ライフサイエンスコーパス: ameboid

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1 of the microglia in allografts displayed an ameboid (activated) configuration, with retracted proces

2 ed on increased staining with OX42), with an ameboid appearance.

3 In this article a mathematical model for ameboid cell movement is developed using a spring-dashpo

4 cells with Trap-14 induced rapid rounding of ameboid cells adherent to fibronectin-coated slides, whe

5 Round and ameboid cells became predominant in the ischemic core le

6 Round and ameboid cells were localized to the inner boundary of th

7 Round and ameboid cells were present throughout the entire ischemi

8 ced significant rounding and inactivation of ameboid cells within 20 min except for arterial endothel

9 In chemotaxing ameboid cells, a complex leading-edge signaling circuit

10 In this time, the leukocyte moves in ameboid fashion across the endothelial borders, which re

11 udies suggest that leukocytes locomote in an ameboid fashion independently of pericellular proteolysi

12 apedesis, in which the leukocyte moves in an ameboid fashion through tightly apposed endothelial bord

13 transglutaminase nor LAV1-2 is found in the ameboid form of the organism.

14 elination is evident, many GFP(+) cells with ameboid form were detected in the white matter of the sp

15 rounding of spontaneously activated (mobile) ameboid human monocytes, granulocytes, or arterial endot

16 However, ameboid-like microglia were evident in an adjacent area

17 Rod, round and ameboid-like microglia were present in the ischemic lesi

18 by inference myosin II, are required for the ameboid locomotion of these cells.

19 lace of an actin cytoskeleton to drive their ameboid locomotion.

20 in, iron was detected in reactive microglia, ameboid microglia and macrophages in MS brains.

21 Neonatal males had twice as many ameboid microglia as females and a more activated morpho

22 CD200R(+) /Iba1(+) ameboid microglia in the cingulum at P1-P5 were the only

23 was a significant increase in the number of ameboid microglia in the D7 group.

24 Only ameboid microglia present in the cultures responded to I

25 , we quantified ramified, hypertrophied, and ameboid microglia using unbiased stereological technique

26 No evidence of ameboid microglia was found.

27 cells and parenchymal microglia with a more ameboid morphology expressed HLA-DR, indicating immune a

28 r their swift transformation to an activated ameboid morphology in response to pathological insults.

29 her signals, microglia assume an activated, "ameboid" morphology and release matrix metalloproteinase

30 because of its ability to mediate actin-like ameboid motility in nematode sperm, despite a lack of se

31 through collagen-lined pores by adopting an ameboid phenotype.

32 results add to the evidence implicating soil ameboid predators as important factors for the maintenan

33 CD200R(+) cells were a population of ameboid/pseudopodic Iba1(+) microglia/macrophages observ

34 nomenon occurs during differentiation of the ameboid sperm of Caenorhabditis elegans as they activate

35 mation of spontaneously activated cells from ameboid to round.

36 rasites was typical of P. vivax, with highly ameboid trophozoites evident; however, infected erythroc

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