ライフサイエンスコーパス: anemia

1 eived primaquine developed moderately severe anemia.

2 patients (71%) manifested with sideroblastic anemia.

3 SAM domain protein that confers survival in anemia.

4 cal properties of sickle RBCs in sickle cell anemia.

5 whose inadequate diet causes iron deficiency anemia.

6 (HX) and/or undiagnosed congenital hemolytic anemia.

7 in infants at high risk for iron deficiency anemia.

8 he overlap of micronutrient deficiencies and anemia.

9 plified by the autoimmune disease pernicious anemia.

10 l or drug abuse or dependence diagnosis, and anemia.

11 usly untreated patients with severe aplastic anemia.

12 ineffective erythropoiesis and iron-loading anemia.

13 luding low birthweight delivery and maternal anemia.

14 me induced during erythropoiesis, results in anemia.

15 ld age, which was negatively associated with anemia.

16 red blood cells, resulting in a compensated anemia.

17 ng independently predicted anemia and severe anemia.

18 h unexplained, mild, chronic iron-deficiency anemia.

19 d14-Enh(-/-) mice died in response to severe anemia.

20 liac disease, gastrointestinal bleeding, and anemia.

21 d not increase SVR12 and was associated with anemia.

22 or children with nutritional iron-deficiency anemia.

23 he power estimate based on the prevalence of anemia.

24 two advances towards low-cost screening for anemia.

25 nd efficacious treatment for iron deficiency anemia.

26 vention of and control strategies to address anemia.

27 reduction of the hepatic iron store without anemia.

28 os died from subsequent liver hypoplasia and anemia.

29 s (or insufficient gain), growth failure, or anemia.

30 disease, liver and cardiac dysfunction, and anemia.

31 sorder causing profound and life threatening anemia.

32 otherapy responses and prevention of Fanconi anemia.

33 fect in mild (p = 0.93) or severe (p = 0.52) anemia.

34 that are potentially useful to treat certain anemias.

35 spread screening for nutritional and genetic anemias.

36 her chronic hereditary or acquired hemolytic anemias.

37 Anemia (107 [47.3%]), history of liver dysfunction (62 [

38 ncluded neutropenia (39%), leukopenia (16%), anemia (14%), and diarrhea (13%); the incidence of febri

39 the most common were hematologic, including anemia (15%), neutropenia (11%), and thrombocytopenia (9

40 e 3/4 cytopenias (thrombocytopenia [29%] and anemia [15%]).

41 older age depends on (1) the severity of the anemia, (2) underlying condition(s), and (3) patient-rel

42 h thrombocytopenia (32%), neutropenia (27%), anemia (23%), and leukopenia (23%) reported.

43 e thrombocytopenia (47%), neutropenia (32%), anemia (27%), leukopenia (16%), fatigue (11%), and hypon

44 >/=3 adverse events were neutropenia (78%), anemia (28%), and leukopenia (24%).

45 In PSC with anemia, 50%, 30%, 55%, and 58% of children had concomita

46 a (53% v 49%), thrombocytopenia (13% v 29%), anemia (7% v 15%), leukopenia (26% v 25%), and neuropath

47 ), leukopenia (84%), thrombocytopenia (82%), anemia (74%), and fatigue (72%); however, the majority o

48 ts were neutropenia (16%), leukopenia (11%), anemia (9%), and diarrhea (7%).

49 Anemia activated Samd14-Enh by inducing enhancer compone

50 TA1 mutations contribute to Diamond-Blackfan anemia, acute megakaryoblastic leukemia, transient myelo

51 n saturation index; intravenous iron use for anemia (adjusted odds ratio, 5.4 [95% confidence interva

52 Anemia affects 45% of women of childbearing age in Cambo

53 not been shown to prevent iron deficiency or anemia after 6 months of age.

54 Autoimmune hemolytic anemia (AIHA) is an uncommon entity that presents diagno

55 opment of warm-antibody autoimmune hemolytic anemia (also known as warm autoimmune hemolytic anemia [

56 Despite a high prevalence of anemia among nonpregnant Cambodian women, current report

57 l framework that guided the BRINDA project's anemia analyses.The BRINDA project refines approaches to

58 Mice lacking Pcbp1 exhibited microcytic anemia and activation of compensatory erythropoiesis via

59 nd T and N stage; the OS model also included anemia and age x pack-years interaction; and the PFS mod

60 decreased bone-marrow cellularity, erythroid anemia and B cell lymphopenia.

61 essential for microRNA biogenesis, leads to anemia and death.

62 ng Erf in the embryo proper exhibited severe anemia and died around embryonic day 14.5.

63 hematopoietic progenitor cells, resulting in anemia and embryonic lethality.

64 measured the bivariate associations between anemia and factors at the country level and by infection

65 including genes and/or pathways controlling anemia and feeding behavior in the trypanotolerant N'Dam

66 tients with myelofibrosis (MF) often develop anemia and frequently become dependent on red blood cell

67 Mechanistically, inflammation causes severe anemia and hypoxia in the bone environment, yet down-reg

68 bolism may be beneficial in ameliorating the anemia and ineffective erythropoiesis in thalassemias.

69 ietary iron repletion completely reversed ID anemia and ineffective erythropoiesis of Hri(-/-) , eAA,

70 ue to the use of ribavirin causing hemolytic anemia and interferon causing cytopenias.

71 VHL is a potential target for treating anemia and ischemic diseases, motivating the development

72 platelet aggregation, stronger and prolonged anemia and lung inflammation.

73 We examined the severity of anemia and measured the bivariate associations between a

74 ssembly.We assessed the associations between anemia and multiple proximal risk factors (e.g., iron an

75 Vitamin B12 deficiency causes megaloblastic anemia and neurologic disorder in humans.

76 roove adducts normally recognized by Fanconi anemia and nucleotide excision repair machinery, althoug

77 nature" in monozygotic twins both prevented anemia and reduced HSP90 binding.

78 Although a high prevalence of anemia and related disease burden have been documented i

79 ciency, and stunting independently predicted anemia and severe anemia.

80 20%), aspartate transaminase increase (15%), anemia and thrombocytopenia (each 14%), diarrhea (11%),

81 eved to occur in cases of various hereditary anemias and during blood storage.

82 ciency is the leading cause of megaloblastic anemia, and although more common in the elderly, can occ

83 renal dysfunction, restrictive lung disease, anemia, and cirrhosis.

84 tients presented early-onset severe aplastic anemia, and ERCC6L2 patients, mild pancytopenia with mye

85 at is associated with diastolic dysfunction, anemia, and high NT-proBNP.

86 ciated with dyskeratosis congenita, aplastic anemia, and idiopathic pulmonary fibrosis disrupt the bi

87 e syndromes dyskeratosis congenita, aplastic anemia, and idiopathic pulmonary fibrosis.

88 osis congenita, pulmonary fibrosis, aplastic anemia, and liver fibrosis.

89 dysfunctions syndrome (MMDS), sideroblastic anemia, and mitochondrial encephalomyopathy.

90 sting TTP signs; thrombocytopenia, hemolytic anemia, and organ damage could not be reversed, as throm

91 endelian ratios, develop hepatosplenomegaly, anemia, and thrombocytopenia, and succumb to a rapidly p

92 sickle cell disease, thalassemia, hemolytic anemias, and malaria, with both arterial and venous thro

93 diagnosed by thrombocytopenia and hemolytic anemia, associated with a deficiency in von Willebrand f

94 y of compatible blood products, resulting in anemia-associated morbidity and mortality.

95 Delayed cord clamping reduces anemia at 8 and 12 months of age in a high-risk populati

96 level, iron deficiency, and iron deficiency anemia at 8 and 12 months of age.

97 The etiology of anemia at older age is complex and ranges from bone marr

98 The prevalence of anemia at older age is increasing, and this is mainly at

99 In conclusion, severe anemia at steady state in SCD patients living in West an

100 Management of anemias at older age depends on (1) the severity of the

101 s article, we review current concepts around anemias at older age, with special emphasis on etiologie

102 EPO administration at doses used to correct anemia augmented the frequency of peripheral CD4(+)CD25(

103 talization, orthopnea, diabetes mellitus and anemia, be treated with beta-blockers, have higher eject

104 g of ESAs used to treat chemotherapy-induced anemia because evidence on the effectiveness of boxed wa

105 sive loss of bone marrow, and fatal aplastic anemia between 3 and 4 months of age.

106 are associated with maternal iron deficiency anemia, both Canada and the United States recommend univ

107 Inflammation and Nutritional Determinants of Anemia (BRINDA) project from 11,913 PSC in 11 surveys an

108 Inflammation and Nutritional Determinants of Anemia (BRINDA) project from 29,765 PSC in 16 surveys an

109 Inflammation and Nutritional Determinants of Anemia (BRINDA) project is a multiagency and multicountr

110 Inflammation and Nutritional Determinants of Anemia (BRINDA) project sought to inform the interpretat

111 Inflammation and Nutritional Determinants of Anemia (BRINDA) project were analyzed individually and c

112 Inflammation and Nutritional Determinants of Anemia (BRINDA) project were analyzed individually and c

113 Inflammation and Nutritional Determinants of Anemia (BRINDA) project were analyzed individually and c

114 Inflammation and Nutritional Determinants of Anemia (BRINDA) project were analyzed individually and p

115 Inflammation and Nutritional Determinants of Anemia (BRINDA) project were analyzed separately and poo

116 Inflammation and Nutritional Determinants of Anemia (BRINDA) project.

117 throferrone mediates hepcidin suppression by anemia, but its mechanism of action remains uncertain.

118 age were also significantly associated with anemia, but malaria and vitamin B-12 and folate deficien

119 E9.5 onwards, with the development of severe anemia by E14.5.

120 othesis that the improvement of inflammatory anemia by MMB results from inhibition of ACVR1-mediated

121 is underscored in 5q- syndrome, a macrocytic anemia caused by a large monoallelic deletion, which we

122 , diabetes mellitus, chronic kidney disease, anemia, coagulopathy, obesity, major bleeding, acute myo

123 mouse CD47 antibodies resulted in short-term anemia compared to controls, consistent with the previou

124 to replication-blocking lesions than Fanconi anemia complementation group L (FANCL)-null mutants, sug

125 ailure (CHF), length of stay, preterm labor, anemia complicating pregnancy, placental abnormalities,

126 laria infection are presented, along with an anemia conceptual framework that guided the BRINDA proje

127 tes of the primary safety endpoint were age, anemia, congestive heart failure, multivessel disease, n

128 ent risk factor for anemia globally and that anemia control must combine iron interventions with cont

129 Anemia control strategies must be informed by local data

130 ia has hampered the design and monitoring of anemia-control efforts.We aimed to evaluate predictors o

131 cross-sectional survey data, results suggest anemia-control programs should address both iron deficie

132 ; we use the well-described Diamond-Blackfan anemia (DBA) as a specific example to highlight some com

133 Diamond-Blackfan anemia (DBA) is a congenital bone marrow failure syndrom

134 Anemia, depression, and lung disease were associated wit

135 ubiquitin-conjugating enzyme of the Fanconi anemia DNA repair pathway and it is overexpressed in sev

136 wide range of phenotypic outcomes, including anemia due to protein instability and red cell lysis.

137 All participants received anemia education and a complete blood count (CBC).

138 A case of malarial anemia emphasizes the complex relationship between iron

139 agnosis after medical evaluation and Fanconi anemia exclusion.

140 Fancd2 is a component of the Fanconi anemia (FA) DNA repair pathway, which is frequently foun

141 isabled in the cancer-prone disorder Fanconi anemia (FA) ensure the maintenance of chromosomal stabil

142 Previous Fanconi anemia (FA) gene therapy studies have failed to demonstr

143 Fanconi anemia (FA) is a rare genetic disease characterized by c

144 Fanconi anemia (FA) is an inherited bone marrow failure syndrome

145 Fanconi anemia (FA) is an inherited cancer predisposition syndro

146 However, the involvement of Fanconi Anemia (FA) signaling, a unique genetic model system for

147 ties of translesion synthesis (TLS), Fanconi anemia (FA), and homologous recombination repair pathway

148 g from a rare human genetic disease, Fanconi anemia (FA), demonstrates the importance of FA genes.

149 itivity is a characteristic trait of Fanconi anemia (FA).

150 ired to suppress diseases, including Fanconi anemia (FA).

151 n the cancer predisposition syndrome Fanconi anemia (FA).

152 perpigmentation, now better known as Fanconi anemia (FA).

153 with the genome instability syndrome Fanconi anemia (FA).

154 well as bone marrow failure disorder Fanconi anemia (FA).

155 the Functional Assessment of Cancer Therapy-Anemia (FACT-An) and the Euro Qol 5 Dimension Questionna

156 o 48 months with nutritional iron-deficiency anemia, ferrous sulfate compared with iron polysaccharid

157 w regression approach as well as proposes an anemia framework to which real-world data can be applied

158 patasvir-voxilaprevir plus RBV were fatigue, anemia, gastroenteritis, and nausea.

159 ious c.2715 + 1G > A mutation in the Fanconi anemia gene, FANCD2, was over two times more common in t

160 y is a common and consistent risk factor for anemia globally and that anemia control must combine iro

161 PT2385 was well tolerated, with anemia (grade 1 to 2, 35%; grade 3, 10%), peripheral ede

162 A lack of information on the etiology of anemia has hampered the design and monitoring of anemia-

163 India's high prevalence of iron-deficiency anemia has largely been attributed to the local diet con

164 Androgens, used in the treatment of aplastic anemia, have been reported to block proliferation of and

165 We assessed relations between anemia (hemoglobin concentration <110 g/L) and severe an

166 emoglobin concentration <110 g/L) and severe anemia (hemoglobin concentration <70 g/L) and individual

167 gory to identify independent determinants of anemia (hemoglobin concertation <120 g/L).Anemia prevale

168 cord clamping also reduced the prevalence of anemia (hemoglobin level <11.0 g/dL) at 8 months in 197

169 clinical jaundice (254/318 [80%]) and severe anemia (hemoglobin level <5 g/dL) (238/310 [77%]).

170 Iron-deficiency anemia (IDA) affects millions of persons worldwide, and

171 , Thalassemia trait (TT) and iron deficiency anemia (IDA) are the two most common anemia types and ca

172 the hazards associated with iron deficiency anemia (IDA) are well known, concerns about risks associ

173 ical deficits despite prompt diagnosis of ID anemia (IDA) by screening of hemoglobin concentrations f

174 ldren in Sub-Saharan Africa, iron-deficiency anemia (IDA) is common, and many complementary foods are

175 nd poor sanitation were each associated with anemia in >50% of surveys.

176 lementary foods introduction age and risk of anemia in 18,446 children from the Jiaxing Birth Cohort,

177 Seven patients with PHD developed anemia in combination with a rise in mean corpuscular vo

178 g the cancer predisposition syndrome Fanconi anemia in depth revealed that mutant FANCA proteins enga

179 Anemia in heart failure (HF) is commonly diagnosed accor

180 Whether true (nonhemodilutional) anemia in HF can or cannot be generally inferred by [Hb]

181 tervention is an attractive option to combat anemia in India.

182 treating iron deficiency and iron deficiency anemia in infants and young children.

183 s are normalized by E2F-2 deletion; however, anemia in mice with deregulated cyclin E is not improved

184 deficiency was consistently associated with anemia in multivariate models, but the proportion of ane

185 Management and treatment of anemia in older patients often require a multidisciplina

186 Presence or absence of true anemia in patients with HF as determined by RBCV status

187 uation of iron deficiency and its associated anemia in pregnancy and early childhood and in character

188 te deficiencies, the most frequent causes of anemia in pregnancy, are discussed.

189 men or routine screening for iron deficiency anemia in pregnant women or young children.

190 l efforts.We aimed to evaluate predictors of anemia in preschool children (PSC) (age range: 6-59 mo)

191 t approaches, and 3) assess risk factors for anemia in preschool children (PSC) and women of reproduc

192 ocrit, 21-30%), and mild (hematocrit, > 30%) anemia in restrictive transfusion protocol ICUs was 67%,

193 stemic effects that exacerbate the intrinsic anemia in the adult and alter correct development in the

194 vivo, resulting in ectopic transcription and anemia in the Nan mouse model.

195 -iron recycling in the liver and ameliorated anemia in the Th3/(+) mice.

196 public health problem globally, and reducing anemia in women by 50% by 2025 is a goal of the World He

197 Anemia in women of reproductive age (WRA) (age range: 15

198 t were neutropenia (in 78% of the patients), anemia (in 43%), and thrombocytopenia (in 38%).

199 Other mechanisms leading to anemia, including malnutrition and infectious diseases,

200 eakness even in the absence of sideroblastic anemia irrespective of ethnicity.

201 In particular, iron deficiency anemia is a major global health issue, but the iron cont

202 Iron deficiency (ID) anemia is a prevalent disease, yet molecular mechanisms

203 Anemia is a widespread public health problem with 1/4 1

204 Iron deficiency anemia is common and consequential in nondialysis-depend

205 Anemia is quite frequently diagnosed in older individual

206 First, anemia is the final state in iron depletion.

207 idual or after recovery from immune aplastic anemia, is uncertain.

208 eatment-related grade 3 or 4 toxicities were anemia, leukopenia, and neutropenia (19% each); lymphope

209 Other drug-related G3 and G4 events included anemia, leukopenia, pancytopenia, nausea, hyperbilirubin

210 Secondary outcomes included hemoglobin and anemia levels at 12 months of age and ferritin level, ir

211 Main outcomes included hemoglobin and anemia levels at 8 months of age with the power estimate

212 d cells in human subjects, causing hemolytic anemia linked to impaired nicotinamide adenine dinucleot

213 l) in all studies including HF patients with anemia (low [Hb]) (7 studies, n=127), whereas only 2 of

214 Even a mild anemia may substantially affect physical and cognitive c

215 leton variably demonstrating severe neonatal anemia, membranoproliferative glomerulonephritis, liver

216 Although rarely resulting in megaloblastic anemia, mild deficiency may be associated with neurocogn

217 and alleviated anemic symptoms in a chronic anemia mouse model by regulating erythroid gene expressi

218 We show that the semi-dominant neonatal anemia (Nan) mutation in the EKLF/KLF1 transcription fac

219 ing for the pathophysiology of megaloblastic anemia observed in vitamin B12 and folate deficiency.

220 Grade 3 and 4 anemia occurred in 19% and 0% of ruxolitinib vs 0% (both

221 gnificantly associated with a higher risk of anemia (odds ratio = 1.14; 95% confidence interval: 1.01

222 cord clamping group and a relative risk for anemia of 0.91 (95% CI, 0.84-0.98), resulting in a NNT o

223 Using a rat model of anemia of chronic disease, we demonstrated that MMB trea

224 he iron absorption; clinically manifested as anemia of inflammation (AI).

225 Anemia of pregnancy, an important risk factor for fetal

226 The possible differential effects of anemia on organ dysfunctions seen in this hypothesis-gen

227 hese patients are referred to as unexplained anemia or idiopathic cytopenia of unknown significance.

228 PMF and are implicated in the development of anemia or pancytopenia.

229 specially those seeking to assess effects of anemia or transfusion on lung function).

230 ences of diagnosed celiac disease (diarrhea, anemia, or fracture) were associated with undiagnosed ce

231 Rodriguez and D'Andrea introduce the Fanconi anemia pathway and its role in DNA repair and other cell

232 Bone marrow transplantation rescued the anemia phenotype in vivo, whereas incubation with HNF1A(

233 rade >/=3 adverse events (AEs; >5%) included anemia, pneumonia, and fatigue.

234 as night blindness, higher child mortality, anemia, poor pregnancy outcomes, and reduced work capaci

235 The anemia prevalence at 90 d was lower for intervention (29

236 ns and hematocrit percentages and to a lower anemia prevalence in the target population with no repor

237 ation is recommended in populations in which anemia prevalence is high.

238 sed to detect structural hemoglobin variants.Anemia prevalence was 44% with the use of a portable hem

239 Baseline anemia prevalence was 58% (venous blood).

240 of anemia (hemoglobin concertation <120 g/L).Anemia prevalence was approximately 40% in countries wit

241 GVHD showed evidence of BM suppression, have anemia, reduced BM cellularity, and showed profound redu

242 Since anemia reduces arterial oxygen content, it further threa

243 ron supplementation will not be an effective anemia reduction strategy.We measured the effect of dail

244 Anemia-reduction programs for WRA can be improved by con

245 Thus, a GATA-2/anemia-regulated enhancer controls expression of an SAM

246 s with acute kidney injury, cardiac surgery, anemia, respiratory failure, heart failure, cardiac arre

247 e propose that Samd14-Enh and an ensemble of anemia-responsive enhancers are essential for erythrocyt

248 We describe a severe anemia resulting from a homozygous mutation (R150Q) in t

249 we chemically induce hypoxemia via hemolytic anemia resulting in HIF stabilization within circulating

250 Acquired aplastic anemia results from immune-mediated destruction of bone

251 nt-based foods was associated with increased anemia risks and lower hemoglobin concentrations, while

252 Acquired severe aplastic anemia (SAA) is a rare hematologic disease associated wi

253 f stroke prevention in pediatric sickle cell anemia (SCA), but the physiology conferring this benefit

254 Sickle cell anemia (SCA)-related cardiomyopathy is characterized by

255 account for the thrombocytopenia, hemolytic anemia, schistocytes, and tissue injury that characteriz

256 y be a significant factor in the unexplained anemias seen in critically ill patients.

257 characterized by microangiopathic hemolytic anemia, severe thrombocytopenia, and organ ischemia link

258 heightened impact of combined etiologies on anemia severity is highlighted.

259 The anemia signature indicated deregulation of host erythrop

260 Anemia suppresses liver hepcidin expression to supply ad

261 ne adults with HCV infection and sickle cell anemia, thalassemia, or hemophilia A/B or von Willebrand

262 response among patients with severe aplastic anemia than in a historical cohort.

263 al failure, hypokalemia, hypomagnesemia, and anemia than patients in the itraconazole group.

264 -globulin and more frequently had microcytic anemia than those without such deposits (P = .0072, P =

265 ysis is a fundamental feature of sickle cell anemia that contributes to its pathophysiology and pheno

266 e cell disease (SCD), a congenital hemolytic anemia that exacts terrible global morbidity and mortali

267 ntribute to the potentially life-threatening anemia that may accompany blood-stage malaria infection.

268 sia, is characterized by nonimmune hemolytic anemia that resolves with antimicrobial treatment and cl

269 In patients with aplastic anemia that was refractory to immunosuppression, eltromb

270 trient assessment and to better characterize anemia.The aims of the project were to 1) identify facto

271 Microangiopathic hemolytic anemia, thrombocytopenia, and acute kidney injury were f

272 A peripheral blood smear revealed anemia, thrombocytopenia, and blast cells, and a diagnos

273 10%, 4%, and 3% of the patients experienced anemia, thrombocytopenia, and leukopenia, respectively.

274 PMF, overt PMF was enriched in patients with anemia, thrombocytopenia, leukopenia, higher blast count

275 Antibiotic-treated mice exhibited anemia, thrombocytosis, and leukopenia, with pronounced

276 l in adults with NDD-CKD and iron deficiency anemia to compare the safety and efficacy of oral ferric

277 ross the full continuum from iron deficiency anemia to iron deficiency to iron replete to iron excess

278 iciency anemia (IDA) are the two most common anemia types and can have a serious impact on quality of

279 phalopathy, hepatocellular carcinoma, severe anemia, untreated hypothyroidism, Addison syndrome and m

280 dehydrogenase, exhibit non-immune hemolytic anemia upon exposure to aspirin and various antimalarial

281 e not.The contribution of iron deficiency to anemia varies according to a country's infection burden.

282 One such orthomyxovirus, infectious salmon anemia virus (ISAV), spreads easily throughout farmed an

283 mia (also known as warm autoimmune hemolytic anemia [WAHA]) in patients with babesiosis has not previ

284 ncy with optic atrophy, and in four patients anemia was present at diagnosis.

285 Associations between breastfeeding and anemia were attenuated by controlling for child age, whi

286 The most consistent predictors of severe anemia were malaria, poor sanitation, and underweight.

287 reah Vihear, 340 women with mild or moderate anemia were randomly assigned to 1) an iron-ingot group,

288 reased dietary iron intake, and iron-loading anemias whereby both increased iron absorption and trans

289 n B12 deficiency can result in megaloblastic anemia, which results from the inhibition of DNA synthes

290 demonstrated that MMB treatment ameliorated anemia, which was unexpected for a JAK1/2 inhibitor, bec

291 hem from doxorubicin-induced neutropenia and anemia while retaining antitumor efficacy.

292 chain variant in a female patient with mild anemia, whose father also carries the trait and is from

293 reduced the odds of transfusion in moderate anemia with an odds ratio of 0.59 (95% CI, 0.36-0.96) wh

294 edictors; we also examined the proportion of anemia with concomitant iron deficiency (defined as an i

295 A(-/-) mice displayed microcytic hypochromic anemia with reticulocytosis that was partially compensat

296 roup of related congenital dyserythropoietic anemias with thrombocytopenia.

297 d reduced erythroid progenitors resulting in anemia, with dysregulated expression of Cebpa and Gata1,

298 lassemia is one of the most common inherited anemias, with no effective cure for most patients.

299 vivo with the injection of histones inducing anemia within minutes of administration, with a concomit

300 ve been identified in X-linked sideroblastic anemia (XLSA) pedigrees, strongly suggesting it could be