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1 stration of the hormone is required for bone anabolism.
2 tease inhibitors did not prevent lean tissue anabolism.
3 as predictive of the extent of intracellular anabolism.
4 ANKL-OPG axis, without interfering with bone anabolism.
5 f a greater ability to utilize nutrients for anabolism.
6 s a novel pharmacologic approach to skeletal anabolism.
7  an osteogenic environment with enhanced PTH anabolism.
8 abecular bone mass and increased PTH-induced anabolism.
9 use oncogenic mutations constitutively drive anabolism.
10 e supporting this novel approach to skeletal anabolism.
11 protein beverages designed to enhance muscle anabolism.
12 ced cancer and the window of possible muscle anabolism.
13 s fatty acid oxidation while promoting lipid anabolism.
14 nase that is obligate for insulin-stimulated anabolism.
15 may provide pharmacological targets for bone anabolism.
16 atch the stoichiometric demands of microbial anabolism.
17 sphorylation of elements thought to increase anabolism.
18 F treatment, where it precedes activation of anabolism.
19 on networks, promote catabolism and suppress anabolism.
20 ition before surgery while augmenting muscle anabolism after resection.
21 panied by different effects on hepatic lipid anabolism and blood TG profiles.
22 ltered, indicating dissociation of cartilage anabolism and breakdown.
23 and mutations in genes involved in vitamin D anabolism and catabolism might be of importance in VKH p
24                                          The anabolism and catabolism of myocardial triacylglycerol (
25 tate mRNA levels of 14 genes involved in the anabolism and catabolism of reactive oxygen species (ROS
26 ory factors and the imbalance between muscle anabolism and catabolism that develops with aging.
27 ke and utilization and direct the balance of anabolism and catabolism to ensure the appropriate use o
28 atic controls regulating the balance between anabolism and catabolism, favoring accelerated cartilage
29 c and nonphysiologic loads leading to matrix anabolism and catabolism, respectively.
30 n cells, insulin and glucocorticoids promote anabolism and catabolism, respectively.
31 e energy substrate flux to coordinate tissue anabolism and catabolism.
32 n of strong oncogenic drivers promoting cell anabolism and cell cycle progression.
33  essential reactions such as ATP production, anabolism and cellular oxidation-reduction (redox) regul
34 ll this metabolic response but can result in anabolism and continued growth in ill children.
35 measurements, and in cellulo analysis of GAG anabolism and decorin glycosylation, we mapped the organ
36 es OS in the rat VP through elevation of ROS anabolism and diminution of antioxidant detoxification.
37  metabolites, leading to suppression of cell anabolism and growth, and marked autophagy.
38 ng translation initiation and muscle protein anabolism and is the focus of ongoing research.
39  proline metabolism between yeast (cytosolic anabolism and mitochondrial catabolism) and humans (excl
40 ased adenosine triphosphate (ATP) to support anabolism and proliferation.
41  x kg(-1) x d(-1) may enhance muscle protein anabolism and provide a means of reducing the progressiv
42                           The markedly lower anabolism and related cytostatic activity of dFdC in myc
43 me Escherichia coli operons that function in anabolism and represses others involved in catabolism.
44  abundance, nutrient-sensing pathways engage anabolism and storage, whereas scarcity triggers homeost
45 he hypoxic response, lipid catabolism, lipid anabolism and the production of cell wall lipids.
46 d with anaplerotic central metabolism, lipid anabolism and the regeneration of oxidized cofactors.
47 ncrease the efficiency of EAA use for muscle anabolism and to lower the meal threshold for stimulatio
48 naling has been recognized in promoting bone anabolism, and application of strain has been shown to i
49 oxygen, with downstream actions on behavior, anabolism, and the cell cycle.
50              NMP4 functions to suppress bone anabolism, and we suggest that this occurs due to decrea
51 le approach for normalizing deranged protein anabolism associated with human diseases.
52 iofilm system, the dimensions of the zone of anabolism at the air interface ranged from 16 to 38 micr
53 s novel mechanism explains how CDK4 promotes anabolism by blocking catabolic processes (FAO) that are
54 ormonal regulation of amino acid and protein anabolism by coupling amino acid uptake and synthesis, a
55                            Further efficient anabolism by nucleotide kinases is compromised apparentl
56  plays an important role in PTH-induced bone anabolism by promoting T-cell production of Wnt10b and s
57        The brain-gut axis is the effector of anabolism by regulating growth, feeding, and metabolism
58 fection allows for increased virulence lipid anabolism by the bacterium.
59                              Drug uptake and anabolism by tumors are prerequisites of response to 5-f
60 entails reprogramming photoreceptors towards anabolism by upregulating the mechanistic target of rapa
61  is decreased in the elderly, muscle protein anabolism can nonetheless be stimulated by increased ami
62 uals is suggested to restore the lean tissue anabolism characteristic of youth.
63 dense foods differentially stimulates muscle anabolism compared with protein-dense foods.
64 utrient oversupply and triacylglycerol (TAG) anabolism contribute to hepatic steatosis, little is kno
65 rigin, our results shed light on how glucose anabolism could have emerged in early life forms.
66          We conclude that high efficiency of anabolism exemplified by this autotrophic cycle perfectl
67 ovide a stimulatory effect on muscle protein anabolism, favoring the retention of lean muscle mass wh
68 ew of eukaryotic NAD+ biosynthesis, that all anabolism flows through nicotinic acid mononucleotide, w
69  activity abolished the induction of protein anabolism genes by BMP2 treatment in vitro.
70 and how periostin expression influences bone anabolism, however, remains unknown.
71               The thesis that EPA impacts on anabolism, immune function, and clinical outcomes post-e
72  Omega-3 (n-3) fatty acids stimulate protein anabolism in animals and might therefore be useful for t
73 signaling in osteocytes is critical for full anabolism in cortical bone, but tempers bone gain in can
74 las) can improve nutrient intake and promote anabolism in critically ill infants.
75  amino acid availability limits postexercise anabolism in older individuals.
76                             Msx2 exerts bone anabolism in part by reducing Dkk1 expression and enhanc
77 hexia, but the potential for skeletal muscle anabolism in patients with advanced cancer is unproven.
78 hat aerobic exercise restores muscle protein anabolism in response to insulin by improving vasodilati
79  Our objective was to measure muscle protein anabolism in response to Leu and its metabolite HMB.
80               AAs largely determined protein anabolism in splanchnic bed by stimulating PS and decrea
81  of an actual protein-rich food to stimulate anabolism in the young and the elderly has not been expl
82                              To target lipid anabolism in tumor cells specifically, it is important t
83  metallothionein blockade on skeletal muscle anabolism in vitro and in vivo We found that concomitant
84                   Factors that affect muscle anabolism, including protein intake, also affect bone ma
85 lude that exogenous HMB induces acute muscle anabolism (increased MPS and reduced MPB) albeit perhaps
86 mal metabolic balance between catabolism and anabolism is altered.
87       Nutrient stimulation of muscle protein anabolism is blunted with aging and may contribute to th
88 t tomatidine stimulated mTORC1 signaling and anabolism, leading to accumulation of protein and mitoch
89 estrates adaptive responses, including lipid anabolism, metabolic reprogramming, increases in protein
90 r long term through microbial catabolism and anabolism, mostly due to uncoupled research on litter de
91 TORC1 senses amino acid abundance to promote anabolism, mTORC2 responds to declining glutamine catabo
92                                           In anabolism, neither clinical symptoms nor biochemical sig
93 g process in which mTORC1, the gatekeeper of anabolism, occupies a privileged regulatory position.
94 sed exposure to 5-FU and, in turn, increased anabolism of 5-FU to cytotoxic nucleotides, resulting in
95 may be feasible to measure the transport and anabolism of [18F]FU in tumors by kinetic modeling and P
96 activity of 5-fluorouracil (5-FU) and in the anabolism of its oral prodrug, capecitabine, through the
97 ction of OPP on Staphylococcus: OPP inhibits anabolism of many amino acids and highly downregulates t
98 ome P-450 systems, which allow catabolism or anabolism of unusual compounds.
99  central to an energy sensor that determines anabolism or catabolism depending on local sucrose avail
100 ration product (Col2-3/4m), as indicators of anabolism or catabolism.
101                                    Enhancing anabolism promoted neuronal survival and function and co
102 inding protein-3 as an index of nutrition or anabolism requires knowledge of its relationship to nonn
103 es the expression of genes involved in lipid anabolism, secretion, and redox metabolism, in response
104 s dispensable for BMP2 to induce the protein anabolism signature, which instead critically depends on
105 nes as mineralization related versus protein anabolism signatures of osteoblasts.
106 s and attenuated PTH-induced trabecular bone anabolism, supporting the positive function of macrophag
107 dioprotection, the balance of catabolism and anabolism, the concept of mitochondrial quality control,
108  by both IGF-1 and BMP-7 greatly potentiated anabolism through complementary and synergistic mechanis
109 ther iPTH induces Wnt10b production and bone anabolism through direct activation of the parathyroid h
110 cidate the complex role of periostin on bone anabolism, through the regulation of Sost, Wnt-beta-cate
111 entary pathways to ATF4 expression, allowing anabolism to be finely tuned to amino acid availability.
112 creases and leads to a metabolic switch from anabolism to catabolism.
113 the response of muscle protein synthesis and anabolism to insulin.
114  Inorganic pyrophosphatases are required for anabolism to take place in all living organisms.
115                                The efficient anabolism to the diglutamate analogue and the lack of de
116 bute to energy sensing and the disruption of anabolism under conditions of nutrient oversupply.
117 and parathyroid hormone (PTH)-dependent bone anabolism using murine models of targeted myeloid-lineag
118       In both nephrotic and control subjects anabolism was due to a suppression of whole-body protein
119  proteins involved in lipid and carbohydrate anabolism was increased in the presence of ethanol, a re
120          Simultaneously, both catabolism and anabolism were probed by coincubation with BODIPY-FL lab
121 GAAC pathway may facilitate insulin-mediated anabolism when exogenous amino acids are limiting.
122 e androgen receptor (AR) ligands that induce anabolism while having reduced effects in reproductive t
123        Submaximal insulin can promote muscle anabolism without eliciting a hypoglycemic response.

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