ライフサイエンスコーパス: anaphylactic reaction

1 ent was also observed in an active cutaneous anaphylactic reaction.

2 95.4%) and in all patients with a history of anaphylactic reaction.

3 symptoms ranging from oral pruritus to fatal anaphylactic reaction.

4 7.7% (95% CI, 5.7% to 9.7%) reported a prior anaphylactic reaction.

5 cted to oral challenge, which resulted in an anaphylactic reaction.

6 ng signals can dominate to initiate a severe anaphylactic reaction.

7 ths, and no patients experienced allergic or anaphylactic reactions.

8 ere confirmed as allergic, including 11 with anaphylactic reactions.

9 the VE IL-4Ralpha and ABL1 pathway in severe anaphylactic reactions.

10 posed to contain milk proteins, which caused anaphylactic reactions.

11 portantly, CpG/PN-NP treatment did not cause anaphylactic reactions.

12 can range from mild local symptoms to severe anaphylactic reactions.

13 rotective immune responses in the absence of anaphylactic reactions.

14 re were 507 uniphasic and 25 (4.5%) biphasic anaphylactic reactions.

15 patients to prevent further life-threatening anaphylactic reactions.

16 ocument a role for cofactors in about 30% of anaphylactic reactions.

17 Symptoms range from mild to anaphylactic reactions.

18 , thereby promoting allergic and potentially anaphylactic reactions.

19 ere, including one moderate reaction and two anaphylactic reactions.

20 developed significantly attenuated cutaneous anaphylactic reactions.

21 allergenic food capable of provoking severe anaphylactic reactions.

22 allergenic foods, which can result in severe anaphylactic reactions.

23 eous tissue, are less likely to give rise to anaphylactic reactions.

24 mice represents a new animal model to study anaphylactic reactions.

25 the most common cause of fatal food-induced anaphylactic reactions.

26 lation of SNAP-23 leads to degranulation and anaphylactic reactions.

27 on of IgG1 Abs and to the risk of triggering anaphylactic reactions.

28 ete Freund's adjuvant has resulted in severe anaphylactic reactions.

29 life of the rMETase apoenzyme and eliminated anaphylactic reactions.

30 diators that cause allergic inflammation and anaphylactic reactions.

31 4 toxicities included acne-like rash (6.1%), anaphylactic reactions (1.5%), and diarrhea (1.5%).

32 ive patients with a convincing history of an anaphylactic reaction after a hymenoptera sting were tes

33 uced anaphylaxis (WDEIA) is characterized by anaphylactic reactions after wheat ingestion and physica

34 ble involvement of augmenting factors; after anaphylactic reactions, always ask for possible augmenta

35 OcAn, both confirmation of the diagnosis of anaphylactic reaction and identification of the trigger

36 ced a serious infusion reaction (one grade 4 anaphylactic reaction and one grade 3 stridor) during th

37 ing patients after complete resolution of an anaphylactic reaction and to dispense with prolonged mon

38 We suspected an anaphylactic reaction and treated him with intramuscular

39 o beta-lactams are the most common causes of anaphylactic reactions and can be life-threatening.

40 pture, as in case of misdiagnosis, may cause anaphylactic reactions and dissemination.

41 ated (four cases of haematoma expansion, one anaphylactic reaction, and one ischaemic stroke) and two

42 p between serum basal tryptase (sBT) levels, anaphylactic reactions, and clonal mast cell diseases wa

43 Elicitors of anaphylactic reactions are any sources of protein with a

44 Severe IgE-mediated, food-induced anaphylactic reactions are characterized by pulmonary ve

45 ate that the vast majority of food-triggered anaphylactic reactions are not life-threatening.

46 Although anaphylactic reactions are potentially life-threatening,

47 Two patients in the reslizumab group had anaphylactic reactions; both responded to standard treat

48 city while reducing their potential to cause anaphylactic reactions by essentially eliminating IgE-me

49 ) disease that can lead to potentially fatal anaphylactic reactions caused by excessive MC mediator r

50 tide-induced EAE models led to a rapid-onset anaphylactic reaction characterized by respiratory distr

51 Systemic anaphylactic reactions did not occur.

52 An anaphylactic reaction due to a Hymenoptera sting is a cl

53 he scientific evidence on self-medication of anaphylactic reactions due to Hymenoptera stings, to inf

54 The underlying mechanisms of anaphylactic reactions due to occupational exposure are

55 Biphasic anaphylactic reactions, especially clinically important

56 The effects on anaphylactic reactions following PN challenge and the as

57 We identified 213 anaphylactic reactions in 192 children (97 male patients

58 teria monocytogenes (HKLM) as an adjuvant on anaphylactic reactions in a mouse model of PN allergy.

59 The prevalence and incidence of anaphylactic reactions in Germany are unknown.

60 IIA may therefore contribute to allergic and anaphylactic reactions in humans.

61 xtravasation in the severity of IgE-mediated anaphylactic reactions in mice.

62 ast to non-PEGylated rMETase, which elicited anaphylactic reactions in monkeys.

63 antibody production in mice and monkeys and anaphylactic reactions in monkeys.

64 inhibitors have been associated with severe anaphylactic reactions in patients with hymenoptera veno

65 s of pathogenic antibody or life-threatening anaphylactic reactions in protein replacement therapy fo

66 c response to infused VWF concentrates up to anaphylactic reactions in rare cases.

67 early-phase and severely blunted late-phase anaphylactic reactions in response to antigen challenge

68 carrying btk mutations exhibited diminished anaphylactic reactions in response to IgE and antigen.

69 ontrast media, the major cause of iatrogenic anaphylactic reactions in the hospital, is explored.

70 s referred to our hospital owing to repeated anaphylactic reactions induced by exercise after meals.

71 Monitoring after complete resolution of anaphylactic reactions is recommended.

72 decrease in body temperature, reflecting the anaphylactic reaction, is substantially enhanced by the

73 with pathological conditions or allergic and anaphylactic reactions, it may contribute beneficially t

74 She experienced anaphylactic reactions just after the inhalation of Inav

75 Anaphylactic reactions may occur among 1%-2% of botulinu

76 sis lesions, and clinical characteristics of anaphylactic reaction might be useful for differential d

77 ema (n=69, 20.9%), asthma (n=65, 19.7%), and anaphylactic reaction (n=19, 5.8%).

78 (NARA), total number of reports on suspected anaphylactic reactions, number of reactions where NMBAs

79 Anaphylactic reactions occurred in 6 of the 149 treated

80 Grade 3 severe anaphylactic reactions occurred in seven cases of nine.

81 oducing Treg cells, without causing allergic/anaphylactic reactions or generalized immunosuppression.

82 We performed a review of PED records for anaphylactic reactions over 5 years.

83 eaction tryptase level) detected most of the anaphylactic reactions, particularly if baseline levels

84 This anaphylactic reaction required a human CD32a transgene b

85 ltuximab (lower respiratory tract infection, anaphylactic reaction, sepsis).

86 Moreover, this treatment eliminated fatal anaphylactic reactions that occurred after four to six e

87 phasic - and clinically important biphasic - anaphylactic reactions, the number of transfers to inten

88 ll deficient and protected from IgE-mediated anaphylactic reactions, their dramatically different res

89 on of pork kidney proteins mediating delayed anaphylactic reactions through specific IgE to alpha-Gal

90 E-dependent effector mechanisms, and a local anaphylactic reaction to an unrelated antigen can enhanc

91 Although anaphylactic reactions to blood products are rare, the i

92 nd distress commonly feature in survivors to anaphylactic reactions to drug.

93 ) is the major cause of fatal and near-fatal anaphylactic reactions to foods.

94 d to treat the few patients who present with anaphylactic reactions to Hymenoptera stings, as well as

95 Anaphylactic reactions to immunoglobulin infusions in im

96 Among the most frequent anaphylactic reactions to insects are those attributed t

97 Anaphylactic reactions to neuromuscular blocking agents

98 as to evaluate mortality rate in France from anaphylactic reactions to NMBAs, to identify risk factor

99 tructure and described as major elicitors of anaphylactic reactions to peanut (allergens Ara h 2 and

100 eggs, or occurrence of Sampson Grade 3 to 5 anaphylactic reactions upon egg ingestion.

101 with the ingestion of only Citrus unshiu, an anaphylactic reaction was induced by additional acetyl-s

102 nses compared to free allergen in mice while anaphylactic reactions were essentially abolished.

103 protein injection of immunized mice induced anaphylactic reactions, which were more severe in multip

104 Such an anaphylactic reaction would also limit potential therape