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1 menopause in women with chemotherapy-induced amenorrhea.
2 s, 16 (26%) irregular bleeding, and 16 (26%) amenorrhea.
3 lar and unpredictable bleeding, and 19 (30%) amenorrhea.
4 truation disorders; premature menopause, and amenorrhea.
5 ived chemotherapy, 35% experienced long-term amenorrhea.
6 ceive chemotherapy, 5.3% developed long-term amenorrhea.
7 cts a genetic predisposition to hypothalamic amenorrhea.
8 oup without tamoxifen had the lowest rate of amenorrhea.
9 deficiency, are associated with hypothalamic amenorrhea.
10 nRH secretion that characterize hypothalamic amenorrhea.
11 eiving adjuvant chemotherapy are at risk for amenorrhea.
12 linical hyperandrogenism, oligomenorrhea, or amenorrhea.
13 rehensive evaluation of chemotherapy-induced amenorrhea.
14 er completion of chemotherapy, she developed amenorrhea.
15 ine, and prednisone (BEACOPP) had continuous amenorrhea.
16 ormone leptin and may result in hypothalamic amenorrhea.
17 ndocrine function in women with hypothalamic amenorrhea.
18 g their role in women with treatment-related amenorrhea.
19 s reported regular menses, and 4.6% reported amenorrhea.
20 of the LH protein and cause anovulation and amenorrhea.
21 a teenage girl with proteinuria and primary amenorrhea.
23 adrenal insufficiency, two had a history of amenorrhea after birth of their children many years earl
27 were assessed concerning their influence on amenorrhea: age, treatment, stage, and the use of oral c
32 re ovarian failure (POF) is characterized by amenorrhea and high serum levels of follicle-stimulating
33 tory menstruation in women with hypothalamic amenorrhea and improves metabolic dysfunction in patient
35 c hypogonadism in 55 women with hypothalamic amenorrhea and performed in vitro studies of the identif
38 ron donor for these enzymes, in a woman with amenorrhea and three children with ABS, even though knoc
40 s of prior studies of increased frequency of amenorrhea and/or irregular menstrual cycles, particular
41 ive pregnancy test, greater than 3 months of amenorrhea, and a follicle-stimulating hormone > or = 30
44 ent rates of permanent amenorrhea, temporary amenorrhea, and oligomenorrhea among different regimens;
47 he possible benefits of chemotherapy-induced amenorrhea, and the challenges of interpreting the exist
48 leptin deficiency in women with hypothalamic amenorrhea appears to improve reproductive, thyroid, and
49 troducing foods at 4 mo on the likelihood of amenorrhea at 6 mo postpartum, but not thereafter, and t
50 with lowest lifetime weight and duration of amenorrhea, but there were no such associations with bon
52 when the patient experienced >/= 2 years of amenorrhea, commencing within 2 years of initiating chem
53 hemotherapy; (2) define chemotherapy-related amenorrhea (CRA); (3) document rates of permanent amenor
57 erall survival was improved in patients with amenorrhea for 6 months or more across all treatment gro
59 ll for more than 6 years, risk ratio = 2.7), amenorrhea (for patients who had this symptom for more t
60 the histologic features and the presence of amenorrhea, galactorrhea, and an elevated serum prolacti
61 myelopathy from syringomyelia, paraparesis, amenorrhea-galactorrhea, and other endocrine problems, h
69 important predictors of chemotherapy-induced amenorrhea in women who carry a BRCA1 or BRCA2 mutation.
70 riages: 72%; mean gestational age 39 days of amenorrhea) in the intention-to-treat analysis; 66.6% of
72 = 40, hyperandrogenism and oligomenorrhea or amenorrhea), intermediate (n = 8, hyperandrogenism), or
78 self-report of climacteric symptoms, and/or amenorrhea lasting >6 months, and/or oophorectomy, and/o
80 for 32% to 66% of adenomas and present with amenorrhea, loss of libido, galactorrhea, and infertilit
82 lly accepted indicators of pregnancy include amenorrhea, morning sickness, tender or tingling breasts
85 ea were 73%, 82%, and 6%, respectively, with amenorrhea occurring within 10 days in the majority of p
87 ion of hormone therapy prior to 12 months of amenorrhea (OR = 2.94, 95% CI: 1.14, 7.58; P = 0.03).
90 ogen receptor status, and treatment regimen, amenorrhea rates on triptorelin were comparable to those
94 onadism are found in women with hypothalamic amenorrhea, suggesting that these mutations may contribu
96 rrhea (CRA); (3) document rates of permanent amenorrhea, temporary amenorrhea, and oligomenorrhea amo
98 als with manifestations ranging from primary amenorrhea to loss of menstrual function prior to age 40
99 ed in 7 of the 55 patients with hypothalamic amenorrhea: two variants in the fibroblast growth factor
100 of spontaneous pubertal development, primary amenorrhea, uterine hypoplasia, and hypergonadotropic hy
107 The probabilities of chemotherapy-induced amenorrhea were 7.2% for women diagnosed before age 30 y
110 This review will discuss the incidence of amenorrhea with commonly-used adjuvant chemotherapeutic
112 experiencing the female triad (hypothalamic amenorrhea) with acquired chronic hypoleptinemia induced
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