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1 A antibodies in both mice and humans may be anamnestic.
2 .64), which suggests that responses were not anamnestic.
5 we assessed the magnitude of the primary and anamnestic Ag-specific IgG responses of mice to four cli
6 at much lower viral loads and coincided with anamnestic anti-hepatitis B surface (HBs) responses and
8 1, MSP-2, and MSP-3 that are needed to evoke anamnestic antibody and effector T-cell responses elicit
9 infection with virulent chlamydiae promotes anamnestic antibody and T cell responses that protect th
10 2 were impaired in their ability to mount an anamnestic antibody response and were more susceptible t
11 Our data suggest that the cross-reactive anamnestic antibody response has a protective capacity d
15 ses in naive hosts, ICC-1132 elicited potent anamnestic antibody responses in mice primed with P. fal
17 owever, neither route of vaccination induced anamnestic antibody responses to the surface antigens of
20 ortant immunologic questions include whether anamnestic CD4 T cell responses drive disease rather tha
21 llowing the CL-13 boost, a greater number of anamnestic CD8 T cells localized to the lymph nodes, exh
24 6a) did not develop viremia but did show an anamnestic cell-mediated immune response after rechallen
27 d macaques exhibited a rapid and substantial anamnestic CTL response specific for the p11C, C-M Gag e
28 ociated with the evolution of high-frequency anamnestic CTL responses specific for a dominant Mamu-A*
32 nths, and re-exposure to antigen resulted in anamnestic effector and central memory CD8(+) T-lymphocy
33 e boost vaccination minimizes contraction of anamnestic effectors and maximizes memory CD8+ T cell qu
34 er, prior immunization capable of priming an anamnestic Env antibody response did not accelerate V1/V
35 t in protective immunity was associated with anamnestic Env-specific cellular immunity that developed
36 wever, 13 blips of virus in six macaques and anamnestic Env-specific rectal IgA responses in three of
37 lls are required for protection, there is no anamnestic expansion of SIV-specific CD8(+) T cells in a
39 able ASC responses to primary infection, and anamnestic fecal IgA responses likely contributed to sel
40 ng of the immune system to induce protective anamnestic heterotypic neutralizing antibody responses u
41 nonvaccinated acutely infected individuals, anamnestic HIV-1-specific B- and T-cell responses appear
43 -VLPs (with or without mLT) developed higher anamnestic IgA and IgG ASC responses in ileum after chal
45 ugate-vaccinated and control G2 mice induced anamnestic IgG- and VH3-positive responses to GXM and wa
46 valent vaccine (HR-Tet) showed a tetravalent anamnestic immune response in 100% (16/16) of AGMs after
49 erosol challenge model, and that primary and anamnestic immune responses to toxin proteins provide pr
52 ivotal role for IL-33 in driving primary and anamnestic immunity against the rodent hookworm Nippostr
57 is a promising vector to use to prime for an anamnestic neutralizing antibody response following infe
59 human immunodeficiency virus (SHIV89.6P), an anamnestic neutralizing antibody response was observed,
62 xhibited functional exhaustion and decreased anamnestic potential following secondary antigen challen
63 IMV to induce affinity maturation results in anamnestic production of nonprotective, complement-fixin
64 ation because it takes advantage of both the anamnestic properties of an adaptive immune response and
65 or T-cell-deficient mice lacked the complete anamnestic protection observed in immunocompetent mice.
66 se to current antigens but instead suggested anamnestic recall of antibody to earlier influenza virus
68 xisting FIX inhibitors exhibited a transient anamnestic response (5 Bethesda units) at 2 weeks after
72 n, and levels of both increased in a typical anamnestic response following a challenge infection.
79 peak anti-Nef titers, a more rapid anti-Nef anamnestic response postchallenge, and expanded CD8(CM)
83 However, such patients are at risk for an anamnestic response resulting from a proinflammatory res
85 fter the initial immunization resulted in an anamnestic response to C-GTF resulting in 10- and 100-fo
88 ing immunization with NYVAC/SIV(gpe) and the anamnestic response to SIV(mac251) at 48 h after challen
89 n there is an association between a systemic anamnestic response to toxin A, as evidenced by increase
90 d an immune reaction compatible with a local anamnestic response upon contact with the replicating FM
91 after primary CHIKV immunization and then an anamnestic response upon subsequent VEEV vaccination.
96 Following challenge infection, the dominant anamnestic response was solely in the B-cell compartment
98 or T cells, memory is often equated with the anamnestic response, the ability of secondary lymphoid t
106 survival after secondary challenge and rapid anamnestic responses directed against toxin proteins.
108 e, concentrations were extremely low with no anamnestic responses in mice with secondary infection.
116 oire of cancers depends, on one hand, on the anamnestic retention of their ontogenesis and, on the ot
121 peptides as immunogens to prime for strong, anamnestic, strain-cross-reactive type 1 immune response
122 of these antigen delivery systems to induce anamnestic systemic and secretory responses to the clone
123 a chlamydial component drives a deleterious anamnestic T cell response upon oviduct reinfection.
126 emia during persistence could result from an anamnestic Th lymphocyte response to conserved regions o
129 eropositive individuals may have resulted in anamnestic, vector-specific CD4(+) T lymphocytes that co
130 ion rate), and all groups showed significant anamnestic virus-specific IgG and IgA ASC responses.
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