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1 from the strong liver carcinogen N-acetyl-2-aminofluorene.
2 be FAF [N-(2'-deoxyguanosin-8-yl)-7-fluoro-2-aminofluorene].
3 catalyzed the conversion of the arylamine 2-aminofluorene (2-AF) to 2-acetylaminofluorene (2-AAF) by
4 , 2-amino-3-methylimidazo[4,5-f]quinoline, 2-aminofluorene, 6-aminochrysene and its 1,2-dihydrodiol,
5 by the C-8 guanine DNA adduct formed by N-2-aminofluorene, a lesion that does not appear to be prefe
6 itioned N-2-aminofluorene (AF) or N-acetyl-2-aminofluorene (AAF) adduct and have determined both the
7 dimers,(6-4) photoproducts, and N-acetoxy-2-aminofluorene (AAF) adducts by an excision repair mechan
8 formed by aromatic amines such as N-acetyl-2-aminofluorene (AAF) and N-2-aminofluorene (AF) are known
10 ducts of 2-aminofluorene (AF) and N-acetyl-2-aminofluorene (AAF) was observed in a bubble of three mi
11 primer-template was modified with N-acetyl-2-aminofluorene (AAF), a well-studied carcinogenic adduct.
12 such as 2-aminofluorene (AF) and N-acetyl-2-aminofluorene (AAF), covalently bind DNA bases and promo
13 A adducts, 2-aminofluoene (AF) or N-acetyl-2-aminofluorene (AAF), using single-molecule FRET (smFRET)
14 ic XPA bound to the DNA adduct of N-acetyl-2-aminofluorene (AAF), while showing little affinity for n
17 e model for transcription-coupled repair, an aminofluorene adduct located on the transcribed strand w
18 on assay were constructed with either an N-2-aminofluorene adduct or the helix-distorting N-2-acetyla
19 syn thymine-thymine dimer, as well as acetyl aminofluorene adducted- and cisplatinated-guanine residu
20 n efficiency for the C8-guanine adducts of 2-aminofluorene (AF) and N-acetyl-2-aminofluorene (AAF) wa
22 ch as N-acetyl-2-aminofluorene (AAF) and N-2-aminofluorene (AF) are known to cause mutations by inter
23 ontaining a site-specifically positioned N-2-aminofluorene (AF) or N-acetyl-2-aminofluorene (AAF) add
24 taining a site-specific C8-guanine adduct of aminofluorene (AF) or N-acetyl-2-aminofluorene (AAF).
25 arcinogens 2-acetylaminofluorene (AAF) and 2-aminofluorene (AF) that form covalent adducts at the C8-
26 guanine adducts formed by the carcinogens 2-aminofluorene (AF), N:-acetyl-2-acetylaminofluorene (AAF
28 structural issues related to the capacity of aminofluorene [AF] for frameshift mutations of the -2 ty
29 hift mutagenesis induced by the carcinogen 2-aminofluorene and its analogues in Escherichia coli.
30 tion of the modified dG with stacking of the aminofluorene and the looped out position of the partner
31 ith benzo[a]pyrene diol epoxide, N-hydroxy-2-aminofluorene, and aflatoxin B1 8,9-epoxide in (1) naked
32 low dose of 2-AAF (and its analogs, 2-AF [2-aminofluorene] and N-OH-2-AAF) elicited a mitogenic resp
33 structural study has been undertaken on the aminofluorene-C8-dG ([AF]dG) adduct located at a single
34 structural study has been undertaken on the aminofluorene-C8-dG ([AF]dG) adduct located at a single-
35 osite it are stacked into the helix with the aminofluorene chromophore displaced into the minor groov
37 alladium-catalyzed asymmetric arylation of 9-aminofluorene-derived imines using a chiral dialkylbiary
38 ene (dG-AAF) and N-(2'-deoxyguanosin-8-yl)-2-aminofluorene (dG-AF) adducts positioned in the Nar I re
40 uorene (dG-AAF) and N-(deoxyguanosin-8-yl)-2-aminofluorene (dG-AF), were used as DNA templates in pri
42 e is the pair of N-(2'-deoxyguanosin-8-yl)-2-aminofluorene (dG-C8-AF) and N-(2'-deoxyguanosin-8-yl)-2
43 uorene (dG-C8-AAF), N-(deoxyguanosin-8-yl)-2-aminofluorene (dG-C8-AF), and 3-(deoxyguanosin-N(2)()-yl
44 s containing 7,8-dihydro-8-oxoguanine, dG-C8-aminofluorene, dG-C8-(acetylamino)fluorene, and the mode
47 ived from the carcinogen 7-fluoro-N-acetyl-2-aminofluorene (FAAF) in three structural contexts: as a
48 tylated N-(2'-deoxyguanosin-8-yl)-7-fluoro-2-aminofluorene (FAF) adducts in the same NarI sequence ar
49 rinated analogs of 4-aminobiphenyl (FABP), 2-aminofluorene (FAF) or 2-acetylaminofluorene (FAAF), and
51 A adduct [N-(2'-deoxyguanosin-yl)-7-fluoro-2-aminofluorene (FAF)] adopts the so-called 'wedge' (W) co
53 anine adducts, the N-(2'-deoxyguanosin-8-yl)-aminofluorene (G-AF) and N-2-(2'-deoxyguanosin-8-yl)-ace
54 dG-FAF, N-(2'-deoxyguanosin-8-yl)-7-fluoro-2-aminofluorene) has been investigated in the presence of
55 [N-(deoxyguanosin-8-yl)-N-acetyl-7-fluoro-2-aminofluorene] in methanol at -30 degrees C exhibited fo
57 ing rabbit P450 1A2-catalyzed N,N-dimethyl-2-aminofluorene N-oxygenation, human P450 3A4-catalyzed qu
58 * was either AF [N-(2'-deoxyguanosin-8-yl)-2-aminofluorene] or the 19F probe FAF [N-(2'-deoxyguanosin
60 se displacement-intercalation type where the aminofluorene ring is intercalated into the helix betwee
62 .dG22 base pair, while the other face of the aminofluorene ring is stacked with the purine ring of th
63 s accompanied by stacking of one face of the aminofluorene ring of [AF]dG4 with the dC5.dG22 base pai
65 and are dependent on the positioning of the aminofluorene rings: B is in the "B-DNA" major groove, S
66 lated AF lesion [N-(2'-deoxyguanosin-8-yl)-2-aminofluorene], the major adduct derived from the strong
67 omycin C, benzo[a]pyrene, aflatoxin B1 and 2-aminofluorene, were compared with the aim of investigati
68 eoxynucleotides modified by the carcinogen 2-aminofluorene, whose sequence around the lesion was vari
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