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1 were influenced by P1-receptor blockade via aminophylline.
2 in cell lines treated by serum starvation or aminophylline.
3 t is consistent with the pharmacokinetics of aminophylline.
4 n after infusion and are rapidly reversed by aminophylline.
5 , is treated with caffeine, theophylline, or aminophylline.
9 ich degrades extracellular adenosine, and by aminophylline, a general adenosine receptor antagonist,
10 ignificantly attenuated by pretreatment with aminophylline, a non-selective Ado receptor antagonist,
11 rterial saline (control) and combined L-NMMA-aminophylline (adenosine receptor antagonist) administra
13 nonselective adenosine receptor antagonist, aminophylline (AMO; theophylline ethylenediamine) and, f
14 A synergistic effect was observed between aminophylline and dexamethasone in maintaining HDAC2 exp
15 roliferation on CEM ALL cells, we found that aminophylline and other nonspecific phosphodiesterase (P
16 -acting inhaled beta2-stimulants, parenteral aminophylline, and slow-release theophylline preparation
17 ally approved pharmacological agents such as aminophylline, ascorbic acid, or furosemide increased or
18 65%) versus older adults (360 +/- 80%), and aminophylline blunted these responses by approximately 5
19 the first 2 hrs after administration of the aminophylline bolus and then returned to baseline by 4 t
22 tion range, 3 x 10(-7) to 1 x 10(-4) M), and aminophylline (concentration range, 10(-7) to 10(-4) M).
23 not different in young and older adults, and aminophylline did not impact the vasodilatation in eithe
24 t patches excised from RGCs, indicating that aminophylline exerts its action on retinal waves by dire
29 [proximal], 55%+/-9% [distal]; p < .05) and aminophylline (maximum relaxation, 32%+/-10% [proximal],
30 e, we investigated the protective effects of aminophylline on HDAC2 expression and glucocorticoid sen
33 L-NMMA only (protocol 1) and combined L-NMMA-aminophylline (protocol 2) at 10% (-17.5 +/- 3.7 vs. -21
34 rotocol 2, administration of combined L-NMMA-aminophylline reduced the DeltaFVC due to hypoxic exerci
35 bronchodilators (terbutaline, diltiazem, and aminophylline) relax bronchiolus to a greater degree tha
37 n the clamp region for the ligands caffeine, aminophylline, theophylline, ATP, and ryanodine but not
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