ライフサイエンスコーパス: aminopyrene

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1 with negatively charged fluorescent label 8-aminopyrene-1,2,6-trisulfonate (APTS) were separated to

2 le, mixture of oligosaccharides labeled by 8-aminopyrene-1,3,6-trisulfonate (APTS) was separated by c

3 addition, CE-MS was used to confirm major 8-aminopyrene-1,3,6-trisulfonate (APTS)-labeled glycans re

4 or resolution over the commonly used label 8-aminopyrene-1,3,6-trisulfonic acid (APTS) in both the CE

5 from these samples were then reacted with 8-aminopyrene-1,3,6-trisulfonic acid (APTS) to provide a f

6 laser-induced fluorescence (CE/LIF) using 8-aminopyrene-1,3,6-trisulfonic acid (APTS)-labeled glycan

7 en derivatized with a charged fluorophore, 8-aminopyrene-1,3,6-trisulfonic acid and further separated

8 glycans that are subsequently reacted with 8-aminopyrene-1,3,6-trisulfonic acid to add charge and a f

9 , the glycan samples were derivatized with 8-aminopyrene-1,3,6-trisulfonic acid to impart needed char

10 activity was also detected when the APTS (8-aminopyrene-1,3,6-trisulfonic acid, trisodium salt)-labe

11 ccharides labeled with a novel fluorophore 1-aminopyrene-3,6,8-trisulfonate (APTS) at the reducing te

12 een used for structure characterization of 1-aminopyrene-3,6,8-trisulfonate (APTS)-derivatized oligos

13 oresis separations of glycans labeled with 1-aminopyrene-3,6,8-trisulfonic acid were achieved with se

14 hangeable and nonexchangeable protons of the aminopyrene and the nucleic acid were assigned following

15 cts, the 1-NP-induced DNA adduct contains an aminopyrene (AP) moiety covalently linked to the C8 posi

16 synalignment at the N-(deoxyguanosin-8-yl)-2-aminopyrene ([AP]dG) adduct site positioned opposite dC

17 lix glucan, laminarin, doubly labeled with 1-aminopyrene as donor probe and fluorescein-5-isothiocyan

18 ructural studies have been undertaken on the aminopyrene-C(8)-dG ([AP]dG) adduct in the d(C5-[AP]G6-C

19 cifically placed 8-(deoxyguanosin-N(2)-yl)-1-aminopyrene (dG(1,8)), one of the DNA adducts derived fr

20 a major DNA adduct, N-(deoxyguanosin-8-yl)-1-aminopyrene (dGAP), both in vitro and in vivo.

21 form predominantly N-(deoxyguanosin-8-yl)-1-aminopyrene (dGAP).

22 ropyrene, 9(10)-hydroxy-4-nitropyrene, and 4-aminopyrene from 4-NP.

23 han the major 1-NP adduct, N-(guanin-8-yl)-1-aminopyrene (Gua-C8-AP).

24 hangeable and nonexchangeable protons of the aminopyrene moiety and the nucleic acid were assigned fo

25 tween the active site residues of Dpo4 and 1-aminopyrene moiety at the first pause site.

26 In summary, intercalation of the aminopyrene moiety is accompanied by displacement of bot

27 Dpo4.DNA-dG(1,8).dCTP ternary structure, the aminopyrene moiety of the dG(1,8) lesion, is sandwiched

28 .DNA-dG(1,8) binary structure shows that the aminopyrene moiety of the lesion stacks against the prim

29 cal electron donor-acceptor triads having an aminopyrene primary donor (APy) and a p-diaminobenzene s

30 AP]dG.dA 11-mer duplex containing the larger aminopyrene ring (reported in this study) relative to th

31 ard the major groove as a consequence of the aminopyrene ring intercalation into the helix.

32 The aminopyrene ring of [AP]dG6 is intercalated into the DNA

33 The aminopyrene ring of [AP]dG6 is intercalated into the DNA

34 agenesis induced by N-(deoxyguanosin-8-yl)-1-aminopyrene, the major DNA adduct formed by the carcinog

35 ctive metabolism leading to the formation of aminopyrene was evident only with 4-NP.

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