1 AI lysine modification to chlorolysine and 2-aminoadipic acid.

2 and rate of the metabolism of an L-[1-13C]-2-aminoadipic acid ([13C]AAA) tracer and of whole-body L-[

3                             The metabolite 2-aminoadipic acid (2-AAA) was most strongly associated wi

4 llysine, ALL) and its oxidized end-product 2-aminoadipic acid (2-AAA) were determined as a function o

5 n to glutamate was also blocked by L-2-alpha aminoadipic acid, a selective astrocyte toxin, and paxil

6 rogenase in liver and plasma levels of alpha-aminoadipic acid (AAA), both of which act as indicators

7 ide strands by a turn unit composed of (S)-2-aminoadipic acid (Aaa).

8                   Replacing Asp25 with alpha-aminoadipic acid (Aad) adjusts the pH50 to 6.74, matchin

9 ehyde) that can be further oxidised to alpha-aminoadipic acid and form Schiff bases structures.

10  However, we find that the activity of alpha-aminoadipic acid-delta-semialdehyde (AAS) dehydrogenase

11 h a glutamate transporter substrate (l-alpha-aminoadipic acid) increased the neuronal firing rate, su

12 either of the selective glial toxins L-alpha-aminoadipic acid (L-AA) or fluorocitrate (FC) blocked mG

13 via intracerebroventricular application of l-aminoadipic acid (l-AAA).

14 cted analogues of proline, pipecolic acid, 2-aminoadipic acid, or glutamic acid.

15 kainate (KA), dihydrokainate (DHK) and alpha-aminoadipic acid produced less than 30% inhibition at 1

16 is pathway catalyzes the conversion of alpha-aminoadipic acid to alpha-aminoadipic-delta-semialdehyde

17                                          The aminoadipic acid unit shows patterns of NOEs and couplin