ライフサイエンスコーパス: ameloblastoma

コーパス検索結果 (１語後でソート)

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1 antly upregulated in KCOT when compared with ameloblastoma.

2 roduced biopsy material that proved to be an ameloblastoma.

3 the most differentially upregulated genes in ameloblastoma.

4 e diagnostic classification and treatment of ameloblastomas.

5 enic behaviors of enamel epithelium cells in ameloblastomas.

6 udy was to characterize the transcriptome of ameloblastoma and identify relevant genes and molecular

7 racterize the molecular relationship between ameloblastoma and keratocystic odontogenic tumor (KCOT)

8 samples of 15 solid/multicystic intraosseous ameloblastomas and 12 sporadic KCOTs was hybridized on A

9 Hierarchical clustering separated ameloblastomas and KCOTs into 2 distinct groups.

10 dental genes showed a similar separation of ameloblastomas and KCOTs.

11 n several tumors, including medulloblastoma, ameloblastoma, and basal cell carcinoma.

12 fused with other pathologic entities such as ameloblastomas, carcinomas, and fibromas and clinically

13 In a similar manner, ameloblastoma cells harboring an activating BRAF mutatio

14 provide clear evidence for the diagnosis of ameloblastoma in Telmatosaurus.

15 nd ISL1 were differentially overexpressed in ameloblastoma, indicating its dental identity.

16 Ameloblastoma is a benign neoplasic tumour with a strong

17 Ameloblastoma is a locally invasive benign neoplasm deri

18 rotein kinase (MAPK) pathways in over 80% of ameloblastomas, locally destructive odontogenic tumors o

19 Gene expression profiles of human ameloblastoma microdissected cells were characterized wi

20 In addition, recognition of an ameloblastoma neoplasm in a taxon lying close to the ori

21 report for the first time the presence of an ameloblastoma neoplasm in the lower jaw of a specimen re

22 SMO (encoding Smoothened, SMO) are common in ameloblastomas of the maxilla, whereas BRAF mutations ar

23 ken together, the gene expression profile of ameloblastoma reflects differentiation from dental lamin

24 Ameloblastoma separated into 2 distinct molecular cluste

25 lying inter-tumor molecular heterogeneity of ameloblastoma sub-types and have implications for the us

26 intense in both the epithelial component of ameloblastomas, the most common epithelial odontogenic t

27 d, decalcified, paraffin-embedded samples of ameloblastoma were subjected to laser capture microdisse

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