

105 between fibrosis on HRCT and the presence of alveolitis on BAL was significant for the lower lobes bu

106 Despite viral clearance, bronchiolitis and alveolitis persisted at day 14 postinfection; histopatho

107 Lymphocytic alveolitis portends a poor prognosis in human immunodefi

108 d cell count obtained after CYC treatment of alveolitis predicts long-term lung function outcomes and

109 ed among patients with cryptogenic fibrosing alveolitis (rate ratio [RR] 7.31, 95% confidence interva

110 Nine of the 18 patients had active alveolitis recorded in both lavaged segments, while in 4

111 e poor prognosis associated with lymphocytic alveolitis reflects a high pulmonary viral burden.

112 ion, the best approach for diagnosing active alveolitis remains controversial.

113 une complex glomerulonephritis and pulmonary alveolitis, similar to that caused by cells treated with

114 ell depletion and dysfunction, CD8(+) T-cell alveolitis, smoking, and poor control of human immunodef

115 almost completely abolished the neutrophilic alveolitis that occurs in rats following i.p. injection

116 cause of infection (six deaths), hemorrhagic alveolitis (three deaths), or bleeding (one death).

117 ical changes consisting of bronchiolitis and alveolitis typical of virus-induced changes.

118 Active alveolitis was defined as the presence of > or =3.0% pol

119 An immature macrophage alveolitis was found in the affected lung and the unaffe

120 To test this hypothesis, C5a-induced alveolitis was studied in wild-type (WT) and proCPB-defi

121 gelatinase B in bleomycin-induced fibrosing alveolitis, we instilled bleomycin intratracheally into

122 bronchiolocentric granulomatous lymphocytic alveolitis, which evolves to fibrosis in chronic advance

123 nchoalveolar lavage or biopsy, patients with alveolitis who did not receive cyclophosphamide therapy

124 s and survival are improved in patients with alveolitis who receive cyclophosphamide.

125 median follow-up of 16 months, patients with alveolitis who received cyclophosphamide were more likel