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1 y secretions that could be attributed to the anesthetic agent.
2 s a powerful anxiolytic, anticonvulsant, and anesthetic agent.
3 endent on a known interaction site for local anesthetic agents.
4 sm, which alter the pharmacokinetics of many anesthetic agents.
5 losporine A, radiocontrast dyes and volatile anesthetic agents.
6 imental evidence supports neuroprotection by anesthetic agents.
7 ro- and anti-convulsant effects of the newer anesthetic agents.
8                                     Volatile anesthetic agent, 1-chloro-1,2,2-trifluorocyclobutane (F
9  and enflurane, three commonly used volatile anesthetic agents, affect glutamate receptor-modulated n
10 ve considerable potential to be developed as anesthetic agents and as drugs to treat brain disorders
11 r any of the commonly available inhalational anesthetic agents and each can be used for general anest
12  associated with the use of lower amounts of anesthetic agents and faster recovery from anesthesia in
13 e cells, and are molecular targets for local anesthetic agents and intracellular free Ca(2+) ([Ca(2+)
14 1 measurements obtained with several inhaled anesthetic agents and propofol dissolved in saline were
15                                  Advances in anesthetic agents and techniques for interventional pulm
16 he level of consciousness in volunteers with anesthetic agents and visualizing the resultant changes
17        All exposed children received inhaled anesthetic agents, and anesthesia duration ranged from 2
18 story of corneal surgery, allergy to topical anesthetic agents, and current eye infection.
19 idence for neuroprotective effects of common anesthetic agents, and presents potential mechanisms inv
20 mparison to other small hydrophobic poisons, anesthetic agents, and protein toxins that inhibit ion c
21             Mechanisms of neuroprotection by anesthetic agents appear to involve suppression of excit
22                                      Gaseous anesthetic agents are among the most potent drugs that c
23 visiae was used to investigate the action of anesthetic agents because of its powerful molecular gene
24 rcotics, antipsychotics, inotropes, digoxin, anesthetic agents, bronchodilators, and drugs that cause
25 corporating standard monitoring of end-tidal anesthetic-agent concentration (ETAC) for the prevention
26 esthetic management directed by an end-tidal anesthetic-agent concentration protocol is equally effec
27 uch higher (toxic) concentrations of certain anesthetic agents did alter lipid bilayer properties.
28              Although it is widely held that anesthetic agents disrupt the processes regulating LH re
29 y benefit from neuroprotective properties of anesthetic agents during surgery.
30 he same surgical preparation and sequence of anesthetic agent exposure but no brain insult.
31 ed mutation at an interaction site for local anesthetic agents (F1760A) partially attenuated the effe
32                       Isoflurane, a volatile anesthetic agent, has been recognized for its potential
33                                 Inhalational anesthetic agents have also been shown to result in brai
34                                              Anesthetic agents have been shown to have a profound eff
35                   All currently used general anesthetic agents have either NMDA receptor-blocking or
36                         Furthermore, because anesthetic agents have the unique ability to reversibly
37 Ca(2+), changes in internal and external pH, anesthetic agents, heat, stretch and membrane deformers.
38 the pharmacokinetics and pharmacodynamics of anesthetic agents in the obese population, we specify ce
39 or measuring the hydrophobicity of the local anesthetic agents in the unionized form.
40                                   The use of anesthetic agents, including propofol, have been adminis
41 nderstanding how illicit drugs interact with anesthetic agents is of paramount importance.
42 that anesthetic management and even specific anesthetic agents may worsen outcomes in high-risk patie
43                  Halogenated inhaled general anesthetic agents modulate voltage-gated ion channels, b
44 a method for studying the interaction of two anesthetic agents, Morphine and Midazolam, acting simult
45 inhaled volatile (isoflurane or sevoflurane) anesthetic agent (n = 67).
46 ting or motion sickness, young age, volatile anesthetic agents, nitrous oxide, and the administration
47 sics, benzodiazepines, opioids, inhalational anesthetic agents, nitrous oxide, ketamine, barbiturates
48 trate clear-cut superiority for any specific anesthetic agent or approach.
49 ion mucociliary transportability with either anesthetic agent or with atropine.
50 tudies that would support the use of any one anesthetic agent over the others.
51  evaluation of patients, discovery of better anesthetic agents, pain control, and the evolution of pe
52  on the precise mechanisms by which hypnotic anesthetic agents produce their effects.
53                                          The anesthetic agent propofol (2,6-diisopropylphenol) is the
54               We used functional MRI and the anesthetic agent propofol to assess the relationship amo
55                            Here, we used the anesthetic agent, propofol, to modulate consciousness an
56            Here we show that two GABAmimetic anesthetic agents--propofol and sodium thiopental--prote
57                                 Many general anesthetic agents regulate voltage-gated Na(+) (NaV) cha
58 s of initiating brexanolone infusion without anesthetic agent reinstatement in the following 24 hours
59 cological postconditioning with the volatile anesthetic agent sevoflurane (n = 48), intermittent clam
60                         Three other volatile anesthetic agents show a similar pattern.
61 pport a neuroprotective potential of several anesthetic agents, specifically isoflurane and xenon, co
62 erefore, we propose that amphiphilic general anesthetic agents such as 1-alkanols may modulate gating
63                   Propofol is a sedative and anesthetic agent that can both activate GABA(A) receptor
64                                              Anesthetic agents that do not interfere with auditory fu
65      Other studies have shown that titrating anesthetic agents to a specific bispectral index value d
66 uring surgery and also to optimize dosing of anesthetic agents to maximize the desired/undesired effe
67 nificant correlation between the duration of anesthetic agent use and DeltaVBR was found (Spearman r
68                                     Volatile anesthetic agent use in the intensive care unit, aided b
69 s, perhaps because of confounding effects of anesthetic agents used in most animal experiments on fun
70 esized, and the utility of these steroids as anesthetic agents was evaluated through determination of
71                                              Anesthetic agents were required for a median of 13 (IQR,
72                                              Anesthetic agents were used in 55 episodes (87.30%), and
73   Xenon and dichloromethane are inhalational anesthetic agents whose binding to myoglobin has been de
74        Propofol is a widely used intravenous anesthetic agent with antioxidant properties.
75 EG parameters to increasing concentration of anesthetic agents with a dosing plateau response over a
76 udies could examine the use of longer-acting anesthetic agents with ultrasonography-guided BRSB.
77 001) and receipt of intravenous propofol, an anesthetic agent without preservative, which was adminis

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