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1 (5)), a known but rare agonist of excitatory amino acid receptors.
2 ors (mGluRs) are a major class of excitatory amino acid receptors.
3 ethyl-D-aspartate (NMDA) and NMDA excitatory amino acid receptors.
4 CB(1) (472 amino-acids) and human CB(2) (360-amino-acid) receptors.
5 tes that interact at the level of excitatory amino acid receptor activation and subsequent intracellu
6  nl) of smaller concentrations of excitatory amino acid receptor agonists (e.g., NMDA, KA and trans-A
7 of important interactions between excitatory amino acid receptors and mu-opioid receptors in the nucl
8 ivation of central nervous system excitatory amino acid receptors and subsequent intracellular cascad
9 erized in vitro for antagonism of excitatory amino acid receptors, and subsequently tested in vivo an
10  injections of the broad-spectrum excitatory amino acid receptor antagonist kynurenate (Kyn) into the
11   Tissue levels of the endogenous excitatory amino acid receptor antagonist kynurenic acid (KYNA) and
12                                   Excitatory amino acid receptor antagonists 2-amino-5-phoshopentanoi
13                 Studies utilizing excitatory amino acid receptor antagonists have been inconsistent i
14 did the presence of excitatory or inhibitory amino acid receptor antagonists in the perfusion medium.
15 potential efficacy of competitive excitatory amino acid receptor antagonists in the treatment of chro
16 HT1D receptor agonists, glutamate excitatory amino acid receptor antagonists, nitric oxide synthase i
17 rmine if a change in brain tissue excitatory amino acid receptor binding occurs during pregnancy usin
18                                   Excitatory amino acid receptor-dependent increases in descending fa
19 review are: biosynthetic pathways for N-acyl amino acids, receptors for N-acyl amino acids, physiolog
20 gest that d-amphetamine increases excitatory amino acid receptor function temporarily by reducing the
21 HS), a neurosteroid that inhibits excitatory amino acid receptor function, in a rabbit reversible spi
22 D-aspartate (NMDA) subtype of the excitatory amino acid receptor has been implicated in several kinds
23 al changes in the distribution of excitatory amino acid receptors in the chicken's auditory brainstem
24 r by prior blockade of ionotropic excitatory amino acid receptors in the commNTS with kynurenate (8 n
25  determine if the distribution of excitatory amino acid receptors in the owl's auditory brainstem and
26 utamate by inhibition of neuronal excitatory amino acid receptors, including the N-methyl-D-aspartate
27 hough pharmacologic modulation of excitatory amino acid receptors is well studied, minimal considerat
28 cribe cloning of a cDNA encoding a novel 358-amino acid receptor (LTB(4)-R2) that possesses seven mem
29 ich act as negative modulators of excitatory amino acid receptors, may improve behavioral functions a
30      The ontogeny of metabotropic excitatory amino acid receptors (mGluRs) in rat barrel field cortex
31 ble to those reported for other full-fledged amino acid receptor proteins.
32                                Both types of amino acid receptors showed electrophysiological and pha
33  of prolonged ethanol exposure on excitatory amino acid receptor stimulated nitric oxide (NO) formati
34 DNAs, gbr-2A and gbr-2B, encoding inhibitory amino acid receptor subunits have been amplified and clo
35 ctions between opiate ligands and excitatory amino acid receptors, the ultrastructural localization o
36               After activation of excitatory amino acid receptors, there is an influx of calcium foll
37      The postnatal development of excitatory amino acid receptor types including kainate, N-methyl-D-
38 te, a wide-spectrum antagonist of excitatory amino acid receptors, while xanthurenate, an inactive an
39 e N-methyl-D-aspartic acid (NMDA) excitatory amino acid receptor with an AAV-delivered antisense olig
40  L-S-nitrosocysteine, blockade of excitatory amino acid receptors with kynurenic acid inhibited respo
41 ution of the different classes of excitatory amino acid receptors with respect to the noradrenergic n

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