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1 ript levels in a mouse model for conditional androgen insensitivity.
2 recurrence after radical prostatectomy, and androgen insensitivity.
3 issense mutation, R405S, that caused partial androgen insensitivity.
4 nis consistent with clinical stage 3 partial androgen insensitivity.
5 presence of ambiguous genitalia and partial androgen insensitivity.
6 of three patients exhibiting either complete androgen insensitivity (CAI) or a contiguous gene syndro
7 ant prostate cancer in man and indicate that androgen insensitivity can be an inherent property of me
8 ctivation may eventually lead to the partial androgen insensitivity during the development of Kennedy
9 yed accelerated neurodegeneration and severe androgen insensitivity in comparison to AR100 littermate
11 of the FGF-R2(111b) isoform correlated with androgen insensitivity in these human prostate cancer mo
13 ain mutations that cause partial or complete androgen insensitivity into fusion proteins containing t
14 neuron degeneration accompanied by signs of androgen insensitivity, such as gynecomastia and reduced
19 ociated with a variety of diseases including androgen insensitivity syndrome and prostate cancer, but
20 Almost all AR missense mutations that cause androgen insensitivity syndrome are located in the highl
21 nar describes the clinical manifestations of androgen insensitivity syndrome from infancy to adulthoo
23 R mutations associated with oligospermia and androgen insensitivity syndrome map to Pro-390, the cons
26 iagnosed with XY ambiguous genitalia/partial androgen insensitivity syndrome, who was found to have a
30 hed fertility, and systemic signs of partial androgen insensitivity that occur in Kennedy disease pat
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