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1 e estimates, dose response curve regression, and comparison analyses were performed.
2 Univariate, multivariate, and time-series analyses were performed.
3 Multiple sensitivity analyses were performed.
4 rd ratios (HRs) and receiver operating characteristic curve analyses were performed.
5 Lung and brain histological analyses were performed.
6 e chronically fed EtOH, and ileum RNA-seq and bioinformatic analyses were performed.
7 Prespecified subgroup analyses were performed.
8 Adjusted logistic regression models and meta-analyses were performed.
9 lcin [OCN], and tartrate-resistant acid phosphatase [TRAP]) analyses were performed.
12 The main limitations of this study are that most analyses were performed at the national rather than individua
24 standing this high mortality, detailed clinical virological analyses were performed in specimens from 180 H5N1 patients,
29 Additional vital status data collection and subsequent analyses were performed post hoc.Measurements and Main Result
30 ty sensitivity analyses and 1-way deterministic sensitivity analyses were performed to assess for uncertainty.
33 erating characteristic and net reclassification improvement analyses were performed to compare LUCK and Killip classifica
38 vival (Cox regression and Kaplan-Meier), and center effects analyses were performed to examine the association of ECLS us
40 Univariable and multivariable logistic regression analyses were performed to identify parameters that were asso
45 and quantitative polymerase chain reaction and Western blot analyses, were performed to assess the potency and to charact
47 ined using Illumina HiSeq 2500, and differential expression analyses were performed using DESeq2 (|fold change|>1.5 and f