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1 atocrit were not different between 'in utero anaemic' and control adults.
2 .89, p=0.0062) decrease for infants of women anaemic at enrolment.
3 zed to a control group (n = 11) or were made anaemic by isovolumetric haemorrhage (n = 12) for 1 week
4 ria and other infections, iron-deficient and anaemic children can benefit from supplementation.
5 els responsible for delivering oxygen to the anaemic fetal heart muscle using contrast-enhanced echoc
6 ximal coronary conductance with adenosine in anaemic fetal sheep is twice that of non-anaemic fetuses
7 nal microvascular adaptations in chronically anaemic fetal sheep.
8                                         Nine anaemic fetuses (118 days gestation) underwent isovolaem
9  coronary microvascular flux rate doubled in anaemic fetuses compared to control fetuses, both at res
10 eight at delivery was 3.5 +/- 0.36 kg in the anaemic fetuses vs. 4.2 +/- 0.83 kg in controls.
11                              At 138 days the anaemic fetuses were transfused; at delivery the haemato
12                                           In anaemic fetuses, microvascular blood flow per volume of
13 icrovascular blood volume was not greater in anaemic fetuses, suggesting that growth of new microvasc
14  in anaemic fetal sheep is twice that of non-anaemic fetuses.
15 ed (mid upper arm circumference <23.5 cm) or anaemic (haemoglobin <110 g/L) at enrolment had a reduct
16                  Adult hearts that were once anaemic in utero are more resistant to hypoxic stress as
17                         The adults that were anaemic in utero weighed less than the controls 39.4 +/-
18 ductance was greater in the adults that were anaemic in utero: 11.2 +/- 4.0 ml min(-1) (100 g)(-1) mm
19 Baltimore high schools were screened for non-anaemic iron deficiency (serum ferritin < or = 12 microg
20                     98 (13.7%) girls had non-anaemic iron deficiency of whom 81 were enrolled in the
21            Of the 81 enrolled girls with non-anaemic iron deficiency, 78 (96%) completed the study (3
22 nitive function in adolescent girls with non-anaemic iron deficiency.
23              In this urban population of non-anaemic iron-deficient adolescent girls, iron supplement
24                                  Children of anaemic mothers in the MMN group scored 0.18 SD (0.06-0.
25  multi-copper ferroxidase) in the sex-linked anaemic mouse (sla) and ferroportin1 (basolateral iron e
26 ldren were oversampled from mothers who were anaemic or malnourished at SUMMIT enrolment.
27  shrinking by 20% between 2008 and 2012, and anaemic or no growth projected for 2014.
28 vely analysed in a multicentre cohort of 934 anaemic patients at 4 UK hospitals.
29 g that biopsy avoidance in Simtomax negative anaemic patients is unlikely to miss other anaemia-relat
30 is gap, by providing rapid results to target anaemic patients who require biopsies, and save costs by
31 ndings should be confirmed in iron-deficient anaemic patients.
32 month mortality (0.71, 0.60-0.86; p=0.04) in anaemic pregnant women (haemoglobin <110g/L) as compared
33 hrocytic stage growth in vitro is reduced in anaemic pregnant women at baseline, but increased during
34 efits for infants born to undernourished and anaemic pregnant women.
35 n (haemoglobin <110g/L) as compared with non-anaemic pregnant women.
36 ernourished (RR 0.85, 0.73-0.98, p=0.022) or anaemic (RR 0.71, 0.58-0.87, p=0.0010) women.
37  mortality, especially in undernourished and anaemic women.

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