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1 r chronic kidney disease (formerly known as "anemia of chronic diseases").
2  hemochromatosis and anemia of inflammation (anemia of chronic disease).
3 ay in treating diseases of iron overload and anemia of chronic disease.
4 with myelodysplastic syndromes (MDS) and the anemia of chronic disease.
5 tical to what is commonly referred to as the anemia of chronic disease.
6 efractory anemia similar to that observed in anemia of chronic disease.
7 NO production and the pathophysiology of the anemia of chronic disease.
8 stinguishing iron deficiency anemia from the anemia of chronic disease.
9  by interleukin 6, eventually leading to the anemia of chronic disease.
10 impaired mobilization of iron stores and the anemia of chronic disease.
11 ism is secondarily disregulated, such as the anemia of chronic disease.
12 ng IDA, and distinguish between IDA from the anemia of chronic disease.
13 seful therapeutic agent for treatment of the anemia of chronic disease, a disorder resulting from hig
14 oluble transferrin receptor (sTfR) values in anemia of chronic disease (ACD) and iron deficiency anem
15 hormone, hepcidin, leading to development of anemia of chronic disease (ACD).
16 n clinical manifestation of these changes is anemia of chronic disease (ACD; also called anemia of in
17 d the erythroid iron restriction response in anemia of chronic disease and inflammation (ACDI), offer
18  chronic inflammation (ACI), formerly termed anemia of chronic disease, and unexplained anemia (UA).
19    Other common causes of anemia in AIDS are anemia of chronic disease consequent on opportunistic in
20 t it is important in the pathogenesis of the anemia of chronic disease in general.
21 is found in many disease states, such as the anemia of chronic disease, iron refractory iron deficien
22                                          The anemia of chronic disease is a prevalent, poorly underst
23 ilability, and its excess contributes to the anemia of chronic diseases, the most prevalent anemia in
24                         Thus, similar to the anemia of chronic disease, these findings demonstrate de
25                         Using a rat model of anemia of chronic disease, we demonstrated that MMB trea
26 e marrow progenitor cells that contribute to anemia of chronic disease, which is very common in acute
27 s has been implicated in the pathogenesis of anemia of chronic disease, while hepcidin deficiency has
28  in heart failure, favors the development of anemia of chronic disease, with defective iron utilizati

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