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1 d responsiveness to therapy in patients with anaplastic oligodendroglioma.
2 ensitivity in some tumor types, particularly anaplastic oligodendroglioma.
3 d 19q loss, which is observed in over 50% of anaplastic oligodendrogliomas.
4 a set of 50 gliomas, 28 glioblastomas and 22 anaplastic oligodendrogliomas.
5 s a genetic predictor of chemosensitivity in anaplastic oligodendrogliomas.
6 e therefore analyzed these three genes in 72 anaplastic oligodendrogliomas.
7 with progression from well-differentiated to anaplastic oligodendrogliomas.
8 nd 11 of these cases were among the 24 (42%) anaplastic oligodendrogliomas.
9 patients had glioblastoma multiforme, 2 had anaplastic oligodendroglioma, 1 had anaplastic ependymom
10 a, malignant glioma, anaplastic astrocytoma, anaplastic oligodendroglioma, anaplastic oligoastrocytom
11 in these specific domains were identified in anaplastic oligodendrogliomas, anaplastic astrocytomas,
12 ade (oligodendroglioma), intermediate-grade (anaplastic oligodendroglioma and anaplastic astrocytoma)
13 ocus is often deleted in high-grade gliomas (anaplastic oligodendroglioma and glioblastoma), we inves
14 de glioma were included: 10 glioblastomas, 1 anaplastic oligodendroglioma, and 1 anaplastic astrocyto
15 ing adult and paediatric glioblastoma (GBM), anaplastic oligodendroglioma, and diffuse intrinsic pont
16 rentiated oligodendrogliomas, in 18/23 (83%) anaplastic oligodendrogliomas, and in 3/8 (38%) oligoast
19 lasms [anaplastic oligoastrocytoma (AOA) and anaplastic oligodendroglioma (AO)] remain subjective, an
20 n = 33; anaplastic astrocytoma [AA], n = 6; anaplastic oligodendroglioma [AO], n = 2; gliosarcoma [G
23 in our mouse model, a decreased incidence of anaplastic oligodendroglioma as well as prolonged surviv
24 ns 15 exons, including an exon defined by an anaplastic oligodendroglioma expressed sequence tag, and
25 as, histologically classic glioblastomas and anaplastic oligodendrogliomas follow markedly different
26 a, oligoastrocytoma, anaplastic astrocytoma, anaplastic oligodendroglioma, glioblastoma multiforme, g
27 inverse phenomenon was found with grade III anaplastic oligodendrogliomas, in which stronger EGFR ex
29 al response, had his diagnosis changed to an anaplastic oligodendroglioma on subsequent central neuro
31 gliomas, 19%; anaplastic astrocytomas, 11%; anaplastic oligodendrogliomas or oligoastrocytomas, 25%;
32 therapeutic response and overall survival in anaplastic oligodendroglioma patients treated with proca
35 rapy Oncology Group (RTOG) clinical trial in anaplastic oligodendroglioma suggested a progression-fre
36 or cell lines DAOY (medulloblastoma), SWB61 (anaplastic oligodendroglioma), SWB40 (anaplastic astrocy
38 with nonclassic glioblastoma and nonclassic anaplastic oligodendroglioma was not significantly diffe
39 based on a subset of 14 glioblastomas and 7 anaplastic oligodendrogliomas with classic histology.
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