ライフサイエンスコーパス: ammonium chloride

1 is therefore more sensitive to inhibition by ammonium chloride.

2 red in the presence of either methylamine or ammonium chloride.

3 o acidify urine after oral administration of ammonium chloride.

4 of PI viruses to grow in cells treated with ammonium chloride.

5 d quickly by a process that was inhibited by ammonium chloride.

6 d VCP1 expression and for a few hours so did ammonium chloride.

7 taneously exposed to the autophagy inhibitor ammonium chloride.

8 aily doses of sodium citrate/citric acid and ammonium chloride.

9 cytoplasmic vacuolization in the presence of ammonium chloride.

10 when the mutant enzymes were presented with ammonium chloride.

11 cidic conditions and are highly sensitive to ammonium chloride.

12 filomycin A1 or the lysosomotropic weak base ammonium chloride.

13 ormation into vacuoles after the addition of ammonium chloride.

14 on of the purified toxin and the presence of ammonium chloride.

15 es, was abrogated by treating the cells with ammonium chloride.

16 r by treatment with either bafilomycin A1 or ammonium chloride.

17 vesicle pH and G34 cleavage were produced by ammonium chloride.

18 alian cell types, and both were inhibited by ammonium chloride.

19 The presence of ammonium chloride (30 mM), monensin (30 microM), or bafi

20 ound state to form the negative ion of ionic ammonium chloride, a species that can also be characteri

21 res (PI viruses) can grow in the presence of ammonium chloride, a weak base that blocks acid-dependen

22 uses, PI viruses can grow in the presence of ammonium chloride, a weak base that blocks acid-dependen

23 rsistent infection and in cells treated with ammonium chloride, a weak base that blocks viral disasse

24 Ammonium chloride (AC) is a weak base that blocks disass

25 By contrast, ammonium chloride affected both apical IgA and basolater

26 The K(+) channel blockers tetraethyl ammonium chloride and 4-aminopyridine (4-AP) both inhibi

27 +) efflux was insensitive to both tetraethyl ammonium chloride and 4-AP.

28 Ammonium chloride and bafilomycin A both inhibited crypt

29 The lysosomotropic agents ammonium chloride and bafilomycin A(1) prevent AAV-media

30 n is distinct from pH-raising agents such as ammonium chloride and bafilomycin A1.

31 ner similar to 2 unrelated alkalizing drugs (ammonium chloride and bafilomycin), which also inhibited

32 s of acidic cysteine proteases, chloroquine, ammonium chloride and bafilomycin, were as effective as

33 r), z-VAD-fmk (a pan-caspase inhibitor), and ammonium chloride and chloroquine (which stabilize lysos

34 heters were pretreated with tridodecylmethyl-ammonium chloride and coated with minocycline and rifamp

35 (VSV) pseudotypes was markedly diminished by ammonium chloride and concanamycin A, a selective inhibi

36 Susceptibility to inhibition by both ammonium chloride and E64 was abolished when either inhi

37 presence of the viral disassembly inhibitors ammonium chloride and E64.

38 Autophagy inhibitors bafilomycin A1, ammonium chloride, and 3-methyladenine failed to increas

39 endocytic pathway, including 0.4 M sucrose, ammonium chloride, and brefeldin A, block the acquisitio

40 oteases, carbenzoxy-tyr-ala-CHN2 (ZYA-CHN2), ammonium chloride, and chloroquine, do not block apoptos

41 A or the lysosomotropic amines methylamine, ammonium chloride, and chloroquine.

42 h lipopolysaccharide, oligodeoxynucleotides, ammonium chloride, and interleukin-8.

43 ient in response to glutamate, nitric oxide, ammonium chloride, and mercury chloride.

44 y responsive to NaCl, sodium acetate (NaAc), ammonium chloride, and sucrose; NaCl and NaAc responses

45 yocytes to small localized doses (3-5 mm) of ammonium chloride (applied in Hepes-buffered solution).

46 ne (DML), and (3-acrylamidopropyl)-trimethyl ammonium chloride (APTA).

47 Using ammonium chloride as an additive improved the instrument

48 We have found that extracellularly applied ammonium chloride as low as 5 mM causes intracellular Ca

49 -octadecyldimethyl[3-(trimethoxysilyl)propyl]ammonium chloride as one of the sol-gel precursors playe

50 tions of (13)C/(12)C glucose and (15)N/(14)N ammonium chloride, as the sole carbon and nitrogen sourc

51 ed compounds that affect endocytic pathways (ammonium chloride, bafilomycin A1, hypertonic sucrose) o

52 tic neutralization of phagolysosomal pH with ammonium chloride, bafilomycin A1, or the antimalaria dr

53 nt modifications in cells recovering from an ammonium chloride block.

54 elease is somewhat delayed by treatment with ammonium chloride, but virus yields gradually reach thos

55 -octadecyldimethyl[3-(trimethoxysilyl)propyl]ammonium chloride (C18-TMS) in the coating sol solutions

56 various amounts of benzyldimethyltetradecyl ammonium chloride cationic surfactant (BDTAC) up to four

57 Ammonium chloride caused a modest increase in inositol p

58 cted host cells with the lysosomotropic base ammonium chloride caused a swelling of all phagosomes an

59 The often-used 50 mM ammonium chloride causes more extensive and persistent c

60 At 2 years of age, an ammonium chloride challenge showed the child has incompl

61 Lysosomotropic agents (ammonium chloride, chloroquine, and amantadine), as well

62 l-4-chlorophenoxy)ethyl-dimethyl-n-octadecyl-ammonium chloride (compound 1) was found to inhibit the

63 n be inhibited by incubation of the cells in ammonium chloride, concanamycin A, leupeptin and E-64.

64 yrene) > HA > PDADMAC (poly diallyl-dimethyl-ammonium-chloride), correlating to pyrene-macromolecules

65 orthosilicate (TEOS) and hexadecyltrimethyl- ammonium chloride (CTACl) in alkaline solution.

66 rants, including adenosine-5'-monophosphate, ammonium chloride, cysteine, glycine, proline, and tauri

67 as similar to VSIV-GNJ regarding its pH- and ammonium chloride-dependent infectivity.

68 ysosomotropic agents such as chloroquine and ammonium chloride did not affect the processing of M. tu

69 since treatment of cells with the weak base ammonium chloride diminished the apoptotic response.

70 stearyl-N-methyl-N-2-(N'-arginyl) aminoethyl ammonium chloride (DSAA), a guanidinium-containing catio

71 CP1 levels were increased in the presence of ammonium chloride for most isolates.

72 radient with Bafilomycin A1, chloroquine, or ammonium chloride greatly enhanced the formation of podo

73 erences of tetraphosphonate cavitands toward ammonium chloride guests 1-4 were independently assessed

74 In the case of tetraalkyl-phosphonium/ammonium chloride guests, the host featuring the two P h

75 acrophages (MDM) treated with chloroquine or ammonium chloride had markedly increased antifungal acti

76 Ammonium chloride had no effect on [Ca2+]i in cells bath

77 pionate, raising extracellular pH, or adding ammonium chloride immediately restores FM1-43 uptake.

78 M N-dodecyl-N-methyl-3-(pyren-1-yl)propan-1-ammonium chloride in 1:9 (v:v) ethanol/water solutions w

79 using indium metal and indium(I) chloride or ammonium chloride in alcohol has been developed.

80 ocatalyst, and for a chloride source, either ammonium chloride in DMF/water mixtures or tetramethylam

81 after acid loading by transient exposure to ammonium chloride in the isolated hippocampal brain slic

82 3) M N-hexyl-N-methyl-3-(pyren-1-yl)propan-1-ammonium chloride in THF consisted of comparable amounts

83 of N-dodecyl-N-methyl-3-(pyren-1-yl)propan-1-ammonium chloride indicate that molecular packing in the

84 ion was also more sensitive to inhibition by ammonium chloride, indicating that the GNJ protein had a

85 o 7.02+/-0.02 and inhibited recovery from an ammonium chloride-induced intracellular acid load in car

86 ontrast, trypsin treatment can both overcome ammonium chloride inhibition and promote cell-cell fusio

87 n, as low-pH treatment is unable to overcome ammonium chloride inhibition, and it also cannot induce

88 Antibody neutralization, ammonium chloride inhibition, and replication levels in

89 Chloroquine and ammonium chloride insensitivity of processing of M. tube

90 -octadecyldimethyl[3-(trimethoxysilyl)propyl]ammonium chloride, into the sol solution proved to be cr

91 ng and analysis purposes, high concentration ammonium chloride is routinely used to alkalize intracel

92 3-isotridecyloxypropyl-bis(polyoxyethylene) ammonium chloride (ITDOP); mild irritants: 5% 3-decyloxy

93 Such degradation was completely inhibited by ammonium chloride (known to neutralize intra-granular pH

94 starch sedimentation but was achievable with ammonium chloride lysis.

95 Ammonium chloride-mediated lysis is the most commonly us

96 n the presence of NaCl, CsCl, and tetrabutyl ammonium chloride (NBu4Cl).

97 ions of the weak bases trimethylamine (TMA), ammonium chloride (NH4Cl) and procaine were accompanied

98 A decrease in pHi induced on washout of ammonium chloride (NH4Cl) resulted in an increase in ton

99 Sodium acetate and ammonium chloride (NH4Cl) were used to acidify and alkal

100 nfected eyes were treated with 1.0% NCT/0.1% ammonium chloride (NH4Cl), 0.1% NCT/1.0% NH4Cl, 0.1% NCT

101 butane-dione-monoxime (BDM); or 4) 50 mmol/L ammonium chloride (NH4Cl).

102 Ammonium chloride, nocodazole, and cycloheximide had no

103 The inhibitory effects of ammonium chloride on anion exchanger processing are rapi

104 for the amidation of aldehydes with economic ammonium chloride or amine hydrochloride salts has been

105 were treated with the lysosomotropic agents ammonium chloride or bafilomycin A1.

106 g chemotaxis in gradients of the attractants ammonium chloride or biotin.

107 Treatment with lysosomal inhibitors (ammonium chloride or chloroquine) or inhibitors of cathe

108 treated with either acidification inhibitor ammonium chloride or cysteine protease inhibitor E64.

109 ain T1L or T3D, and medium containing either ammonium chloride or E64d, a membrane-permeable form of

110 The administration of ammonium chloride or of sodium or potassium bicarbonate

111 ion of HeLa and CHO cells with the weak base ammonium chloride or the ionophore monensin, which eleva

112 the product aldols, even weak acids (such as ammonium chloride) or protic solvents, can induce racemi

113 acidified by 5 minutes of exposure to 20 mM ammonium chloride, pH(i) recovery was partially inhibite

114 Recovery from an ammonium chloride prepulse-induced acid load occurred mo

115 However, ammonium chloride rescued the activities of these mutant

116 anamycin A, concanamycin B, chloroquine, and ammonium chloride resulted in increases in HIV-1 infecti

117 lar phospholipid bilayers, nanoscopic films, ammonium chloride salt crystals and Montmorillonite clay

118 inding affinities for quaternary phosphonium/ammonium chloride salts to the one seen in DCM solution.

119 ilic N-alkyl-N-methyl-3-(pyren-1-yl)propan-1-ammonium chloride salts were investigated in solvents of

120 e regained the parental fusion phenotype and ammonium chloride sensitivity were shown to have also re

121 Glycine ethyl ester (GEE) and ammonium chloride served as replacements for lysine.

122 mtAQP8 knockdown cells in the presence of ammonium chloride showed a decrease in ureagenesis of ap

123 VSIV G antibodies and inhibition of entry by ammonium chloride showed that RSdeltaSH,G,F/G(vsv) infec

124 Entry was not inhibited by bafilomycin A1 or ammonium chloride, showing that passage of the virion th

125 in, but addition of the lysosomotropic agent ammonium chloride significantly increased the amount of

126 ctively, whereas soot, ammonium sulfate, and ammonium chloride simulated adsorption processes onto PM

127 t solutes (ethylene glycol, diethyl urea, or ammonium chloride) stimulates the appearance of skAE1 in

128 ins large amounts of ions, including sodium, ammonium, chloride, sulfate, and nitrate.

129 to the potassium channel blocker tetraethyl ammonium chloride (TEA).

130 Conversely, for the primary alkyl ammonium chloride, the most effective receptor is the di

131 ated airway epithelial cells were exposed to ammonium chloride to alkalinize the cytosol.

132 rus particles, propagated in the presence of ammonium chloride to block virus maturation in the low-p

133 3-(trihydroxysilyl) propyldimethyloctadecyl ammonium chloride (TPA) or silver nanoparticles (AgNP).

134 , 3-(trimethoxysily)propyl-octadecyldimethyl-ammonium chloride (TPODAC), has a reactive endgroup -Si(

135 Growth of reassortant viruses in ammonium chloride-treated cells segregated with the S1 g

136 The similarity of the ammonium chloride-treated particles and NOIPs suggests e

137 The ammonium chloride treatment did not reduce virion stabil

138 and they show relatively low sensitivity to ammonium chloride treatment in cells.

139 Ammonium chloride treatment of macrophages did not incre

140 the compartment where they accumulate due to ammonium chloride treatment, and prior to their transit

141 n cells in the presence of weak bases (e.g., ammonium chloride), VacA induces the formation of large

142 Ammonium chloride was also beneficial, suggesting that e

143 the lysosomotropic agents bafilomycin A1 and ammonium chloride, we also demonstrated that surface TNF

144 The sand was also enriched with ammonium-chloride, where ammonia-oxidation was observed

145 and infection are blocked by agents such as ammonium chloride, which act by raising the pH in the en

146 stearyl-N-methyl-N-2-(N'-arginyl) aminoethyl ammonium chloride, which can induce reactive oxygen spec

147 treatment with the lysosomotropic weak base ammonium chloride, which raises intravesicular pH.

148 , MA uptake was significantly antagonized by ammonium chloride with inhibition kinetics (IC(50) = 1.1