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1 eus, and the magnocellular part of the basal amygdaloid.
2 by combining anterograde labeling of thalamo-amygdaloid afferents with postembedding immunoelectron m
3 usality methods to evaluate task-based intra-amygdaloid and extra-amygdaloid connectivity with the ne
5 pothalamic VN target-sites and also in a few amygdaloid and hypothalamic sites outside the traditiona
6 d and sustained expression was noted in most amygdaloid and hypothalamic VN target-sites and also in
7 express neither CRF-R, including the central amygdaloid and paraventricular hypothalamic nuclei, and
8 To determine whether discrete components of amygdaloid and striatal memory systems could interact to
13 affected twin had markedly smaller caudate, amygdaloid, and hippocampal volumes, and smaller cerebel
14 the caudal sublenticular region and anterior amygdaloid area (cSLR/AAA) differ from other basal foreb
16 ral cortex, hypothalamus, thalamus, cortical amygdaloid area, and white matter tracts did not show an
17 uclei (including anterior cortical, anterior amygdaloid area, periamygdaloid cortex, medial, posterio
18 of the lateral olfactory tract, the anterior amygdaloid area, the posterolateral cortical amygdaloid
20 ngulate cortex, project to medially situated amygdaloid areas such as the cortical and medial nuclei
23 is study investigated the involvement of the amygdaloid BDNF system in molecular mechanisms underlyin
25 e ELS demonstrated atypical intra- and extra-amygdaloid CeA-dominant paths with compensatory modulati
27 rch sought to determine if activation of the amygdaloid central nucleus (ACe) modulates the activity
28 rostral ventromedial medulla) and forebrain (amygdaloid central nucleus and nucleus parafascicularis
30 the amygdalo-hypothalamic projections, intra-amygdaloid circuitry and afferents from the lateral cort
32 ey limbic-related cortical/thalamic/striatal/amygdaloid circuits, and the promise of subtle modulatio
35 adult human subjects was used to investigate amygdaloid complex (AC) activity associated with the sto
37 hypothalamus, the lateral hypothalamus (LH), amygdaloid complex (AD) and thalamus (TH), and to a less
38 ily deactivated sites within the basolateral amygdaloid complex (BLA) or central amygdaloid region (C
39 esent study examined whether the basolateral amygdaloid complex (BLA) participates in the expression
41 ateral lesions restricted to the basolateral amygdaloid complex (i.e., not including the Ce) did not
42 080411 genotype by sex was found in the left amygdaloid complex (male risk allele carriers showed les
43 e) plus adjacent portions of the basolateral amygdaloid complex (with either the excitotoxin NMDA or
45 with bilateral ibotenic acid lesions of the amygdaloid complex and the hippocampus or were sham oper
46 s mediating anxiety responses, including the amygdaloid complex and the paraventricular hypothalamic
47 ta-opioid receptors is also decreased in the amygdaloid complex and ventral putamen of Alzheimer's di
48 Projection neurons of the basolateral (BL) amygdaloid complex are regulated by an intrinsic inhibit
49 n animals implicates stress hormones and the amygdaloid complex as key, interacting modulators of mem
52 hological changes in neuronal density in the amygdaloid complex have been associated with various neu
53 Single-unit activity was recorded from the amygdaloid complex in freely moving rats during an infus
54 as well as the critical contribution of the amygdaloid complex in modulation of memory by emotional
60 azabemide to MAO-B was measured in the right amygdaloid complex of 15 major depressive subjects and 1
61 ns by injecting anterograde tracers into the amygdaloid complex of Macaca fascicularis monkeys and ex
63 ine (DA) transporter and D2 receptors in the amygdaloid complex of subjects with major depression ind
64 nd D2/D3 receptors have been observed in the amygdaloid complex of subjects with major depression.
66 We conclude from these observations that the amygdaloid complex provides an excitatory input to areas
68 ing magnetic resonance imaging guidance, the amygdaloid complex was lesioned bilaterally in six rhesu
71 x (Brodmann area 11/32) bilaterally, and the amygdaloid complex were affected, but no significant atr
72 tion could be relayed from the cortex to the amygdaloid complex were investigated by using the antero
73 lesions of the hippocampal formation or the amygdaloid complex were tested on concurrent discriminat
74 l, basal, and accessory basal) nuclei of the amygdaloid complex were the source of most connections b
76 s found in limbic system areas: (1) extended amygdaloid complex, (2) lateral septum, and (3) infralim
77 PH cortices are also interconnected with the amygdaloid complex, although comparatively little is kno
79 Brain areas analyzed were caudate, putamen, amygdaloid complex, hippocampal formation and various ce
80 on of NK(1) receptor immunoreactivity in the amygdaloid complex, induction of NK(1) receptor endocyto
81 he medial temporal polar cortex, most of the amygdaloid complex, most or all of the entorhinal cortex
82 euniens and anterior medial thalamic nuclei, amygdaloid complex, piriform cortex and subfornical orga
83 which include the lateral hypothalamic area, amygdaloid complex, septal-ventral striatal areas, and i
84 the components of the basal ganglia and the amygdaloid complex, the alar and basal hypothalamic regi
86 shed afferent to the ventral striatum is the amygdaloid complex, which projects throughout the shell
97 btained for the basal ganglia and septal and amygdaloid complexes in amphibians (anamniotic tetrapods
100 aluate task-based intra-amygdaloid and extra-amygdaloid connectivity with the network underlying impl
101 onal relationships for both intra- and extra-amygdaloid connectivity, while ELS demonstrated atypical
105 ults provide evidence for the involvement of amygdaloid dopamine in regulating surprise in fear and t
106 In conclusion, these results suggest that amygdaloid dopamine transmission at D2 receptors contrib
108 olved in high-order functions in prefrontal, amygdaloid, entorhinal, and perirhinal cortex, to which
109 he surface of the telencephalic vesicles, an amygdaloid group of cells originated in the caudal pole
112 tudy examined the effects of bilateral intra-amygdaloid infusions of the D2 receptor antagonist, etic
113 investigated the effects of bilateral intra-amygdaloid infusions of the selective D1 receptor antago
115 , caudal ventral striatal areas that receive amygdaloid inputs also receive significant innervation b
119 Evidence based on cortical, thalamic, and amygdaloid inputs to the striatum suggests a medio-later
120 nterior insula, which integrates sensory and amygdaloid inputs, projects to the classic ventral stria
123 APDC, were tested on both the development of amygdaloid kindling and on fully developed stage 5 amygd
128 antagonists of the NMDA receptor block both amygdaloid LTP induction and fear conditioning, fear con
129 hypothalamic arcuate and dorsomedial nuclei, amygdaloid medial and centromedial nuclei, thalamic cent
130 uated for 0-90 min in rats receiving central amygdaloid microinjection of RX 77368 (0.1, 0.5 or 1.0 m
131 d the bed n of the stria terminalis), medial amygdaloid n; the thalamus (anteromedial thal. n; reunie
132 reverse the amphetamine-induced increase in amygdaloid neuronal activity and required more than 20 m
133 trates that a small group of arcopallial and amygdaloid neurons constitute a wide range of contralate
135 for glutamate and aspartate to determine if amygdaloid neurons projecting to different targets in th
136 ls of glutamate and aspartate in basolateral amygdaloid neurons projecting to the prefrontal cortex a
141 animals suggests that structures within the amygdaloid nuclear complex (ANC) are critical for acquir
142 nal cortex of the inferior colliculis (2.1), amygdaloid nuclei (1.5-2.1), cingulate cortex (1.8), CA1
143 sory olfactory tract, and innervates several amygdaloid nuclei (nucleus of the accessory olfactory tr
144 unts for the amygdala and each of four major amygdaloid nuclei (the lateral, basal, accessory basal,
145 rolateral cortical, and posterior basomedial amygdaloid nuclei and amygdalostriatal transition zone.
148 levation in binding to MAO-B observed in all amygdaloid nuclei in major depressive subjects as compar
150 t distinct neural systems involving separate amygdaloid nuclei mediate different types of conditioned
152 sed in the amygdala (lateral and basolateral amygdaloid nuclei) and hippocampal formation (pyramidal
153 reflect the known neurobiology of individual amygdaloid nuclei, allowing for human imaging studies to
156 lexus of Meynert, basolateral and basomedial amygdaloid nuclei, anterior pretectal and interpeduncula
157 presubiculum, parasubiculum, CA1-CA4 fields, amygdaloid nuclei, basal forebrain, thalamus, hypothalam
158 r nucleus, zona incerta, medial and cortical amygdaloid nuclei, cerebellum, nucleus of the solitary t
159 omotor cortices, the limbic system including amygdaloid nuclei, hippocampus and septum, the basal gan
160 of the stria terminalis, medial and cortical amygdaloid nuclei, preoptic area, lateral habenula, peri
161 l formation, presubiculum and parasubiculum, amygdaloid nuclei, thalamic nuclei, locus coeruleus, and
162 agonal band of Broca, the basal and cortical amygdaloid nuclei, the entopeduncular nucleus, the subth
164 brain regions connected to the BSTL are the amygdaloid nuclei, the paraventricular and ventromedial
166 ells were present in the cortical and medial amygdaloid nuclei, which are known to have strong recipr
174 tly higher in female rats, and in the medial amygdaloid nucleus (Am), GAD(67) mRNA was higher in male
179 iform cortex (Pir), posteriolateral cortical amygdaloid nucleus (PLCo), and the amygdalopiriform tran
181 area of the hypothalamus and also the medial amygdaloid nucleus and CA1 subfield of the hippocampus.
182 l in naked mole-rats include the basolateral amygdaloid nucleus and dentate gyrus, but the septohippo
183 nuclei, hippocampal CA3 region, centromedial amygdaloid nucleus and thalamic paraventricular and reun
184 mporal lobe was associated with the cortical amygdaloid nucleus and the pyramidal cells of the hippoc
187 intaining a central role for the basolateral amygdaloid nucleus in the acquisition and storage of thi
188 ing increased c-fos expression in the medial amygdaloid nucleus of both DOM and SUB males as well as
190 ampus and magnocellular cells in basolateral amygdaloid nucleus were also intensely FLNa immunoreacti
191 levels of mRNA of DA receptors in the basal amygdaloid nucleus were measured postmortem in subjects
192 olus vulgaris-leucoagglutinin in the central amygdaloid nucleus were shown to contact cSLR/AAA cholin
193 s-expression by 3.7-times in the basolateral amygdaloid nucleus, 1.2-times in the supraoptic nucleus
194 c area, several hypothalamic nuclei, central amygdaloid nucleus, amygdalohippocampal area, dorsal per
195 followed by moderate labeling of the medial amygdaloid nucleus, amygdalostriatal zone and caudoputam
196 amygdaloid area, the posterolateral cortical amygdaloid nucleus, and the dorsolateral part of the lat
197 al parvocellular part of the basal accessory amygdaloid nucleus, and the magnocellular part of the ba
198 alon: IRP-LI was concentrated in the central amygdaloid nucleus, bed nucleus of stria terminalis and
200 at in Cape mole-rats include the basolateral amygdaloid nucleus, hippocampal CA3 subfield, and dentat
201 specifically, the lateral septum and medial amygdaloid nucleus, indicate greater c-fos mRNA inductio
202 ory tubercle, nucleus accumbens, basolateral amygdaloid nucleus, rostroventrolateral medulla and nucl
203 in the lateral septal nucleus and the medial amygdaloid nucleus, which have numerous Fos-stained nucl
209 GABAergic, and project to the medial CEA (an amygdaloid output region), this may be a novel mechanism
211 brachial area (part of the spino-parabrachio-amygdaloid pain pathway) in brain slices from control ra
212 monstrated narrowly defined BLA-driven intra-amygdaloid paths and concise orbitofrontal cortex (OFC)-
214 e pathway, the spino-(trigemino)-parabrachio-amygdaloid pathway, was performed using an alpha-herpes
221 olateral amygdaloid complex (BLA) or central amygdaloid region (CeA) via GABA(A) agonist (muscimol) i
222 higher FLI than the tested groups in all the amygdaloid regions and in the hypothalamic paraventricul
223 ntal motor cortex, hippocampus, thalamus and amygdaloid) regions based on their relevance to ASD.
224 artially overlapping brain mechanisms (i.e., amygdaloid-reticular projections) mediate these effects.
226 CI, Pandey and colleagues identify a central amygdaloid signaling pathway involved in anxiety-like an
227 t is expressed in the majority of in thalamo-amygdaloid spines and that within these spines, the vari
228 he prevalence of the NR2B subunit in thalamo-amygdaloid spines provides morphological evidence suppor
231 lamic 'attack' site in the absence of medial amygdaloid stimulation suppressed predatory attack, thus
235 define a unique set of molecular markers for amygdaloid subnuclei and provide tools to genetically di
236 ields, dysfunctional pulvinar, claustrum and amygdaloid subnuclei of the amygdala, the latter progres
237 ellular and neurochemical composition of the amygdaloid subnuclei suggests their clustering into subu
238 pes of DA receptor mRNA were detected in all amygdaloid subnuclei, although D1, D2, and D4 receptor m
240 ovide support for the hypothesis that medial amygdaloid suppression of lateral hypothalamically elici
241 second limb of the pathway subserving medial amygdaloid suppression of predatory attack behavior proj
242 Drug infusion produced a blockade of medial amygdaloid suppression of predatory attack in a time- an
243 sent study tested the hypothesis that medial amygdaloid suppression of predatory attack is mediated,
244 ning, fear conditioning induces increases in amygdaloid synaptic transmission that resemble LTP, and
245 d anxiolysis was measured in adult rats, and amygdaloid tissues were used for miRNA profiling by micr
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