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1  A antibodies in both mice and humans may be anamnestic.
2 .64), which suggests that responses were not anamnestic.
3 s were functional and able to mount a robust anamnestic Ab response upon revaccination.
4  latter stages of the GC response and in the anamnestic AFC response.
5 we assessed the magnitude of the primary and anamnestic Ag-specific IgG responses of mice to four cli
6 at much lower viral loads and coincided with anamnestic anti-hepatitis B surface (HBs) responses and
7                                              Anamnestic anti-SEB serum immunoglobulin G (IgG) respons
8 1, MSP-2, and MSP-3 that are needed to evoke anamnestic antibody and effector T-cell responses elicit
9  infection with virulent chlamydiae promotes anamnestic antibody and T cell responses that protect th
10 2 were impaired in their ability to mount an anamnestic antibody response and were more susceptible t
11     Our data suggest that the cross-reactive anamnestic antibody response has a protective capacity d
12                                           An anamnestic antibody response was also detected.
13 he absence of viraemia and the absence of an anamnestic antibody response.
14  undetectable viral loads and the lack of an anamnestic antibody response.
15 ses in naive hosts, ICC-1132 elicited potent anamnestic antibody responses in mice primed with P. fal
16 s in the peripheral blood and the absence of anamnestic antibody responses postchallenge.
17 owever, neither route of vaccination induced anamnestic antibody responses to the surface antigens of
18                Secondary patients had robust anamnestic antibody responses.
19                                              Anamnestic binding and neutralizing antibody responses w
20 ortant immunologic questions include whether anamnestic CD4 T cell responses drive disease rather tha
21 llowing the CL-13 boost, a greater number of anamnestic CD8 T cells localized to the lymph nodes, exh
22                                              Anamnestic CD8(+) T cell responses were further enhanced
23 s known regarding how AhR activation affects anamnestic CD8+ T cell responses.
24  6a) did not develop viremia but did show an anamnestic cell-mediated immune response after rechallen
25                                Massive early anamnestic cellular immune responses controlled acute-ph
26                                     However, anamnestic cellular immune responses developed during th
27 d macaques exhibited a rapid and substantial anamnestic CTL response specific for the p11C, C-M Gag e
28 ociated with the evolution of high-frequency anamnestic CTL responses specific for a dominant Mamu-A*
29 from PPD Ag-challenged lungs conferred a Th1 anamnestic cytokine response in recipients.
30 , suggests that infection induces suboptimal anamnestic defenses.
31  all four of the immunized animals developed anamnestic E-SPOT responses after challenge.
32 nths, and re-exposure to antigen resulted in anamnestic effector and central memory CD8(+) T-lymphocy
33 e boost vaccination minimizes contraction of anamnestic effectors and maximizes memory CD8+ T cell qu
34 er, prior immunization capable of priming an anamnestic Env antibody response did not accelerate V1/V
35 t in protective immunity was associated with anamnestic Env-specific cellular immunity that developed
36 wever, 13 blips of virus in six macaques and anamnestic Env-specific rectal IgA responses in three of
37 lls are required for protection, there is no anamnestic expansion of SIV-specific CD8(+) T cells in a
38 tibodies against all vaccine genotypes in an anamnestic fashion.
39 able ASC responses to primary infection, and anamnestic fecal IgA responses likely contributed to sel
40 ng of the immune system to induce protective anamnestic heterotypic neutralizing antibody responses u
41  nonvaccinated acutely infected individuals, anamnestic HIV-1-specific B- and T-cell responses appear
42                             After challenge, anamnestic IgA and IgG ASC and memory B-cell responses w
43 -VLPs (with or without mLT) developed higher anamnestic IgA and IgG ASC responses in ileum after chal
44                                  We found an anamnestic IgG response in one pretransfusion seropositi
45 ugate-vaccinated and control G2 mice induced anamnestic IgG- and VH3-positive responses to GXM and wa
46 valent vaccine (HR-Tet) showed a tetravalent anamnestic immune response in 100% (16/16) of AGMs after
47 ers of Clostridium difficile have a systemic anamnestic immune response to toxin A.
48                                 The observed anamnestic immune response was characterized by (i) more
49 erosol challenge model, and that primary and anamnestic immune responses to toxin proteins provide pr
50                                              Anamnestic immune responses were demonstrated for HI, ne
51 ine adjuvants for eliciting both primary and anamnestic immune responses.
52 ivotal role for IL-33 in driving primary and anamnestic immunity against the rodent hookworm Nippostr
53  cleared from the lower respiratory tract by anamnestic immunity.
54      One of these two monkeys also showed no anamnestic increases in antibody levels following challe
55                   By corroborating clinical, anamnestic, laboratory and imaging data, the case was di
56                                        Thus, anamnestic memory T cell differentiation is flexible, an
57 is a promising vector to use to prime for an anamnestic neutralizing antibody response following infe
58                                         This anamnestic neutralizing antibody response was also detec
59 human immunodeficiency virus (SHIV89.6P), an anamnestic neutralizing antibody response was observed,
60                                     Although anamnestic neutralizing antibody responses against labor
61  vaccination elicited comparable primary and anamnestic neutralizing antibody responses.
62 xhibited functional exhaustion and decreased anamnestic potential following secondary antigen challen
63 IMV to induce affinity maturation results in anamnestic production of nonprotective, complement-fixin
64 ation because it takes advantage of both the anamnestic properties of an adaptive immune response and
65 or T-cell-deficient mice lacked the complete anamnestic protection observed in immunocompetent mice.
66 se to current antigens but instead suggested anamnestic recall of antibody to earlier influenza virus
67        A fourth dog exhibited a strong early anamnestic response (216 BU), with slow decline to 0.8 B
68 xisting FIX inhibitors exhibited a transient anamnestic response (5 Bethesda units) at 2 weeks after
69 vage and a stronger rectal anti-envelope IgA anamnestic response 2 weeks postchallenge.
70                            Development of an anamnestic response after vaccination would suggest prev
71 with recombinant virus, animals exhibited an anamnestic response against SNV.
72 n, and levels of both increased in a typical anamnestic response following a challenge infection.
73 acaques exhibited a CD4(+) and CD8(+) T cell anamnestic response following booster immunization.
74                                          The anamnestic response in immunized cattle was specific for
75 y responses following challenge indicated an anamnestic response in the vaccinated monkeys.
76 V, both CD4 Th1 and Th2 memory cells made an anamnestic response in vivo.
77                                          The anamnestic response is the property of the immune system
78       In experiments designed to compare the anamnestic response of susceptible and resistant mouse s
79  peak anti-Nef titers, a more rapid anti-Nef anamnestic response postchallenge, and expanded CD8(CM)
80           After hepatitis B vaccination, the anamnestic response rate in HIV-1-positive subjects who
81                                  The overall anamnestic response rate was 16% and was not significant
82                                          The anamnestic response rate was higher in subjects who test
83    However, such patients are at risk for an anamnestic response resulting from a proinflammatory res
84        They also developed a potent and fast anamnestic response to a subsequent parenteral boost wit
85 fter the initial immunization resulted in an anamnestic response to C-GTF resulting in 10- and 100-fo
86                 This may have represented an anamnestic response to HCV antigens translated directly
87 r SIV challenge, apparently the result of an anamnestic response to SIV antigens.
88 ing immunization with NYVAC/SIV(gpe) and the anamnestic response to SIV(mac251) at 48 h after challen
89 n there is an association between a systemic anamnestic response to toxin A, as evidenced by increase
90 d an immune reaction compatible with a local anamnestic response upon contact with the replicating FM
91 after primary CHIKV immunization and then an anamnestic response upon subsequent VEEV vaccination.
92                                           An anamnestic response was defined as an anti-HBs titer of
93                                           An anamnestic response was observed in all monkeys after th
94                                    A classic anamnestic response was observed in patients dosed with
95                                     A strong anamnestic response was observed when convalescent baboo
96  Following challenge infection, the dominant anamnestic response was solely in the B-cell compartment
97 imals and showed little or no evidence of an anamnestic response when measured with SIVsmE660.
98 or T cells, memory is often equated with the anamnestic response, the ability of secondary lymphoid t
99 but lacking detectable antibody abrogates an anamnestic response.
100 patients and provide treatment to prevent an anamnestic response.
101 ted bacterial clearance, and orchestrated an anamnestic response.
102 tive persons marked the capacity to mount an anamnestic response.
103 y levels in serum, suggesting the lack of an anamnestic response.
104  of the serotype-cross-reactive "recall" or "anamnestic" response.
105 zyme-linked immunosorbent assay demonstrated anamnestic responses after each boost.
106 survival after secondary challenge and rapid anamnestic responses directed against toxin proteins.
107                                              Anamnestic responses in front-line tissues are thus an i
108 e, concentrations were extremely low with no anamnestic responses in mice with secondary infection.
109 trachoma vaccine and their peripheral T cell anamnestic responses studied.
110 rise to long-lived memory T cells capable of anamnestic responses to antigenic rechallenge.
111 nd IgG antibodies were induced in serum with anamnestic responses to secondary infection.
112                                              Anamnestic responses were observed in preimmune individu
113 unoglobulin G responses, and near absence of anamnestic responses.
114 nerated long-lived memory T cells capable of anamnestic responses.
115 ion with nonconjugated Pfs25H induced strong anamnestic responses.
116 oire of cancers depends, on one hand, on the anamnestic retention of their ontogenesis and, on the ot
117                By use of a murine model, the anamnestic serologic response was characterized followin
118 el, which is likely to be a reflection of an anamnestic serum IgG response.
119                             In contrast, the anamnestic SIV Gag-specific CD4(+) T cell response in BA
120                                 However, the anamnestic, SIV Gag-specific CD8(+) T cell response to S
121  peptides as immunogens to prime for strong, anamnestic, strain-cross-reactive type 1 immune response
122  of these antigen delivery systems to induce anamnestic systemic and secretory responses to the clone
123  a chlamydial component drives a deleterious anamnestic T cell response upon oviduct reinfection.
124                                              Anamnestic T cell responses to Ag85A/b were not detected
125 s in the vaccine phase, and occurred without anamnestic T-cell responses.
126 emia during persistence could result from an anamnestic Th lymphocyte response to conserved regions o
127                                              Anamnestic uncertainty, found in 88 families (8.5%), if
128  mutation rate; and evaluation of cases with anamnestic uncertainty.
129 eropositive individuals may have resulted in anamnestic, vector-specific CD4(+) T lymphocytes that co
130 ion rate), and all groups showed significant anamnestic virus-specific IgG and IgA ASC responses.

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