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1 capillary electrophoresis (MD-CE) assays for amino acid neurotransmitters.
2 ther calcium binding proteins and inhibitory amino acid neurotransmitters.
3 y of proteins that mediates the packaging of amino acid neurotransmitters.
4 rations, decreased the release of excitatory amino acid neurotransmitters, and decreased the total fr
5 affold to which diverse moieties from lipid, amino acid, neurotransmitter, and nucleoside metabolism
6 between the apoE4 allele and the recovery of amino acid neurotransmitters (aspartate, glutamate, and
7 dic chip that allows for in vivo sampling of amino acid neurotransmitters by low-flow push-pull perfu
8 the peptides into the brain while monitoring amino acid neurotransmitters by microdialysis sampling c
9 he balance between inhibitory and excitatory amino acid neurotransmitters contributes to the control
10 mals to determine the effects of ketamine on amino acid neurotransmitter cycling and glial metabolism
11 banesthetic doses of ketamine increased mPFC amino acid neurotransmitter cycling, as well as neuronal
13 n of the immunohistochemical localization of amino acid neurotransmitters demonstrated that the riMLF
14 ed to include increased de novo synthesis of amino acid neurotransmitters, especially glutamate and G
18 he lateral hypothalamus (LH), the inhibitory amino acid neurotransmitter, GABA, has had a long-standi
19 ons have suggested a role for the inhibitory amino acid neurotransmitter gamma-aminobutyric acid (GAB
22 ty of small organic molecules, including the amino acid neurotransmitters glutamate, aspartate and ta
23 ied by morphology and immunolabeling for the amino acid neurotransmitters glutamate, aspartate, gamma
24 trastructural localization of the excitatory amino acid neurotransmitters, glutamate and aspartate, u
25 ), which mimics the action of the excitatory amino acid neurotransmitter glutamic acid, releases LH-R
26 ontrast, the distributions of the inhibitory amino acid neurotransmitters glycine and GABA were not a
28 the growing weight of evidence for a role of amino acid neurotransmitters in pancreatic islets and in
29 ts of histaminergic agents on the release of amino acid neurotransmitters in slices of the medial ves
30 rs sensory processing, we measured levels of amino acid neurotransmitters in spinal dialysates from a
34 h-level exposure to glutamate, an excitatory amino acid neurotransmitter, leads to neuronal death.
35 ule cells normally contain two "fast-acting" amino acid neurotransmitters, one excitatory and one inh
36 e addressed the topographic distribution and amino acid neurotransmitter phenotype of endocannabinoid
38 e, and allopregnanolone, modulate ionotropic amino acid neurotransmitter receptors, and may function
42 involve alterations in the major inhibitory amino acid neurotransmitter system of gamma-aminobutyric
43 lance of the major inhibitory and excitatory amino acid neurotransmitter systems of gamma-aminobutyri
44 ough the differential release of peptide and amino acid neurotransmitters to coordinate reproductive
45 Robust efflux of excitatory and inhibitory amino acid neurotransmitters was detected during ischemi
46 Both markers for inhibitory and excitatory amino acid neurotransmitters were found in varicose axon
48 mate and aspartate are endogenous excitatory amino acid neurotransmitters widely distributed in the m
49 for simultaneous monitoring of all the major amino acid neurotransmitters with 10-s temporal resoluti
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