ライフサイエンスコーパス: aminobutyric acid

1 ment of glutamate, glutamine and GABA (gamma-aminobutyric acid).

2 as a receptor of the priming activator beta-aminobutyric acid.

3 gamma-Aminobutyric acid, 4-hydroxyproline, glycine, leucine+is

4 lateral infusions into the RMTg of the gamma-aminobutyric acid A (GABAA) agonist, muscimol, indeed ac

5 ha cell dysfunction was due to reduced gamma-aminobutyric acid A receptor activation in pancreatic al

6 Nlrp3 inflammasome were independent of gamma-aminobutyric acid A receptor activation or N-methyl-d-as

7 ve action on the omega1 subtype of the gamma-aminobutyric acid A receptor, zolpidem tartrate presents

8 to awake NHP brain PET studies with a gamma-aminobutyric acid A-benzodiazepine receptor ligand, (11)

9 Here, we report on the effects of beta-aminobutyric acid, a priming agent with an exceptionally

10 l-peptidase-like protein-6 (DPPX), and gamma-aminobutyric acid-A receptor (GABAAR).

11 o neurotransmitter receptor genes, the gamma-Aminobutyric acid-A receptor delta and gamma-aminobutyri

12 protein-like 2), glycine receptor, and gamma-aminobutyric acid-A receptor.

13 -isoxazolepropionic acid, glycine, and gamma-aminobutyric acid-A receptors), were prevalent in patien

14 gamma-Aminobutyric acid aminotransferase (GABA-AT) is a pyrido

15 in barrier and inhibit the activity of gamma-aminobutyric acid aminotransferase (GABA-AT), the enzyme

16 ivator that regulates transcription of gamma-aminobutyric acid aminotransferase (GABA-AT; GabT) upon

17 GAD65 synthesizes gamma-aminobutyric acid, an important autocrine and paracrine

18 e stress, and is associated with GABA (gamma-aminobutyric acid, an inhibitory neurotransmitter) signa

19 by MOA: sodium channel blockers (SC), gamma-aminobutyric acid analogs (G), synaptic vesicle protein

20 furosine (2-FM-lysine), 2-furoylmethyl-gamma-aminobutyric acid and 2-FM-arginine were detected.

21 some neurotransmitter systems, such as gamma-aminobutyric acid and glutamate, mainly in the cortex.

22 dependent functional plasticity of the gamma-aminobutyric acid and glycinergic system by targeting KC

23 sidues p2C and p5M to the conservative alpha-aminobutyric acid and norleucine, respectively, signific

24 the inhibitory neurotransmitter GABA (gamma-aminobutyric acid) and are inhibited by iSPNs and dSPNs

25 ll as regulating release of glutamate, gamma-aminobutyric acid, and acetylcholine from presynaptic ax

26 d by l-glutamate (l-Glu), l-aspartate, gamma-aminobutyric acid, and acetylcholine, with l-Glu eliciti

27 eractions between the endocannabinoid, gamma-aminobutyric acid, and glutamate systems and their indiv

28 nesis including synthesis of glutamate/gamma-aminobutyric acid as a potential transcriptional target

29 the neurons also release glutamate and gamma-aminobutyric acid as cotransmitters, with striking regio

30 Aminobutyric acid-A receptor delta and gamma-aminobutyric acid B receptor subunit 1; their differenti

31 ging evidence suggests that functional gamma-aminobutyric acid B receptors (GABABRs) are expressed by

32 xazolepropionic acid receptor (AMPAR), gamma-aminobutyric acid-B receptor (GABABR), leucine-rich glio

33 eptors, particularly retinoic acid and gamma-aminobutyric acid-B2 receptor signalling, among novel me

34 h genes responsive to the priming agent beta-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSC

35 n genes responsive to the priming agent beta-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSC

36 donic acid (AA), jasmonic acid (JA) and beta-aminobutyric acid (BABA).

37 Decreased expression of the gamma-aminobutyric acid binding site, a transmembrane domain a

38 BrdU(+) /GABA(+) (gamma-aminobutyric acid) cells were also found but no new Cr(+

39 the specific impairment of perisomatic gamma-aminobutyric acid circuits are hallmarks of the schizoph

40 d lower limb was associated with lower gamma-aminobutyric acid concentration in the sensorimotor cort

41 Additionally, the reduced gamma-aminobutyric acid concentration may contribute to the ne

42 r adults to a likely decrease in GABA (Gamma Aminobutyric Acid) concentration in visual cortex, an as

43 ls were used to compare differences in gamma-aminobutyric acid concentrations between patients and co

44 injection to the NAcSh decreased local gamma-aminobutyric acid concentrations.

45 Here we report a high GABA (gamma-aminobutyric acid) content in reactive astrocytes in the

46 Fast glutamate and gamma-aminobutyric acid cotransmission convey discrete pattern

47 the distribution of neurotransmitters-gamma-aminobutyric acid, dopamine and serotonin-with high spec

48 real-time-corrected three-dimensional gamma-aminobutyric acid-edited magnetic resonance (MR) spectro

49 -range projections that activate local gamma-aminobutyric acid-ergic (GABAergic) circuits.

50 iety of neurotransmitters, such as the gamma-aminobutyric acid-ergic system, the study of prefrontal

51 part by direction-selective release of gamma-aminobutyric acid from starburst amacrine cells onto dir

52 The effects of gamma-aminobutyric acid (GABA) A receptor activation on physio

53 mine whether antecedent stimulation of gamma-aminobutyric acid (GABA) A receptors with the benzodiaze

54 gamma-Aminobutyric acid (GABA) administration can inhibit infl

55 Gamma-aminobutyric acid (GABA) and enkephalin tonically inhibi

56 amino acid neurotransmitter systems of gamma-aminobutyric acid (GABA) and glutamate, respectively, pl

57 Levels of gamma-aminobutyric acid (GABA) and glutamic acid decarboxylase

58 e inhibitory transmitters include both gamma-aminobutyric acid (GABA) and glycine (GLY).

59 " proton in the decarboxylated product gamma-aminobutyric acid (GABA) and is then exported via GadC.

60 of glutamate, inhibitory transmitters gamma-aminobutyric acid (GABA) and N-acetylaspartylglutamate (

61 ecarboxylase, suggesting that they use gamma-aminobutyric acid (GABA) as their neurotransmitter.

62 The deficit reflected weakened gamma-aminobutyric acid (GABA) circuits and compromised postna

63 hat pairing of acetylcholine (ACh) and gamma-aminobutyric acid (GABA) Comment: Please use the greek l

64 Gamma-aminobutyric acid (GABA) concentrations and GAD67 protei

65 pertensive activity due to their large gamma-aminobutyric acid (GABA) content (6.8-10.6 mg/g) and ang

66 The inhibitory neurotransmitter gamma-aminobutyric acid (GABA) contributes to the network dyna

67 hanced vulnerability may be related to gamma-aminobutyric acid (GABA) deficits observed in schizophre

68 estored the impaired oxytocin-mediated gamma-aminobutyric acid (GABA) excitatory-inhibitory shift dur

69 f the cockroach Periplaneta americana, gamma-aminobutyric acid (GABA) has been identified as the pote

70 to map histamine (HA), FMRF-amide, and gamma-aminobutyric acid (GABA) immunoreactivity in the central

71 o compared the distribution of ASP and gamma-aminobutyric acid (GABA) in brainstem neurons by using d

72 Abnormally elevated levels of gamma-aminobutyric acid (GABA) in the medial prefrontal cortex

73 ehaviors were consistent with abnormal gamma-aminobutyric acid (GABA) interneuron function, which pro

74 rtate receptor (NMDAR) hypofunction on gamma-aminobutyric acid (GABA) interneurons disinhibiting pyra

75 Gamma-aminobutyric acid (GABA) is a major inhibitory neurotran

76 Gamma-aminobutyric acid (GABA) is a widely conserved signaling

77 We discovered that gamma-aminobutyric acid (GABA) is co-released with its functio

78 The main inhibitory neurotransmitter gamma-aminobutyric acid (GABA) is known to play a pivotal role

79 he role of VGLUT3 in neurons releasing gamma-aminobutyric acid (GABA) is not settled.

80 gamma-Aminobutyric acid (GABA) is one of the major inhibitory

81 gamma-Aminobutyric acid (GABA) is the major inhibitory transmi

82 gamma-Aminobutyric acid (GABA) is the most abundant inhibitory

83 gamma-Aminobutyric acid (GABA) is the primary inhibitory neuro

84 In contrast, KATPHI islets had low gamma-aminobutyric acid (GABA) levels and lacked (13)C incorpo

85 Low-pressure plasma also increased gamma-aminobutyric acid (GABA) levels from approximately 19 to

86 and at that time, postmortem striatal gamma-aminobutyric acid (GABA) levels were elevated and mutant

87 he density of cartridges detectable by gamma-aminobutyric acid (GABA) membrane transporter 1 immunore

88 Early-born gamma-aminobutyric acid (GABA) neurons (EBGNs) are major compo

89 gonists interfere with the function of gamma-aminobutyric acid (GABA) neurons and alter the brain osc

90 stimulation of mouse zona incerta (ZI) gamma-aminobutyric acid (GABA) neurons or their axonal project

91 show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to cue prese

92 s expression in VTA 5-HT2CR expressing gamma-aminobutyric acid (GABA) neurons, but not 5-HT2CR expres

93 Aberrant glutamate and gamma-aminobutyric acid (GABA) neurotransmission contribute to

94 Gamma-aminobutyric acid (GABA) neurotransmission in the latera

95 either the amino acid (AA) transmitter gamma-aminobutyric acid (GABA) or glutamate.

96 ed that dopaminergic neurons often use gamma-aminobutyric acid (GABA) or glutamatergic cotransmission

97 s did not confirm colocalizations with gamma-aminobutyric acid (GABA) or the circadian coupling pepti

98 sponses to hypoglycemia, and increased gamma-aminobutyric acid (GABA) output contributes to counterre

99 The non-protein amino acid, gamma-aminobutyric acid (GABA) rapidly accumulates in plant ti

100 neurites are also immunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA Ralpha1

101 Nonselectively reducing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition synchroni

102 ngly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

103 rinking, and basal and alcohol-induced gamma-aminobutyric acid (GABA) release in the central nucleus

104 Our results demonstrate that the gamma-aminobutyric acid (GABA) release is lower and opioid eff

105 We found that local gamma-aminobutyric acid (GABA) release on dendrites of mouse c

106 es revealed a significant reduction in gamma-aminobutyric acid (GABA) release probability without alt

107 r, in vitro, neurounina-1 also reduced gamma-aminobutyric acid (GABA) release, enhanced GABA(A) curre

108 nitrate stimulated PGC1alpha-mediated gamma-aminobutyric acid (GABA) secretion from muscle.

109 xendin-4, a GLP-1 receptor agonist, on gamma-aminobutyric acid (GABA) signaling in hippocampal CA3 py

110 Altered gamma-aminobutyric acid (GABA) signaling in the prefrontal cor

111 target, NKCC1, initiates the switch in gamma-aminobutyric acid (GABA) signaling, limits early spontan

112 al cord to determine whether glutamate-gamma-aminobutyric acid (GABA) switching is cell autonomous.

113 Deficits in the gamma-aminobutyric acid (GABA) system have been reported in po

114 The gamma-aminobutyric acid (GABA) system in the nucleus accumbens

115 ociated with reduced concentrations of gamma-aminobutyric acid (GABA) that are normalized by antidepr

116 onally stained with antibodies against gamma-aminobutyric acid (GABA) to identify GABAergic interneur

117 te signaling (P-value=7.22 x 10(-15)), gamma-aminobutyric acid (GABA) transport (P-value=1.36 x 10(-1

118 leads to a large increase in vesicular gamma-aminobutyric acid (GABA) transporter (vGAT) and glutamic

119 We saw that stimulation of vesicular gamma-aminobutyric acid (GABA) transporter (VGAT)-expressing B

120 encoded by SLC6A1, is one of the major gamma-aminobutyric acid (GABA) transporters in the brain and i

121 sis of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) using pyridoxal 5'-phosphate as

122 evels of glutamate+glutamine (Glx) and gamma-aminobutyric acid (GABA) were measured before, during, a

123 eic acids, as well as higher amount of gamma-aminobutyric acid (GABA) were observed in biodynamic gra

124 investigate in vivo concentrations of gamma-aminobutyric acid (GABA) within primary and secondary mo

125 excitatory (glutamate) and inhibitory (gamma aminobutyric acid (GABA)) neurotransmitter circuits in a

126 Gamma aminobutyric acid (GABA), a neurotransmitter of the cent

127 tant ion channels, gated by glutamate, gamma-aminobutyric acid (GABA), and acetylcholine, also have a

128 ibitory neurotransmitter of the brain, gamma-aminobutyric acid (GABA), can be released through the re

129 rentially methylated probes implicated gamma-aminobutyric acid (GABA), dopamine and serotonin neurotr

130 r various neurotransmitters-glutamate, gamma-aminobutyric acid (GABA), dopamine, serotonin, acetylcho

131 dicted to form anion channels gated by gamma-aminobutyric acid (GABA), glutamate, histamine, or chang

132 ajor sleep-promoting neurotransmitter, gamma-aminobutyric acid (GABA), in the GABA shunt generates ca

133 ay of the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), in this putative imbalance: GA

134 Low levels of gamma-aminobutyric acid (GABA), one of two major neurotransmit

135 rs for the inhibitory neurotransmitter gamma-aminobutyric acid (GABA), particularly within the fronta

136 ecently, it has been demonstrated that gamma-aminobutyric acid (GABA), the chief inhibitory neurotran

137 gamma-Aminobutyric acid (GABA), the major inhibitory neurotran

138 hila optic lobes that possibly release gamma aminobutyric acid (GABA), the major inhibitory neurotran

139 A considerable increase in gamma-aminobutyric acid (GABA), together with some other amino

140 ation time and temperature to maximise gamma-aminobutyric acid (GABA), total phenolics compounds (TPC

141 acting neurotransmitters glutamate and gamma-aminobutyric acid (GABA), two major neurotransmitters in

142 a potential lipophilic bioisostere of gamma-aminobutyric acid (GABA), various late-stage diversifica

143 The inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), was then actively delivered th

144 ometry (MS)-based transport assays for gamma-aminobutyric acid (GABA), which is the major inhibitory

145 nal fluid (CSF)-induced enhancement of gamma-aminobutyric acid (GABA)-A receptor activity was found i

146 spartate (NMDA) glutamate receptor and gamma-aminobutyric acid (GABA)-A receptor during progression o

147 ith a positive allosteric modulator of gamma-aminobutyric acid (GABA)-A receptors in cerebrospinal fl

148 unique role of GABARAPs, in particular gamma-aminobutyric acid (GABA)-A-receptor-associated protein-l

149 amus, a well-defined system containing gamma-aminobutyric acid (GABA)-ergic and glutamatergic neurons

150 n is likely due to a reduced number of gamma-aminobutyric acid (GABA)-ergic boutons, which may result

151 Gamma-aminobutyric acid (GABA)-ergic disturbances are hallmark

152 uses stress-sensitive, PVH-projecting, gamma-aminobutyric acid (GABA)-ergic neurons as representing a

153 Dysfunction related to gamma-aminobutyric acid (GABA)-ergic neurotransmission in the

154 The regulation of synaptic strength at gamma-aminobutyric acid (GABA)-ergic synapses is dependent on

155 hanges the number of dopamine (DA)- or gamma aminobutyric acid (GABA)-expressing neurons, with corres

156 This study describes the gamma-aminobutyric acid (GABA)-immunoreactive (GABA-ir) neuron

157 Calretinin- and gamma-aminobutyric acid (GABA)-immunoreactive (IR) cells were

158 spines received inputs from symmetric gamma-aminobutyric acid (GABA)-immunoreactive terminals, where

159 spartate (NMDA) excitation balanced by gamma-Aminobutyric acid (GABA)-mediated inhibition and leads t

160 ve higher IFITM levels and deficits in gamma-aminobutyric acid (GABA)-related markers that are simila

161 At the circuit level, these gamma-aminobutyric acid (GABA)-releasing projections target hi

162 that CR-positive terminals lacked the gamma-aminobutyric acid (GABA)-synthesizing enzyme glutamate d

163 ion of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA).

164 tions in regulation of the HPA axis by gamma-aminobutyric acid (GABA).

165 h slightly different sensitivities, on gamma-aminobutyric acid (GABA)A , alpha-amino-3-hydroxy-5-meth

166 Native gamma-aminobutyric acid (GABA)A receptors consisting of alpha4

167 gamma-Aminobutyric acid (GABA)A receptors were blocked by bicu

168 be due to internalization of synaptic gamma-aminobutyric acid (GABA)A receptors, and withdrawal of b

169 s modulated by intrinsic and extrinsic gamma-aminobutyric acid (GABA)ergic and glutamatergic afferent

170 to involve functional deficits in both gamma-aminobutyric acid (GABA)ergic and glutamatergic synaptic

171 change factor selectively localized to gamma-aminobutyric acid (GABA)ergic and glycinergic postsynaps

172 rgic cells and decreased the number of gamma-aminobutyric acid (GABA)ergic cells, whereas (-)BayK 864

173 ergent findings indicate that cortical gamma-aminobutyric acid (GABA)ergic circuitry is altered in sc

174 inergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements involved with the

175 inergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements of these nuclei.

176 s glutamatergic excitatory neurons and gamma-aminobutyric acid (GABA)ergic inhibitory interneurons.

177 uperior colliculus receives inhibitory gamma-aminobutyric acid (GABA)ergic input from the basal gangl

178 gamma-Aminobutyric acid (GABA)ergic inputs are strategically p

179 egmental nucleus (RMTg), which contain gamma-aminobutyric acid (GABA)ergic neurons that mediate rewar

180 types (cholinergic, glutamatergic, and gamma-aminobutyric acid (GABA)ergic neurons) across its differ

181 gic but also include a small number of gamma-aminobutyric acid (GABA)ergic neurons.

182 potentiate glutamatergic and attenuate gamma-aminobutyric acid (GABA)ergic neurotransmission, and BDN

183 rontal cortex (mPFC) glutamatergic and gamma-aminobutyric acid (GABA)ergic receptors in controlling r

184 x underlies the developmental shift in gamma-aminobutyric acid (GABA)ergic responses, whereas the phy

185 ed a form of long-term potentiation of gamma-aminobutyric acid (GABA)ergic synapses on these neurons

186 e, and are also sites of clustering of gamma-aminobutyric acid (GABA)ergic synapses.

187 ognitive alterations and damage to the gamma-aminobutyric acid (GABA)ergic system in the hippocampus

188 he retina, the ratio of glycinergic to gamma-aminobutyric acid (GABA)ergic to amacrine cells remained

189 Abnormalities in prefrontal gamma aminobutyric acid (GABA)ergic transmission, particularly

190 tic connectivity and function of a few gamma-aminobutyric acid (GABA)ergic wide-field amacrine cells

191 ent solely in neurons, specifically in gamma-aminobutyric acid (GABA)ergic, glutamatergic, and catech

192 heel-running performances of wildtype (gamma-aminobutyric acid [GABA]-CB(1)(+)/(+)) and mutant (GABA-

193 tatory (glutamatergic) and inhibitory (gamma-aminobutyric acid [GABA]ergic) neurons of the prefrontal

194 The gamma-aminobutyric acid (GABAA) receptor gamma2 subunit gene,

195 iated with mutations in the inhibitory gamma-aminobutyric acid (GABAA) receptor gamma2 subunit gene,

196 ne, a positive allosteric modulator of gamma-aminobutyric acid (GABAA) receptors, for the treatment o

197 synaptic inhibition mediated by type A gamma-aminobutyric acid (GABAA) receptors, which are Cl(-)-per

198 Ionotropic receptors of gamma-aminobutyric acid (GABAAR) regulate neuronal inhibition

199 Val > Lys, Tyr, Pro > hydroxyproline > alpha-aminobutyric acid > Gln, Thr, Ser > Glu, Ala, Gly, Asn,

200 altered brain levels of glutamate and gamma-aminobutyric acid have been identified in both animal an

201 study, we examined the distribution of gamma-aminobutyric-acid;-immunoreactive (GABA-ir) neurons in f

202 uropil organization and the pattern of gamma-aminobutyric acid immunostaining of the medulla and LOX

203 ortical axons and normal intracortical gamma-aminobutyric acid inhibition in contrast with what has b

204 Gamma-aminobutyric acid is the principle inhibitory neurotrans

205 differences in prefrontal or striatal gamma-aminobutyric acid level.

206 s of this study were to investigate if gamma-aminobutyric acid levels (i) are abnormal in patients wi

207 Specifically for each unit decrease in gamma-aminobutyric acid levels (in mM), there was a predicted

208 Patients had significantly lower gamma-aminobutyric acid levels in the hippocampus (adjusted di

209 ate + glutamine, N-acetylaspartate, or gamma-aminobutyric acid levels in the left DLPFC.

210 nance spectroscopy at 3 T, to quantify gamma-aminobutyric acid levels in the prefrontal cortex, right

211 This study suggests that reduced gamma-aminobutyric acid levels reflect pathological abnormalit

212 d to assess cortical glutamatergic and gamma-aminobutyric acid-mediated tone in adults with major dep

213 The gamma-aminobutyric acid modulator propofol induces neuronal ce

214 Mice lacking GIRK2 in gamma-aminobutyric acid neurons (GAD-Cre:Girk2(flox/flox) mice

215 nd a specific population of inhibitory gamma-aminobutyric acid neurons (i.e., parvalbumin-containing

216 endent signaling in dorsal hippocampal gamma-aminobutyric acid neurons but no evident behavioral phen

217 ting and inhibiting neighbouring GABA (gamma-aminobutyric acid) neurons in the ventral tegmental area

218 he pre- and postsynaptic components of gamma-aminobutyric acid neurotransmission and in the density o

219 y supports the idea that modulation of gamma-aminobutyric acid neurotransmission may be an important

220 ered cyanobacterial TCA cycle (via the gamma-aminobutyric acid pathway or alpha-ketoglutarate decarbo

221 ion causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit composition

222 echanisms controlling the metabotropic gamma-aminobutyric acid receptor (GABAB) cell surface stabilit

223 d antidepressants cause a shift in the gamma-aminobutyric acid receptor (GABABR) signaling pathway, s

224 he type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potential or

225 d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 patient

226 te-a glutamate receptor antagonist and gamma-aminobutyric acid receptor agonist-would result in effic

227 ration model, we studied glutamate and gamma-aminobutyric acid receptor regulation in the synaptic me

228 behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the hypoph

229 in's effects on energy balance through gamma-aminobutyric acid receptor signaling.

230 The gamma-aminobutyric acid receptor-associated protein (GABARAP)

231 duced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GABARAP)+

232 ociated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GABARAP)]

233 ell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001).

234 become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells can pr

235 Type-A gamma-aminobutyric acid receptors (GABAARs) are the principal

236 The accumulation of gamma-aminobutyric acid receptors (GABAARs) at the appropriate

237 synaptic inhibition upon activation of gamma-aminobutyric acid receptors.

238 ncludes the metabotropic glutamate and gamma-aminobutyric acid receptors.

239 Specifically, deficits in inhibitory gamma-aminobutyric acid regulating excitatory cell input/outpu

240 B1R); 3) binds to CB1R, which inhibits gamma-aminobutyric acid release from the cholecystokinin-conta

241 se brain, prevented ethanol-stimulated gamma-aminobutyric acid release in the central amygdala, and r

242 Elevated gamma-aminobutyric acid release under chronic stress is accomp

243 donoylglycerol-dependent inhibition of gamma-aminobutyric acid release without altering postsynaptic

244 t chronic stress increases presynaptic gamma-aminobutyric acid release.

245 ell types include hypocretin and GABA (gamma-aminobutyric-acid)-releasing neurons of the lateral hypo

246 esult from the synchronous activity of gamma-aminobutyric acid-releasing cells.

247 including the migration of GABAergic (gamma-aminobutyric-acid-releasing) neurons from ventral to dor

248 Here we show that a GABAergic (gamma-aminobutyric-acid-releasing) pathway originating from th

249 tion is mediated largely by GABAergic (gamma-aminobutyric acid-secreting) interneurons, a cell type t

250 Moreover, up-regulation of the gamma-aminobutyric acid shunt and alanine metabolism explained

251 nate-semialdehyde dehydrogenase in the gamma-aminobutyric acid shunt pathway and an aconitase family

252 mentation, alanine metabolism, and the gamma-aminobutyric acid shunt, while [(13)C]glutamate and [(15

253 be temporarily reverted by inhibiting gamma-aminobutyric acid signaling but not by a Ras-ERK blockad

254 This results in delayed gamma-aminobutyric acid switch and higher susceptibility to se

255 process, the excitatory-to-inhibitory gamma-aminobutyric acid switch; defects in this switch have be

256 Immunohistochemistry for the gamma aminobutyric acid synthesizing enzyme glutamic acid deca

257 l tremor may be neurodegenerative; low gamma aminobutyric acid tone seems to be a central feature of

258 reated with melatonin exhibited higher gamma-aminobutyric acid transaminase (GABA-T) enzyme activity

259 es the CG7433 protein, a mitochondrial gamma-aminobutyric acid transaminase (GABAT), and reduces GABA

260 n prefrontal signaling pathways (e.g., gamma-aminobutyric acid transmission) and neural network synch

261 sion or pharmacological enhancement of gamma-aminobutyric acid transmission.

262 bitory synaptic currents and vesicular gamma-aminobutyric acid transporter (vGAT) staining intensity

263 Using vesicular gamma-aminobutyric acid transporter-channelrhodopsin 2-enhance

264 (-)](i) is an important determinant of gamma-aminobutyric acid type A (GABA(A)) receptor (GABA(A)R)-m

265 Benzodiazepines modulate gamma-aminobutyric acid type A (GABA(A)) receptors throughout

266 ial firing by local application of the gamma-aminobutyric acid type A (GABA-A) agonist muscimol incre

267 entified four de novo mutations in the gamma-aminobutyric acid type A (GABAA ) receptor beta3 subunit

268 entified a cysteine substituted mutant gamma-aminobutyric acid type A (GABAA) receptor with unique ch

269 Proteostasis maintenance of gamma-aminobutyric acid type A (GABAA) receptors dictates thei

270 rosteroids at the same binding site on gamma-aminobutyric acid type A (GABAA) receptors was evaluated

271 isplayed micromolar affinity for brain gamma-aminobutyric acid type A (GABAA) receptors.

272 The extrasynaptic alpha-5 gamma-aminobutyric acid type A receptor (alpha5-GABAAR) regula

273 steroids are efficacious modulators of gamma-aminobutyric acid type A receptor (GABA(A)) receptor fun

274 The gamma-aminobutyric acid type A receptor (GABAA-R) is a major i

275 deficient neurons show reduced surface gamma-aminobutyric acid type A receptor (GABAAR) levels and im

276 sporter expression and impaired spinal gamma-aminobutyric acid type A receptor function, indicative o

277 wever, DCS-LTD persists despite either gamma-aminobutyric acid type A receptor or N-methyl-D-aspartat

278 tic and biological evidence implicates gamma-aminobutyric acid type A receptor subunits on chromosome

279 ibutor to the central disinhibition of gamma-aminobutyric acid type A receptor- and glycine receptor-

280 Alpha-5 gamma-aminobutyric acid type A receptors (alpha5-GABAARs) are

281 ted primarily to increased activity of gamma-aminobutyric acid type A receptors (GABA(A)Rs), and it i

282 region of glycine receptors (GlyRs) or gamma-aminobutyric acid type A receptors (GABA(A)Rs), which ar

283 pe 1 receptor) receptors and modulates gamma-aminobutyric acid type A receptors (GABAAR).

284 c acetylcholine receptors (nAChRs) and gamma-aminobutyric acid type A receptors (GABAARs) are members

285 t act as convulsants and inhibitors of gamma-aminobutyric acid type A receptors (GABAARs) rather than

286 as a positive allosteric modulator of gamma-aminobutyric acid type A receptors (GABAARs), an interac

287 Extrasynaptic gamma-aminobutyric acid type A receptors (GABAARs),which contr

288 hibitory neurotransmission mediated by gamma-aminobutyric acid type A receptors (GABAARs).

289 he scaffolding protein Gephyrin and of gamma-aminobutyric acid type A receptors at inhibitory neurona

290 ll known for benzodiazepines acting at gamma-aminobutyric acid type A receptors.

291 ing synaptic inhibition through GABAA (gamma aminobutyric acid type A) and glycine receptors depends

292 n of synaptic acetylcholine and GABAA (gamma-aminobutyric acid type A) receptors into extrasynaptic c

293 gamma-Aminobutyric acid type B (GABAB) receptor autoantibodies

294 Autoantibodies to the gamma-aminobutyric acid type B (GABAB) receptor have recently

295 The gamma-aminobutyric acid type B (GABAB) receptor undergoes spli

296 ned neuronal inhibition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which are he

297 ol, like rapid antidepressants, alters gamma-aminobutyric acid type B receptor (GABABR) expression an

298 gamma-Aminobutyric acid type B receptor autoimmunity deserves

299 The gamma-aminobutyric acid type B-receptor agonist lesogaberan (A

300 l-norvaline and, to a lesser extent, C4 of l-aminobutyric acid when SyrB1 presents these non-native a