ライフサイエンスコーパス: amnesic

1 hbourhood, to which he moved after he became amnesic.

2 CA1 of brain histamine-depleted rats (hence, amnesic) 10 min before the retention test restored IA me

3 reciprocal manner during the anxiolytic and amnesic action of the benzodiazepine agonist.

4 nerative diseases, included 6 3 with typical amnesic Alzheimer disease and 3 with atypical variants (

5 ude of [18F]AV-1451 binding (3 patients with amnesic Alzheimer disease, r = -0.82; P < .001; r = -0.7

6 We addressed this issue by asking MTL amnesics and controls to generate exemplars of three typ

7 egy; (ii) following the organization trials, amnesics and non-amnesics retained information to a comp

8 These data suggest that at least some severe amnesics are able to acquire new semantic knowledge, pro

9 ptide is thought to contribute to the subtle amnesic changes in Alzheimer's disease (AD) by causing s

10 ry during the 13-year period since he became amnesic, despite having no measurable anterograde episod

11 The labels generated and used by the amnesics during interactions with their partners became

12 The present experiment investigated the amnesic effect of lidocaine and ECS in 25 rats trained o

13 previous learning protected animals from the amnesic effect of NMDA antagonists on new learning (of a

14 Furthermore, the amnesic effect persists for at least 24 h, cannot be att

15 These two distinct amnesic effects are abolished if protein degradation by

16 at potential for clinical practice, the fear-amnesic effects are typically demonstrated through Pavlo

17 tic effects during the post-shock period and amnesic effects during a retention test 24 h later.

18 The amnesic effects of atr/scop in the MSDB have been presum

19 campus, is a critical locus in mediating the amnesic effects of atr/scop.

20 cilitative effects that outweighed potential amnesic effects of cortical beta amyloid.

21 experiment 2, we determined the anterograde amnesic effects of large lesions of the hippocampus that

22 methamphetamine, and an insensitivity to the amnesic effects of the cholinergic receptor antagonist,

23 nd therefore support the hypothesis that the amnesic effects of this lesion are caused primarily by t

24 The results show that the amnesic effects of TS+AM+FX cannot be generally attribut

25 ures for faster onset of action, predictable amnesic effects, and relatively rapid recovery.

26 otoxic, antidepressive, anxiolytic, and anti-amnesic effects.

27 esis that STEP may be required for ethanol's amnesic effects.

28 over a 30-min delay; (iii) two subgroups of amnesics emerged, those subjects impaired in acquisition

29 In addition to the amnesic episodes, many patients describe significant int

30 e patients with otherwise typical FTD can be amnesic from presentation, or even present solely with a

31 Studies of fluency in MTL amnesics have yielded mixed results but were not designe

32 E.P. became profoundly amnesic in 1992 after viral encephalitis, which damaged

33 ive temporal lobe damage, all of whom became amnesic in a known year, were given tests of anterograde

34 trieval despite the fact that these mice are amnesic in long-term memory tests when natural recall cu

35 Patients with typical AD and non-amnesic initial presentation had a significantly higher

36 scertain since damage to this tract in human amnesics is invariably accompanied by atrophy to surroun

37 We report the case of R.S., a 49-year-old amnesic man, who we found to have acquired information a

38 ficits in multiple cognitive domains and 31% amnesic MCI.

39 Amnesic participants made more errors than nonamnesic pa

40 us in spatial learning and memory, we tested amnesic participants with hippocampal damage in a virtua

41 Amnesic participants' latencies were longer and distance

42 The amnesic participants' learning resulted in their arrivin

43 perceptual memory (priming) in a profoundly amnesic patient (E.P.), despite at-chance recognition me

44 We tested a profoundly amnesic patient (E.P.), who has virtually complete bilat

45 Amnesic patient D.A. demonstrated intact TP, and perform

46 The amnesic patient E.P. has demonstrated normal levels of r

47 r semantic (fact) learning in the profoundly amnesic patient E.P., who has extensive damage limited p

48 Although neuropsychological studies of the amnesic patient H.

49 trasted the findings for E.P. with the noted amnesic patient H.M, whose surgical lesion is strikingly

50 ted to the hippocampal formation, a severely amnesic patient with extensive medial temporal lobe dama

51 he ability of bilateral medial temporal lobe amnesic patients (MT; n=8) and normal participants (NC;

52 Amnesic patients (n = 8), who have severely impaired dec

53 Amnesic patients acquired delay conditioning at a normal

54 Amnesic patients also produced significantly more nonada

55 easures that vary with word repetition in 12 amnesic patients and 12 control subjects: (i) a late pos

56 Seven bitemporal amnesic patients and 7 controls were randomly presented

57 Amnesic patients and control participants generated exem

58 this issue, resting fMRI was performed with amnesic patients and control participants.

59 Amnesic patients and controls listened to verbal descrip

60 Amnesic patients and nondemented patients with Parkinson

61 Eight medial temporal lobe amnesic patients and their demographically matched contr

62 Amnesic patients are impaired on recall tests [1, 2], an

63 reported perceptual deficits in the group of amnesic patients at the heart of this debate.

64 ning of the transverse patterning problem by amnesic patients derives from their general impairment i

65 Controls performed well, but the amnesic patients did not acquire categorical knowledge.

66 Amnesic patients exhibited normal learning of the task b

67 Two amnesic patients failed to recover fear responses follow

68 emonstrations of intact category learning by amnesic patients for dot patterns, artificial grammars,

69 Specifically, it was widely held that amnesic patients have impaired explicit memory performan

70 Amnesic patients K.C. and R.F.R. were tested to provide

71 ruction observed in some hippocampal-damaged amnesic patients may be supported by residual function i

72 Amnesic patients often show improved performance when st

73 Experiment 1 examined the performance of 6 amnesic patients on 11 to 25 different recognition memor

74 On the tests of semantic knowledge, the amnesic patients performed markedly better than the pati

75 relations in paired-associate learning, the amnesic patients showed remarkably robust collaborative

76 of conflicting results from studies in human amnesic patients suggests that extraneous damage to extr

77 The amnesic patients tended to be impaired on the same items

78 stem, providing preliminary support from MTL amnesic patients that the default network can be fractio

79 ion is a processing mechanism that may allow amnesic patients to compensate for relational memory def

80 The amnesic patients underwent a sadness induction procedure

81 The nondeclarative memory available to amnesic patients was relatively inflexible and available

82 In the present studies, category learning in amnesic patients was tested with stimuli that both exhib

83 Amnesic patients were able to distinguish between 2 tone

84 Amnesic patients were as competent as healthy volunteers

85 o discourage the use of episodic memory, the amnesic patients were impaired and E.P. performed at cha

86 The amnesic patients were impaired at solving both the trans

87 Amnesic patients were not able to reverse the previously

88 Numerous results in the amnesic patients were predicted in advance by the single

89 In contrast, amnesic patients were unimpaired on the category decisio

90 investigated object perception in two human amnesic patients who were chosen on the basis of their l

91 nd familiarity judgement tasks compared with amnesic patients with Alzheimer's disease and healthy co

92 ings are described here for three additional amnesic patients with bilateral damage limited to the hi

93 Three amnesic patients with damage limited to the hippocampal

94 Amnesic patients with damage to the hippocampal formatio

95 Here, we addressed this topic in amnesic patients with damage to the hippocampus (HC grou

96 Most amnesic patients with damage to the medial temporal lobe

97 To resolve this discrepancy, amnesic patients with either selective hippocampal damag

98 Three amnesic patients with extensive bilateral medial tempora

99 It is unknown whether amnesic patients with hippocampal damage would exhibit s

100 ddressed in 5 experiments with E.P., 3 other amnesic patients with hippocampal damage, and 8 healthy

101 ces, we compared the performance of severely amnesic patients with large medial temporal lobe lesions

102 me intervals has sometimes been described in amnesic patients with medial temporal lobe damage.

103 Amnesic patients with medial temporal system damage show

104 Specifically, amnesic patients with MTL damage were impaired at probab

105 Amnesic patients with MTL lesions that converged on the

106 hy participants, but occurred only rarely in amnesic patients with severe damage to the hippocampus.

107 Human amnesic patients with well characterized MTL damage and

108 It was found that amnesic patients' CRs occurred significantly earlier tha

109 matomotor network connectivity was intact in amnesic patients, indicating that bilateral MTL lesions

110 with the findings from patient RB and other amnesic patients, make three important points about memo

111 ruous than for incongruous words, as did the amnesic patients, who performed less well overall.

112 nimals are consistent with the findings from amnesic patients.

113 nd medial prefrontal cortex were observed in amnesic patients.

114 dementia performed markedly better than the amnesic patients.

115 d an aspect of memory that was intact in the amnesic patients.

116 ative memory that are beyond the capacity of amnesic patients.

117 a 'Schema for the Examination of Aphasic and Amnesic Persons'.

118 45 [IQR 0.035-0.056]) than did those with an amnesic presentation (0.041 [0.031-0.057]; p=0.001).

119 We observed two distinct amnesic profiles in bvFTD patients that could reflect tw

120 Studies have investigated the nootropic and amnesic properties of both systems.

121 ciceptive effects of THC and its detrimental amnesic properties.

122 in individuals with Alzheimer disease and in amnesic rats have demonstrated that DBS targeted to the

123 BLA of brain histamine-depleted rats, hence amnesic, restored long-term memory; however, the time fr

124 ng the organization trials, amnesics and non-amnesics retained information to a comparable extent ove

125 This amnesic shadow follows a dose-response function, becomes

126 P), diarrheic shellfish poisoning (DSP), and amnesic shellfish poisoning (ASP) toxins in seafood is a

127 for the neurologic condition referred to as amnesic shellfish poisoning.

128 rent evidence suggests that after an initial amnesic stage in Alzheimer's disease, attention is the f

129 Ten chronic and stable amnesic subjects (nine with bilateral medial temporal lo

130 paired declarative memory for the tasks, the amnesic subjects demonstrated acquisition and retention

131 dings of impaired item memory in hippocampal amnesics suggest a more nuanced role for the hippocampus

132 presentation: a typical pattern with initial amnesic syndrome (n = 4 cases), progressive visual dysfu

133 ropine and scopolamine (atr/scop) produce an amnesic syndrome in humans, subhuman primates, and roden

134 eptal muscarinic agonists also alleviate the amnesic syndrome produced by systemic administration of

135 rief survey of the clinical varieties of the amnesic syndrome, transient and persistent, selected the

136 sly accessible memories, producing a classic amnesic syndrome.

137 particular, mechanisms underlying the human amnesic syndrome.

138 when active, the internal state provided by amnesic treatments is represented and integrated within

139 major findings were: (i) immediate recall in amnesics was improved by providing an organizational str

140 Amnesic windows might also occur in healthy people due t