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1 immune system to interact with the invading ameba.
2 amebic infection were spiked with Balamuthia amebas.
3 a food source) contained, in addition to the ameba, a variety of soil organisms, including other ameb
4 use of monkey kidney cells for growth of the amebas and can be helpful in isolation of these amebas f
5 In subsequent transfers, axenic Naegleria amebas and, later, tissue cultures (monkey kidney cells)
7 report describes the first isolation of the ameba Balamuthia mandrillaris from an environmental soil
9 than five times greater than that of control amebas, but surface staining of several antigens, includ
10 idine isethionate was most effective against amebas (ca. 90% inhibition after 6 days of exposure), bu
11 , we demonstrate that the LRRK2 homolog from ameba can be mutated to approximate some aspects of the
12 a variety of soil organisms, including other amebas, ciliates, fungi, and nematodes, as contaminants.
14 re also used for communication in the social ameba Dictyostelium discoideum when the solitary cells a
15 modifies Skp1 in the cytoplasm of the social ameba Dictyostelium, we have identified an enzyme, polyp
19 eristic of tissue invasion by the intestinal ameba Entamoeba histolytica, which causes amebic dysente
20 complex is concentrated in the cortex of the ameba, especially in linear structures, possibly actin f
21 fowleri is a climate-sensitive, thermophilic ameba found in the environment, including warm, freshwat
23 bas and can be helpful in isolation of these amebas from brain tissue from cases in which amebic meni
24 We searched for substrates encoded in the ameba genome and found EhROM1 was able to cleave a cell
26 of mitochondrial 16S rRNA gene DNA from the ameba in clinical specimens such as brain tissue and cer
27 c symptoms and the difficulty in recognizing amebas in biopsied tissues, most cases are not diagnosed
33 erythrocytes, which we utilized to separate ameba-induced exposure of erythrocyte PS from the proces
35 nted filamentous bacteria (SFB) protect from ameba infection, and protection is transferable with bon
39 assay used by the Free-Living and Intestinal Amebas Laboratory of the CDC was considered the referenc
41 ncephalitis (PAM), caused by the free-living ameba Naegleria fowleri, has historically been associate
45 ns multiple mitochondrial targets; thus, 0.2 ameba represents multiple targets for amplification and
47 Balamuthia mandrillaris is a free-living ameba that causes granulomatous amebic encephalitis in b
49 eba histolytica, an extracellular, parasitic ameba that is second only to malaria in medical burden g
53 itted Balamuthia mandrillaris, a free-living ameba, were detected by recognition of severe unexpected
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