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1 B (PTP1B) in Jurkat cells after contact with amebae.
2 ia, RBC, and mucin-coated beads by wild-type amebae.
3 s the inhibition of cytotoxic proteinases of amebae.
4 histolytica trophozoites or from solubilized amebae.
5 npathogenic, complement-sensitive N. gruberi amebae.
7 nisms of phagocytosis of bacteria and RBC by amebae and macrophages, support the idea of coincidental
8 CD59 is present on the surface of N. fowleri amebae and may play a role in resistance to lysis by cyt
10 serine involvement in erythrophagocytosis by amebae and suggest the existence of a PS receptor on the
11 , a protective antigen derived from virulent amebae, and a bacterially derived maltose-binding protei
15 e diverse mechanisms for pathogen expulsion, amebae have also developed complex strategies to modulat
19 gested that lysis of neutrophils by virulent amebae may exacerbate the damage seen in amebic liver ab
22 amoeba is a genus of ubiquitous, free-living amebae that can be difficult to isolate by standard micr
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