ライフサイエンスコーパス: aminoacid

1 acetone, and a selected panel of circulating aminoacids.

2 on risk was not modified by polymorphisms at aminoacid 16 in the tiotropium group.

3 th asthma who are homozygous for arginine at aminoacid 16 of the beta2-adrenergic receptor (ADRB2) mi

4 Polymorphisms at aminoacid 27 did not affect exacerbation outcomes.

5 dies targeted a main epitope region at GluN1 aminoacid 369; the epitope repertoire did not differ bet

6 n of orf 65 is within the carboxyterminal 80 aminoacids, a region with little sequence similarity to

7 acterized as vaccine-matched and bore just 2 aminoacid (AA) differences from vaccine.

8 n (78% identity) and to a short stretch of 7 aminoacids also found in other homeoproteins such as Eng

9 oradic colorectal carcinomas and comparative aminoacid analysis were utilized to identify mutations i

10 e-labeling experiments with [3H]GlcN and 14C-aminoacids and analysis of GalNAc or GalNAc-ol:protein r

11 on of functional monomers based on different aminoacids and coumarin allowed isolation of polymers wi

12 Glutathione and its constituting aminoacids and dipeptides have been used as ligands.

13 eveloping diabetes, with particular focus on aminoacids and lipid metabolism.

14 might have transformed these molecules into aminoacids and nucleobases, the molecular building block

15 r detecting terpenes, sugars, organic acids, aminoacids and osmolites.

16 molecular vibrations associated with diverse aminoacids and protein conformations indicates that nucl

17 er, blood flow, metabolism (oxygen, glucose, aminoacids), and neurotransmission (dopamine, benzodiaze

18 hways were identified, including glycolysis, aminoacid, and lipids.

19 o establish the important role that hexoses, aminoacids, and fatty acids have in insulin resistance a

20 a-analysis (p=1.58 x 10(-8)), and encodes an aminoacid change (Pro67Ala) in MSH3.

21 rphism rs11762213, which causes a synonymous aminoacid change in MET (144G-->A, located in exon 2), w

22 psilon3, and varepsilon4, result in a single aminoacid change in the APOE protein.

23 We identified four mutations encoding single aminoacid changes in P450 oxidoreductase.

24 atins with readily available N-Boc-protected aminoacids followed by an intramolecular aldol reaction

25 al arginine (R374X), with the deletion of 32 aminoacids from the C-terminus of the 11 beta-HSD2 enzym

26 th different mutations of the same conserved aminoacid in antithrombin.

27 ydroxyacids, (S)-aminoalcohols and (S)-alpha-aminoacids in high e.e. and high yields, respectively.

28 nger b3a2 CML-breakpoint-derived peptide, 25 aminoacids in length (b3a2-25), was studied for its abil

29 Sycp3 cDNA and human genome sequences at the aminoacid level.

30 less encodes several short peptides 11 to 32 aminoacids long.

31 d, there is much promise that branched-chain aminoacids might provide a screening biomarker for type

32 dy were to investigate the effect of various aminoacid modifications of PYY3-36 on pharmacokinetics a

33 dependent complex with EB3 via Ser-x-Ile-Pro aminoacid motif and that disruption of STIM2-EB3 interac

34 roteins characterized by an approximately 40 aminoacid motif, the F box (so named because cyclin F wa

35 p17 (a peptide consisting of 17 NH2-terminal aminoacids of MBP) in complete Freund's adjuvant (CFA) c

36 oped in a microtiter plate format based on d-aminoacid oxidase/horseradish peroxidase (DAO/HRP)-coupl

37 acid position 161 or the insertion of a six-aminoacid peptide at the hexamer-hexamer interface disru

38 The new protocol has been tested on 15-aminoacid peptide guanylin containing four cysteine resi

39 Mac-1 and contains a common non-conservative aminoacid polymorphism, G241R.

40 se had median mutation locations further 59 (aminoacid position 1811, p=0.0018; 1671, p=0.0052; and 1

41 population than in the 87 control pedigrees (aminoacid position 2163 vs 2773, p=0.0034).

42 oxidative folding of guanylin within the 94-aminoacid prohormone proguanylin.

43 Apolipoprotein E (APOE) is a 299-aminoacid protein encoded by the APOE gene.

44 (Arg/Arg), rather than glycine (Gly/Gly), at aminoacid residue 16 of the beta2-adrenergic receptor.

45 Genotype at the 16th aminoacid residue of the beta2-adrenergic receptor affec

46 s the class II MHC region suggests that four aminoacid residues (Gly84-Gly85-Pro86-Met87) in near-com

47 hols and (S)-alpha-hydroxyacids to (S)-alpha-aminoacids, respectively.

48 10 g-->A, which produces a glycine to serine aminoacid substitution at codon 2019 (Gly2019 ser), in i

49 the population attributable risk due to this aminoacid substitution for clinically significant diseas

50 ffer from the wild type CYP2C9*1 by a single aminoacid substitution in each case.

51 The A49T aminoacid substitution in the SRD5A2 gene increased the

52 A field isolates were screened for specific aminoacid substitutions in the M2 gene known to confer d

53 We removed the polybasic aminoacids that are associated with high virulence from

54 e statistical predictors always assigning an aminoacid to the most numerous state, 3% to 7% better th

55 ved in T-cell activation, cell adhesion, and aminoacid transport.

56 In animal models, decreased excitatory aminoacid transporter 2 (EAAT2) overexpression delays di

57 and sufficient for the downregulation of the aminoacid transporter TOV3.

58 mmercially available umami products and some aminoacids were submitted to sensory analysis.