ライフサイエンスコーパス: anaerobiosis

1 tochondrial NADH/NAD+ ratios associated with anaerobiosis.

2 the FNR-dependent narK operon promoter under anaerobiosis.

3 hment to surfaces, were downregulated during anaerobiosis.

4 s also required for effective acclimation to anaerobiosis.

5 tes that luminal lactate is a marker mucosal anaerobiosis.

6 itions that was particularly striking during anaerobiosis.

7 mRNA abundance) in the hydEF-1 mutant during anaerobiosis.

8 icroaerobiosis, but not during aerobiosis or anaerobiosis.

9 ignificantly enhanced survival during sudden anaerobiosis.

10 thought to mediate the cellular response to anaerobiosis.

11 that can be induced, among other things, by anaerobiosis.

12 by MvaT and positively controlled by Anr and anaerobiosis.

13 ating persistent state in response to NO and anaerobiosis.

14 issimilatory pathways required for long-term anaerobiosis.

15 l modes of pH regulation were revealed under anaerobiosis.

16 heat shock response, DNA recombination, and anaerobiosis.

17 trate- or nitrite-dependent induction during anaerobiosis.

18 by macrophages of M. tuberculosis exposed to anaerobiosis.

19 tion of the ResE phosphatase activity during anaerobiosis.

20 cells, which was completely abolished under anaerobiosis.

21 early stages of mitochondrial adaptation to anaerobiosis.

22 expression insensitive to H-NS repression in anaerobiosis.

23 romoter, does not appear to be active during anaerobiosis.

24 vity approximately three-fold in response to anaerobiosis.

25 ene is induced nearly 30-fold in response to anaerobiosis.

26 egulated by both Fnr and ArcA in response to anaerobiosis.

27 nscript levels were also induced for gpc4 by anaerobiosis.

28 d by a nutritional downshift or by transient anaerobiosis.

29 rmitted expression of increased tolerance to anaerobiosis.

30 d the dynamics of the response to short-term anaerobiosis (2 generations) in both catabolite-represse

31 tion defects, suggesting that in addition to anaerobiosis, adaptation to cell envelope stress is a cr

32 sion of citB and citZ gene expression during anaerobiosis also proved to be mediated by CcpC.

33 using the yeast model of NP-C disease during anaerobiosis, an auxotrophic condition that requires yea

34 thionyl-tRNAs with methionine in response to anaerobiosis and antibiotic exposure via the methionyl-t

35 activated by the Fnr protein in response to anaerobiosis and by the NarQ-NarP two-component regulato

36 on of these genes was found in situations of anaerobiosis and cell contact.

37 the FNR transcription factor in response to anaerobiosis and further increased in response to nitrit

38 trate reduction, is increased in response to anaerobiosis and further stimulated by the addition of n

39 Expression of these genes is induced during anaerobiosis and in some cases during cold shock.

40 der to understand the gonococcal response to anaerobiosis and its potential role in virulence.

41 ttern of dmsA-lacZ expression in response to anaerobiosis and nitrate addition was identical in both

42 phospho-NarP proteins, acting in response to anaerobiosis and nitrate or nitrite, respectively.

43 which activate transcription in response to anaerobiosis and nitrate, respectively.

44 tional response of Pseudomonas aeruginosa to anaerobiosis and nitrate.

45 vivo; aegA expression was induced 11-fold by anaerobiosis and repressed 5-fold by nitrate.

46 O/TMAO reductase is increased in response to anaerobiosis and repressed by nitrate.

47 activates expression of hbaR in response to anaerobiosis and that HbaR, in turn, activates expressio

48 nonreplication in response to starvation or anaerobiosis and that PonA3 may have a similar role.

49 responsive to intracellular pH as well as to anaerobiosis and that residues in the ligand-binding poc

50 Both anaerobiosis and the addition of catalase significantly

51 ation, (2) production of N2 and N2O requires anaerobiosis, and (3) hybrid N2 is evidence of codenitri

52 e in expression that occurred in response to anaerobiosis, and badR was responsible for a further 5-f

53 s known to be regulated by the SOS response, anaerobiosis, and catabolite repression.

54 activated by the Fnr protein in response to anaerobiosis, and it is further activated and/or repress

55 s of ethylene production such as cold, heat, anaerobiosis, and Li(+) ions enhance or suppress the exp

56 nutrient limitation (particularly iron) and anaerobiosis are major stresses experienced by V. choler

57 ulation of cupA gene expression and identify anaerobiosis as an inducer of phase-variable cupA gene e

58 od-shaped to filamentous form in response to anaerobiosis by regulating RNase E subcellular distribut

59 rease being independent of the SOS response, anaerobiosis, catabolite repression, and integration hos

60 s elevated in galactose-containing medium in anaerobiosis compared to growth in glucose, and the muta

61 omyces cerevisiae which are expressed during anaerobiosis (DAN1, DAN2, DAN3, DAN4, TIR1, TIR2, TIR3,

62 e cycles, and ecophysiology (cold hardiness, anaerobiosis, desiccation resistance) are discussed.

63 l more pH-regulated proteins; in particular, anaerobiosis enabled induction of several additional cat

64 We show that anaerobiosis enhances dimerization and activity of AphB,

65 ns are dispensable for UQ biosynthesis under anaerobiosis, even though they were expressed in the abs

66 y carbon starvation, respiratory inhibitors, anaerobiosis, freezing or boiling) lost the ability to g

67 amely exponential growth, stationary growth, anaerobiosis, heat shock, oxidative stress, nitrogen lim

68 esponse." By approximately 0.2 generation of anaerobiosis in both media, more chronic, heme-dependent

69 reductase, a major respiratory complex under anaerobiosis in Escherichia coli, is submitted to tight

70 cells in the light depleted O(2) and caused anaerobiosis in the culture, which was necessary and suf

71 olic and regulatory responses that accompany anaerobiosis in wild-type C. reinhardtii cells and a nul

72 tion of sdaA encoding serine deaminase under anaerobiosis; in addition, the glutamate decarboxylase g

73 7H10 Middlebrook medium, both acidic pH and anaerobiosis increased the uptake of the H37Rv strain fo

74 immunofluorescence microscopy, we show that anaerobiosis-induced alginate production by planktonic P

75 Increased moisture and anaerobiosis initially suppress soil C mineralization, c

76 that the bacteria experienced conditions of anaerobiosis, iron limitation, and nutrient deprivation

77 Anaerobiosis is a stress condition for aerobic organisms

78 h and that adaptation to nutrient stress and anaerobiosis is crucial for H. ducreyi survival in human

79 pIV-dependent induction of psp expression in anaerobiosis is independent of PspBC, establishing that

80 on of cadC by AphB in response to low pH and anaerobiosis is mirrored in the heterologous organism Es

81 Moreover, the results indicate that mere anaerobiosis is not sufficient to maintain hydrogenase g

82 The molecular basis of obligate anaerobiosis is not well established.

83 e (appY) implicated in E. coli's response to anaerobiosis, is repressed by DpiA.

84 nd NsrR in transcriptional regulation during anaerobiosis of B. subtilis.

85 the observed effect of the pgk-1 mutation on anaerobiosis of resE mutant cells.

86 y, since cells remain sensitive to Mn during anaerobiosis or when hydrogen peroxide biogenesis is sig

87 er oxidation by PSII, (ii) a faster onset of anaerobiosis preserves PSII from irreversible photoinhib

88 E. coli cultures because of reports that in anaerobiosis, pyruvate kinase represents the major route

89 ars to involve a regulatory cascade in which anaerobiosis, signaled through Anr, stimulates expressio

90 ls to oxidation lies at the root of obligate anaerobiosis, spontaneous mutagenesis, and the use of ox

91 ble to nitrogen fixation (limiting nitrogen, anaerobiosis, temperatures of 30 degrees C).

92 In anaerobiosis, the green alga Chlamydomonas reinhardtii e

93 incubation conditions, such as the length of anaerobiosis, the readdition of O2, the presence of acet

94 Under anaerobiosis, the reduction of PchF[Y384F]FAD by substra

95 her, these data provide evidence that during anaerobiosis, the Rpd3 complex acts at the DAN1 promoter

96 n a seconds time scale, of haem a3 following anaerobiosis, there was no indication of accumulation of

97 ox state resembling either reduced oxygen or anaerobiosis, thereby resulting in increased expression

98 o the regulatory region of genes involved in anaerobiosis to activate their transcription.

99 lly and that this process may reverse during anaerobiosis to produce proton motive force.

100 d acid (pH 5 to 6) restored acid resistance; anaerobiosis was not required, as it is for acid resista

101 Also induced at high pH, but only in anaerobiosis, was glutamate decarboxylase (GadA).

102 e reductions in activity under conditions of anaerobiosis were found to be primarily the result of re

103 ponsive to UPC2 that were also induced under anaerobiosis, when sterol uptake is essential.