ライフサイエンスコーパス: amacrine

1 The majority of amacrine (ACs) and ganglion cells (GCs) showed kainate r

2 The neurites of the retinal ganglion, amacrine and bipolar cell subtypes that form synapses in

3 he Ascl1-expressing Muller glia give rise to amacrine and bipolar cells and photoreceptors.

4 s of horizontal cells and reduced numbers of amacrine and bipolar cells, while the number of Muller g

5 nti-VEGF-treated WT mice also presented mild amacrine and ganglion cell death, but no overt abnormali

6 sensor (iGluSnFR) expressed on postsynaptic amacrine and ganglion cell dendrites.

7 ed this approach to characterize the retinal amacrine and ganglion cell types in five GABAergic Cre m

8 eurons that form microcircuits with bipolar, amacrine and ganglion cells to process visual informatio

9 y-stage retinal progenitors, differentiating amacrine and ganglion cells, and late-stage progenitors

10 essed strongly in BP cells and a fraction of amacrine and ganglion cells.

11 ceptor beta gene (Rorb) is critical for both amacrine and horizontal cell differentiation in mice.

12 tina confirmed that Bclaf1 serves to promote amacrine and horizontal cell differentiation.

13 nducible and proangiogenic factors, and that amacrine and horizontal cell dysfunction induces alterat

14 xn4), two early-acting factors necessary for amacrine and horizontal cell generation.

15 ealed RORbeta1 as a novel marker of immature amacrine and horizontal cells and of undifferentiated, d

16 d that a subset of retinal interneurons, the amacrine and horizontal cells, form neurovascular units

17 ficient to directly induce the generation of amacrine and horizontal cells.

18 Amacrine and horizontal interneurons integrate visual in

19 pressing RPCs, which are destined to produce amacrine and photoreceptor cells, can be re-programmed i

20 sruptions of synaptic structures and loss of amacrine and retinal ganglion cells in anti-VEGF treated

21 the inner nuclear layer as well as widefield amacrine and small bistratified ganglion cells in the ga

22 us studies have suggested the involvement of amacrine and/or horizontal cells.

23 to multiple cell types, including ganglion, amacrine, and bipolar cells and photoreceptors, but not

24 to early-born cell types, such as ganglion, amacrine, and horizontal cells.

25 o spatially map individual GCs to underlying amacrine, bipolar, horizontal, photoreceptor, and retina

26 ence of impaired vesicular GABA release from amacrine but not horizontal interneurons.

27 ipolar cell (BC) classes inhibit rod BCs via amacrine cell (AC) motifs (C1-6); that all cone BC class

28 Although no change was detected in total amacrine cell (AC) numbers, increased PRKCA(+) and choli

29 The AII amacrine cell (AII) is a key information hub in the reti

30 ized type of amacrine cell, the dopaminergic amacrine cell (DAC).

31 cine and glutamate, by the vGluT3-expressing amacrine cell (GAC) in the mouse retina.

32 rientation-selective, wide-field, polyaxonal amacrine cell (PAC) in the rabbit retina and demonstrate

33 individual dendritic sectors of a starburst amacrine cell (SAC) are preferentially activated by diff

34 The AII amacrine cell also makes direct glycinergic synapses wit

35 cted ON-OFF segregation within a small-field amacrine cell arose from local synaptic processing, medi

36 of other ON parasol cells recorded near the amacrine cell axonal projections.

37 p to further our understanding of how single amacrine cell circuits act together to help decompose th

38 Studies investigating individual amacrine cell circuits like the starburst or A17 circuit

39 of postsynaptic plasticity in a subgroup of amacrine cell dendrites.

40 neurotransmitter release sites on starburst amacrine cell dendrites: the excitatory input distributi

41 The amacrine cell density ranged from 30,000 cells/mm(2) at

42 cadherin Fat3 acts during multiple stages of amacrine cell development in mice to orient overall chan

43 beta1 expression in neonatal retina promotes amacrine cell differentiation.

44 Light-evoked depolarizations of the AII-amacrine cell elicited exocytosis that was graded to lig

45 Finally, Sox2, Sox8, and Sox9 promoted amacrine cell formation in Lhx2-deficient cells, but not

46 rom rare counterexamples (e.g. the starburst amacrine cell in retina), neurons are polarized into two

47 The starburst amacrine cell in the mouse retina presents an opportunit

48 e from the presynaptic interneuron starburst amacrine cell in the mouse retina.

49 our understanding of how general features of amacrine cell inhibition lead to general features of com

50 vity of a retinal interneuron called the AII amacrine cell is responsible for anti-correlated spiking

51 ogical blockade of GJs eradicated nearly all amacrine cell loss and reduced retinal ganglion cell los

52 e cells immunolabeled for an RGC marker, not amacrine cell markers, suggesting that they are dopamine

53 the periphery, via the electrically coupled amacrine cell network.

54 examined the genetic control of cholinergic amacrine cell number and distribution between these two

55 We focused on amacrine cell number control, identifying three downstre

56 yses placed Pten downstream of TgfbetaRII in amacrine cell number control.

57 we show that Pten is a critical regulator of amacrine cell number in the retina, acting via multiple

58 The total cholinergic amacrine cell number was found to vary across the strain

59 s sufficient to recapitulate the Fat3 mutant amacrine cell phenotype.

60 We identify a type of polyaxonal amacrine cell physiologically via its distinctive electr

61 ic, suggesting that they constitute a single amacrine cell population.

62 ual signaling by a distinctive, high-density amacrine cell population.

63 istry and major cell types of a VIP-ires-Cre amacrine cell population.

64 etinal explants phenocopies the reduction in amacrine cell production observed in Pten cKOs.

65 Pten is thus a positive regulator of amacrine cell production, acting via multiple downstream

66 ts, which is a negative feedback pathway for amacrine cell production.

67 levels to promote the differentiation of all amacrine cell subtypes, which are each reduced in number

68 urotransmitter release at bipolar neuron/AII amacrine cell synapses and rendered spontaneous miniatur

69 Our results suggest that AII-amacrine cell synapses are capable of providing both pha

70 response to contrast, whereas depression in amacrine cell synapses causes sensitization.

71 Bipolar but not amacrine cell synapses have presynaptic ribbon-like stru

72 aptic transmission at rod bipolar neuron-AII amacrine cell synapses in acute mouse retina slices as a

73 l synapses onto specific targets and receive amacrine cell synapses in the nominal OFF layer, creatin

74 tput was anticorrelated with the activity of amacrine cell synapses providing inhibitory feedback.

75 isolates a specific pathway through the AII amacrine cell that does not require iGluRs: cone-->ON co

76 that enables a small-field, dual-transmitter amacrine cell to process diverse dendritic functions in

77 el cholinergic, non-GABAergic, non-starburst amacrine cell type described for the first time in teleo

78 se and test a model for the function of this amacrine cell type, in which the extra-classical recepti

79 ar layer, indicating they represent a single amacrine cell type.

80 We also labeled several wide-field amacrine cell types that have been previously identified

81 The VIP-ires-Cre amacrine cell types were identified in VIP-Brainbow2.1 r

82 s some other subsets of retinal ganglion and amacrine cell types, along with horizontal cells, while

83 function of most of the approximately 30-40 amacrine cell types, each of which synapses onto a subse

84 es and intercellular connections of specific amacrine cell types.

85 (Mus musculus) lines, and identified two new amacrine cell types: an asymmetric medium-field type and

86 The AII amacrine cell uses sign-conserving electrical synapses t

87 s for one type of rod pathway interneuron (A amacrine cell) in the retina of some but not all mammali

88 he exception of the rod pathway-specific AII amacrine cell, the connectivity of glycinergic small-fie

89 esized and released by a specialized type of amacrine cell, the dopaminergic amacrine cell (DAC).

90 re iGluRs: cone-->ON cone bipolar cell-->AII amacrine cell-->RGC.

91 ating direction selectivity in the starburst amacrine cell.

92 ipolar cells as the rod pathway-specific AII amacrine cell.

93 d connectivity pattern of the small-field A8 amacrine cell.

94 ized circuit: rods-->rod bipolar cells-->AII amacrine cell.

95 While all five types exhibited inhibitory amacrine-cell and excitatory bipolar-cell inputs from th

96 Retinal amacrine cells (ACs) may make inhibitory chemical synaps

97 glutamate transporter 3 (VGluT3)-expressing amacrine cells (ACs) to a broad set of visual stimuli.

98 We genetically identify a class of amacrine cells (ACs) with elongated dendritic arbors tha

99 GCs), followed by horizontal cells (HCs) and amacrine cells (ACs), beginning with the early stages of

100 RBC output onto putative AII amacrine cells (ACs), the crucial interneurons of the ro

101 r the structural and functional integrity of amacrine cells (ACs), the largest cohort of neurons in t

102 from bipolar cells and inhibitory input from amacrine cells (ACs).

103 onses of individual bipolar cells (BCs), AII amacrine cells (AIIACs), and ON and sustained OFF alpha-

104 ced glial activation and loss of function of amacrine cells (brain nitric oxide synthetase/tyrosine h

105 Dopaminergic amacrine cells (DACs) release dopamine in response to li

106 ar layer amacrine cells (iACs) and displaced amacrine cells (dACs)--reach their specific laminar posi

107 e source of retinal dopamine is dopaminergic amacrine cells (DACs).

108 make connections with upstream dopaminergic amacrine cells (DACs): (1) ipRGC signaling to DACs is bl

109 e found that vGluT3-expressing glutamatergic amacrine cells (GACs) generate ON-OFF somatic responses

110 ent glutamate release from vGluT3-expressing amacrine cells (GACs) in the mouse retina.

111 amacrine cells (gammaACs), glycine-positive amacrine cells (GACs), and ganglion cells.

112 include gamma-aminobutyrate (GABA)-positive amacrine cells (gammaACs), glycine-positive amacrine cel

113 -horizontal cells (HCs), inner nuclear layer amacrine cells (iACs) and displaced amacrine cells (dACs

114 Polyaxonal amacrine cells (PACs) are a unique subset identified by

115 lues, here we reconstruct Off-type starburst amacrine cells (SACs) and bipolar cells (BCs) in serial

116 during null-direction motion from starburst amacrine cells (SACs) and DS acetylcholine and glutamate

117 etina to show connectivity between starburst amacrine cells (SACs) and their known synaptic partners,

118 Starburst amacrine cells (SACs) are thought to mediate this direct

119 The dendrites of starburst amacrine cells (SACs) in the mammalian retina are prefer

120 inhibition arising from GABAergic starburst amacrine cells (SACs) strongly contributes to direction

121 inhibitory neurotransmission from starburst amacrine cells (SACs) to direction selective ganglion ce

122 acetylcholine (ACh) and GABA from starburst amacrine cells (SACs) to direction-selective ganglion ce

123 nglion cells (DSGCs) and GABAergic starburst amacrine cells (SACs), and the SACs then provide FF inhi

124 In this study, we used retinal starburst amacrine cells (SACs), critical components of a directio

125 rs (beta2-nAChRs) selectively from starburst amacrine cells (SACs), we show that mutual excitation am

126 iated by the spontaneous firing of Starburst Amacrine Cells (SACs), whose dense, recurrent connectivi

127 hibition, which is provided by OFF starburst amacrine cells (SACs).

128 Directional GABA release from starburst amacrine cells (SBACs) is critical for generating direct

129 release from the dendritic tips of starburst amacrine cells (SBACs).

130 sitive and shows the morphology of widefield amacrine cells (stellate, semilunar, and thorny amacrine

131 study visual processing in VGluT3-expressing amacrine cells (VG3-ACs) in the mouse retina.

132 s to the recruitment of GABAergic wide-field amacrine cells (WACs) endowing the DS circuit with an ad

133 typical network formed by different types of amacrine cells across the inner plexiform layer prompts

134 ce that general functions of the ensemble of amacrine cells across types are critical for establishin

135 of glutamate (or AMPA) onto the dendrites of amacrine cells also significantly potentiated evoked cur

136 sity of parvalbumin- and calretinin-positive amacrine cells and a loss of ganglion cells was detected

137 diated light responses from ganglion and AII amacrine cells and by recording RB-mediated synaptic cur

138 ied input to the melanopsin-ir RGCs from AII amacrine cells and directly from rod bipolar cells via r

139 ity in the shape and function of the studied amacrine cells and elucidate their connections with spec

140 plexiform layer (IPL) and from dopaminergic amacrine cells and GABAergic processes in the outermost

141 identified in photoreceptors, bipolar cells, amacrine cells and ganglion cells, but have not been con

142 ns, including photoreceptors, bipolar cells, amacrine cells and ganglion cells, but they have not bee

143 on in the numbers of retinal ganglion cells, amacrine cells and horizontal cells and an increase in t

144 DA amacrine cells and M1 ipRGCs express the SRIF receptor s

145 circuit composed of dopamine (DA)-containing amacrine cells and melanopsin-containing, intrinsically

146 Retinae derived from f-iPSCs had fewer amacrine cells and other inner nuclear layer cells.

147 c "retinal waves" are initiated in starburst amacrine cells and propagate to retinal ganglion cells a

148 and bipolar cells were next most central and amacrine cells and retinal ganglion cells were on the ou

149 ndene-1,4'-piperidine]-1'-carboxamide) in DA amacrine cells and the selective sst4 agonist L-803,087

150 l-field amacrine cells closely resembling A8 amacrine cells and their cone-dominated bipolar cell inp

151 eir stratification pattern with narrow-field amacrine cells and their neurotransmitter with wide-fiel

152 Starburst amacrine cells and their synaptic partners, ON-OFF direc

153 Dopamine is released by retinal dopaminergic amacrine cells and transmits signaling either by convent

154 that melanopsin cells were tracer coupled to amacrine cells and would be applicable to electrophysiol

155 Amacrine cells are a heterogeneous group of interneurons

156 Segretagogin amacrine cells are also immunopositive to antibodies aga

157 VIP-1 amacrine cells are bistratified, wide-field cells that r

158 AII-amacrine cells are interneurons in the retina that exhib

159 apse imaging assay, we found that developing amacrine cells are less directed towards the IPL in the

160 ramify in strata 1, 4, and 5, VIP-2A and 2B amacrine cells are medium-field cells that mainly ramify

161 amify in strata 3 and 4, and VIP-3 displaced amacrine cells are medium-field cells that ramify in str

162 Amacrine cells are the most diverse and least understood

163 Amacrine cells are thought to be a major locus for mecha

164 eceiving direct photoreceptor input, whereas amacrine cells are usually monopolar inhibitory interneu

165 Most of these amacrine cells are wide field, although some are medium

166 A labels astrocytes on the day of birth, AII amacrine cells at postnatal (P) day 5, and Muller glia b

167 ased the generation of mislocalized ganglion/amacrine cells at the expense of rod and cone photorecep

168 However, AII amacrine cells avoid making synapses with numerous RGC t

169 tina contains at least 30 different types of amacrine cells but not many are well characterized.

170 ibe a population of bistratified small-field amacrine cells closely resembling A8 amacrine cells and

171 Amacrine cells comprise approximately 30 morphological t

172 The spatial density of secretagogin amacrine cells decreases from a peak of about 400 cells/

173 ber of RGCs, whereas the number of displaced amacrine cells decreases.

174 of the inner plexiform layer, similar to A8 amacrine cells described in cat and human retina.

175 Developing AII amacrine cells exhibit accumulating DNER labeling at the

176 VIP-ires-Cre amacrine cells form a neuropeptide-expressing cell popul

177 x of Ca(2+) in dendritic varicosities of A17 amacrine cells from diabetic compared with normal animal

178 The labeled amacrine cells had a bistratified appearance with a dens

179 ntributed to the reversal response, but that amacrine cells had, at best, a minor role.

180 the connectivity of glycinergic small-field amacrine cells has not been investigated in the mouse re

181 mma-aminobutyric acid (GABA)ergic wide-field amacrine cells have recently been studied; however, with

182 ctrical coupling between RGCs and polyaxonal amacrine cells in mouse retina forms the synaptic mechan

183 ciated virus-mediated technique to label AII amacrine cells in mouse retina, we observed diminished d

184 rding RB-mediated synaptic currents from AII amacrine cells in mouse retina.

185 he full complement of rod photoreceptors and amacrine cells in mouse.

186 rved diminished dopaminergic contacts to AII amacrine cells in RD mice.

187 d with targeted patch-clamp recordings of DA amacrine cells in TH-RFP mice and M1 ipRGCs in OPN4-EGFP

188 ear total RGC loss with a marked increase in amacrine cells in the ganglion cell layer.

189 ina; these densities account for about 1% of amacrine cells in the inner nuclear layer and for up to

190 against calretinin can be used to identify A amacrine cells in the inner nuclear layer as well as wid

191 ns, receive synaptic inputs from bipolar and amacrine cells in the inner plexiform layer (IPL) and se

192 cal connectivity between rod bipolar and A17 amacrine cells in the mammalian retina, specifically tha

193 fia), that are each heavily expressed in AII amacrine cells in the mature mouse retina, and which con

194 zed in a population of regular and displaced amacrine cells in the retina of the common marmoset Call

195 edianus magnocellularis, the spinal cord and amacrine cells in the retina.

196 ateral inhibition onto Off SACs from non-SAC amacrine cells is required for optimal direction selecti

197 The amacrine cells labeled in Tg(mglur6b:EGFP)zh1 constitute

198 All Cre-expressing amacrine cells labeled with an antibody to GABA.

199 e from its presynaptic arrays of bipolar and amacrine cells less efficiently than the OFF cell does.

200 These recordings show that AII amacrine cells make direct synapses with OFF Alpha, OFF

201 Because Ca(2+)-permeable receptors in A17 amacrine cells mediate synaptic release of GABA, the red

202 For instance, retinal amacrine cells migrate towards the inner plexiform layer

203 the inner plexiform layer, where inhibitory amacrine cells modulate the excitatory signal of bipolar

204 terneuron in the mouse retina that resembles amacrine cells morphologically but is glutamatergic and,

205 soma location, the dendrites of secretagogin amacrine cells occupy strata 2, 3, and 4 of the inner pl

206 esynaptic inhibition is generated by spiking amacrine cells on a larger spatial scale covering severa

207 NER, by contrast, is present in ganglion and amacrine cells on P1, also labeling the horizontal cells

208 se of gamma-aminobutyric acid from starburst amacrine cells onto direction-selective ganglion cells (

209 2 from the population of nascent cholinergic amacrine cells perturbed the normal ratio of cells situa

210 KEY POINTS: Bipolar and amacrine cells presynaptic to the ON sustained alpha cel

211 However, unlike AII cells, A8 amacrine cells provide glycinergic input to ON A-type ga

212 Our data suggest that A8 amacrine cells receive glutamatergic input from both OFF

213 Our data show that Syt2-labeled amacrine cells receive input from both OFF and ON cone b

214 ic to gamma-aminobutyric acid (GABA)ergic to amacrine cells remained relatively constant.

215 Wide-field amacrine cells span large segments of the retina, making

216 sion largely restricted to a small subset of amacrine cells that express disabled-1 (Dab1) but lack e

217 in cells in marmoset retina are medium-field amacrine cells that share their stratification pattern w

218 Coupling causes the amacrine cells to fire spikes that propagate radially ov

219 This allows the actions of SRIF on DA amacrine cells to proceed with adjusting retinal DA leve

220 The total number of cholinergic amacrine cells was quantified in the C57BL/6J and A/J in

221 bipolar cells and conventional synapses from amacrine cells were identified in electron microscopic i

222 ion of M1 ipRGCs and DA amacrine cells, SRIF amacrine cells would provide inhibitory modulation to bo

223 crine cells (stellate, semilunar, and thorny amacrine cells).

224 r of the retina derives from the activity of amacrine cells, a large and diverse group of GABAergic a

225 ons exhibit aberrant activity, driven by AII amacrine cells, a primary target of the retinal dopamine

226 inal precursors into retinal ganglion cells, amacrine cells, and horizontal cells rather than into co

227 SRIF amacrine cells, DA amacrine cells, and M1 ipRGCs form numerous contacts.

228 ds, bipolar cells, amacrine cells, displaced amacrine cells, and Muller glia were generated between F

229 d by recurrent connectivity within starburst amacrine cells, and retinal ganglion cells act as "reado

230 teraction is reciprocal: M1 ipRGCs excite DA amacrine cells, and these, in turn, feed inhibition back

231 es dendrite targeting in type 2 dopaminergic amacrine cells, by restricting the stratum in which expl

232 SRIF amacrine cells, DA amacrine cells, and M1 ipRGCs form nu

233 Rods, bipolar cells, amacrine cells, displaced amacrine cells, and Muller gli

234 RGCs, derived from electrical coupling with amacrine cells, encodes information critical to global o

235 ic for bipolar cells, and therefore resemble amacrine cells, excite inner retinal circuits using glut

236 sponses in DACs, which are mediated by other amacrine cells, likely driven by type 1 and type 2/3a OF

237 tina contains two populations of cholinergic amacrine cells, one positioned in the ganglion cell laye

238 points, such as the neurites of cholinergic amacrine cells, or to define a number of bins into which

239 idirectional interaction of M1 ipRGCs and DA amacrine cells, SRIF amacrine cells would provide inhibi

240 another subpopulation of upstream GABAergic amacrine cells, thereby sustaining the GABAC receptor ac

241 ecordings between bipolar cell terminals and amacrine cells, we have simultaneously measured presynap

242 ractions in the inner retina are mediated by amacrine cells, which are thought to be inhibitory neuro

243 ss in the Ndufs4 KO is the loss of starburst amacrine cells, which may be an important target in the

244 their dyad postsynaptic targets, AII and A17 amacrine cells, which play an essential role in processi

245 is provided by a subpopulation of wide-field amacrine cells, which stimulate the GABAC receptors at r

246 equency signals was regulated by glycinergic amacrine cells, while GABAergic inhibition regulated the

247 was localized in a population of small-field amacrine cells, whose cell bodies formed a regular mosai

248 originating from a population of wide-field amacrine cells.

249 d inputs from axon terminals of dopaminergic amacrine cells.

250 tentiated evoked EPSCs in a subpopulation of amacrine cells.

251 bition from the ON pathway through GABAergic amacrine cells.

252 cells, but also in a subset of ganglion and amacrine cells.

253 is propagated to an atypical subtype of AII amacrine cells.

254 g horizontal cells and a subset (25%) of AII amacrine cells.

255 ter 1, and shows the typical morphology of A amacrine cells.

256 for each gene in the differentiation of AII amacrine cells.

257 on between retinal rod bipolar cells and AII amacrine cells.

258 of M1 ipRGCs caused by SRIF inhibition of DA amacrine cells.

259 erminals, ganglion cell dendrites, and other amacrine cells.

260 recursive bistratified ganglion cells and A1 amacrine cells.

261 s the synaptic receptors on A17, but not AII amacrine cells.

262 d GABA-immunoreactivity indicating they were amacrine cells.

263 with inhibitory interneurons: horizontal and amacrine cells.

264 mber and increase in the number of displaced amacrine cells.

265 s and their neurotransmitter with wide-field amacrine cells.

266 between the two bands formed by cholinergic amacrine cells.

267 bust and selective expression in cholinergic amacrine cells.

268 r cells activates postsynaptic AMPARs on AII amacrine cells.

269 idance in many types of retinal ganglion and amacrine cells.

270 n (by 30%) of RGCs, and absence of starburst amacrine cells.

271 n of W3 ganglion cells, but not to starburst amacrine cells.

272 th their identity as RGCs, and not displaced amacrine cells.

273 nuclear layer and for up to 27% of displaced amacrine cells.

274 OFF CBCs via diffusely stratified inhibitory amacrine cells.

275 ming new architectures for generating ON-OFF amacrine cells.

276 ticipating in the development of retinal AII amacrine cells.

277 orny ganglion cells, 14 cells were displaced amacrine cells.

278 ic/cholinergic signals mediated by starburst amacrine cells.

279 er are ganglion cells, and 20% are displaced amacrine cells.

280 ion mediated by wide-field spiking GABAergic amacrine cells.

281 DACs, impairing functional regulation of AII amacrine cells.

282 cal varieties of such ipRGC-driven displaced amacrine cells: (1) monostratified cells with dendrites

283 t from rod bipolar cells to both AII and A17 amacrines, diabetes changes the synaptic receptors on A1

284 AII amacrines forward their signals to ON- and OFF-cone bipo

285 Here, we investigated functional AII amacrine-->RGC synaptic connections in the retina of the

286 ever, showed the opposite effect of removing amacrine input, which was a decrease in SNR.

287 glion cell layer of rat retinas, all spiking amacrine interneurons with sustained ON photoresponses r

288 mation about many of the retina's inhibitory amacrine interneurons.

289 k required for generating the horizontal and amacrine lineages.

290 When the amacrine-modulated conductance was removed, the ON cell'

291 The fractions of rod bipolar, cone bipolar, amacrine, Muller, and horizontal cells of all cells in t

292 ng R7 photoreceptors and specific wide-field amacrine neurons called Dm8.

293 In contrast, the Dm8 amacrine neurons form a wide dendritic field to receive

294 Finally, we show that the Dm8 amacrine neurons, a synaptic target of R7 photoreceptors

295 expression of Pk1 is detected in cholinergic amacrine neurons.

296 the brain, but the interneuron (bipolar and amacrine) populations providing input to ganglion cells

297 ls to ON- and OFF-cone bipolar cells and A17 amacrines provide GABAergic feedback inhibition to rod b

298 d for RB and type 2 OFF-CB cell survival and amacrine subtype identity, and they present PRDM8 as a c

299 c ACs were seen, suggesting an alteration in amacrine subtype identity.

300 formed between rod bipolar cells (RBCs) and amacrine type-2 (AII) cells in the mouse retina but dram