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1 e only detected in poorly differentiated and anaplastic carcinomas.
2 lowing loss of p53 function, may progress to anaplastic carcinomas.
3 PTCs), 12 were follicular carcinomas, 9 were anaplastic carcinomas, 5 were Hurthle cell carcinomas, 2
4 was associated with aggressive medullary and anaplastic carcinomas, and its expression pattern in med
5  thyroid cancer to poorly differentiated and anaplastic carcinomas, both in terms of the presence of
6  of 2 papillary carcinoma cell lines, 4 of 5 anaplastic carcinoma cell lines, 1 of 2 follicular carci
7 nt thyroid tumor cell lines, such as thyroid anaplastic carcinoma cells (ARO cells), while either age
8 choalveolar cell carcinoma) or Calu 6 (human anaplastic carcinoma) NSCLC cells.
9  in pulmonary adenocarcinomas and large-cell anaplastic carcinomas (P=0.006).
10  (one PTC, one follicular carcinoma, and one anaplastic carcinoma) showed evidence of hemizygous dele
11 nd 11q13, and loss of 5q11-31 were unique to anaplastic carcinomas, suggesting they may be markers fo
12 cell cancer of the right lung (microcellular anaplastic carcinoma), was admitted with focal neurologi

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