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1 Thirty-three patients had also received androgen therapy.
2 tations in telomerase genes can improve with androgen therapy.
3 ive androgen receptor modulators (SARMs) for androgen therapy.
4 ese androgen-independent tumors despite anti-androgen therapy.
5 ncer resistance to hormone-ablative and anti-androgen therapy.
6 ese mutations may be the best candidates for androgen therapy.
7 direct role in preventing resistance to anti-androgen therapy.
8 and restores responsiveness of CRPC to anti-androgen therapy.
9 s, associated genetic factors, or history of androgen therapy.
11 w serum testosterone concentrations (bipolar androgen therapy [BAT]) in this setting might induce tum
12 blockade for prostate cancer prevention and androgen therapy for andropause treatment in elderly men
14 findings support the clinical evaluation of androgen therapy in the prevention and perhaps treatment
15 red physical changes from oestrogen and anti-androgen therapy include decreased body and facial hair,
16 drogens led to the development of novel anti-androgen therapies including abiraterone acetate and enz
19 cancers (PCs), initially responsive to anti-androgen therapies, often advance to a hormone-refractor
20 t the majority of patients treated with anti-androgen therapy progress to androgen-independence chara
21 through the transient generation of an anti-androgen therapy-resistant cell population, suggesting t
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